Kelly Spong, RN, BSN, MBA, CWOCN, CHRN & Sandra Macfarlane, PT, DPT
Kelly Spong RN BSN MBA CWOCN CHRN amp Sandra Macfarlane PT DPT
List the characteristics of lower extremity ulcers
Compare and contrast arterial and venous leg ulcers
Verbalize what an ABI test is and what the results indicate
Chronic venous insufficiency accounts for 70 of leg ulcers (mostly female)
Arterial insufficiency accounts for 10 Mixed disease accounts for 10-15 of the
ulcers Other trauma neoplasms etc
General appearance of the limb Edema color changes elevational pallor dependent
rubor skin temperature Perfusion capillary refill time le3 seconds Skin temperature Pulses Ankle-brachial index (ABI) Toe-brachial index (TBI)
Sensory function Range of motion Pain
Extravasation of the skin and underlying tissues and structures from ischemia due to insufficient perfusion to an extremity or location
Atherosclerosis is the most common cause (thickening and decreased elasticity of the arterial walls)
Accumulation of plaque fibrin platelets and cellular debris in arteries
Diabetes and smoking accelerates development of atherosclerosis
Hyperlipidemia and hypertension contribute to the development of atherosclerosis
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
List the characteristics of lower extremity ulcers
Compare and contrast arterial and venous leg ulcers
Verbalize what an ABI test is and what the results indicate
Chronic venous insufficiency accounts for 70 of leg ulcers (mostly female)
Arterial insufficiency accounts for 10 Mixed disease accounts for 10-15 of the
ulcers Other trauma neoplasms etc
General appearance of the limb Edema color changes elevational pallor dependent
rubor skin temperature Perfusion capillary refill time le3 seconds Skin temperature Pulses Ankle-brachial index (ABI) Toe-brachial index (TBI)
Sensory function Range of motion Pain
Extravasation of the skin and underlying tissues and structures from ischemia due to insufficient perfusion to an extremity or location
Atherosclerosis is the most common cause (thickening and decreased elasticity of the arterial walls)
Accumulation of plaque fibrin platelets and cellular debris in arteries
Diabetes and smoking accelerates development of atherosclerosis
Hyperlipidemia and hypertension contribute to the development of atherosclerosis
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Chronic venous insufficiency accounts for 70 of leg ulcers (mostly female)
Arterial insufficiency accounts for 10 Mixed disease accounts for 10-15 of the
ulcers Other trauma neoplasms etc
General appearance of the limb Edema color changes elevational pallor dependent
rubor skin temperature Perfusion capillary refill time le3 seconds Skin temperature Pulses Ankle-brachial index (ABI) Toe-brachial index (TBI)
Sensory function Range of motion Pain
Extravasation of the skin and underlying tissues and structures from ischemia due to insufficient perfusion to an extremity or location
Atherosclerosis is the most common cause (thickening and decreased elasticity of the arterial walls)
Accumulation of plaque fibrin platelets and cellular debris in arteries
Diabetes and smoking accelerates development of atherosclerosis
Hyperlipidemia and hypertension contribute to the development of atherosclerosis
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
General appearance of the limb Edema color changes elevational pallor dependent
rubor skin temperature Perfusion capillary refill time le3 seconds Skin temperature Pulses Ankle-brachial index (ABI) Toe-brachial index (TBI)
Sensory function Range of motion Pain
Extravasation of the skin and underlying tissues and structures from ischemia due to insufficient perfusion to an extremity or location
Atherosclerosis is the most common cause (thickening and decreased elasticity of the arterial walls)
Accumulation of plaque fibrin platelets and cellular debris in arteries
Diabetes and smoking accelerates development of atherosclerosis
Hyperlipidemia and hypertension contribute to the development of atherosclerosis
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Extravasation of the skin and underlying tissues and structures from ischemia due to insufficient perfusion to an extremity or location
Atherosclerosis is the most common cause (thickening and decreased elasticity of the arterial walls)
Accumulation of plaque fibrin platelets and cellular debris in arteries
Diabetes and smoking accelerates development of atherosclerosis
Hyperlipidemia and hypertension contribute to the development of atherosclerosis
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Atherosclerosis is the most common cause (thickening and decreased elasticity of the arterial walls)
Accumulation of plaque fibrin platelets and cellular debris in arteries
Diabetes and smoking accelerates development of atherosclerosis
Hyperlipidemia and hypertension contribute to the development of atherosclerosis
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Pain Pain with exercise Rest pain with elevation Decreased pain with limb in dependent position
Pulse Diminished or absent
Pallor Pallor with elevation Dependent rubor Skin appears shiny smooth and hairless Thickened yellow nails
Gangrene Gangrene wet or dry
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
History and physical Ulcer Location distal lower leg (usually ankle area down) Pulses proximal an distal (may be weak to absent) ldquopunched outrdquo appearance and wound edges Pale or necrotic ulcer base Usually deeper in nature Minimal exudate Infection common Surrounding skin (erythema possible induration or
cellulitis) More common in males
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Non-invasive Ankle-brachial index normal= 10-095 Mild insufficiency= 094-074 Moderate insufficiency= 074-050 Severe insufficiency= below 05
Toe pressures (should be done with DM) Dopplers with waveforms Transcutaneous oxygen tension Pulse volume recording Segmental pressure recordings Color duplex imaging
Invasive Angiography (arteriogram)
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Manage cause and correct underlying pathology Revascularization (bypass grafts angioplasty) Control diabetes (monitor routinely HgA1c) Lifestyle changes (smoking obesity caffeine BP) Therapies HBO proper wound care Pharmacotherapy (antiplatelet vasodilators
antilipemics analgesics)
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Impaired return of venous blood from the tissues to the heart Aka Lower extremity venous disease (LEVD) More common than arterial 70-90 of leg ulcers Female dominant Valvular incompetence results in venous congestion Obstruction of deep venous system Calf muscle dysfunction
Venous System Veins Deep veins-femoral popliteal tibial Superficial veins- greaterlessor saphenous Perforator veins
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Location Medial aspect of the lower leg and ankle Superior to medial malleolous Seldom occur on foot or above knee
Wound appearance Irregular wound margins Granulation frequently present Usually shallow in depth Exudate usually moderate to heavy
Surrounding skin Erythema Cellulitis Hemosiderin staining
Painpulse Minimal unless infected Present
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Patient history Lower extremity characteristics Edema Hemosiderosis Dermatitis Atrophie blanche (picture) Varicose veins Ankle flaringvisible capillaries in sunburst pattern Scaring Tinea pedis (athletes foot) Lipodermatasclerosis (picture)
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Non-invasive Ankle brachial index (to rule out arterial or mixed
disease) Toe pressure Doppler
Invasive Venography uses dye
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Gold standard Compression Therapy Provide gradient pressure from ankle to knee Ranges in compression from 20-60 mm HG LEVD needs at least 30-40 mm Hg at ankle Contraindicated with moderate to severe arterial disease
ABIrsquos must be done prior to compression Normal ge10-13 Borderline le06-08 Severe ischemia le05 Critical limb ischemia le04 gt13=abnormally high typically due to calcification of
vessel wall in patients with DMmdashneed to perform TBI
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence
Physical therapy and exercise Eliminate edema Weight control Pharmacologic Trental-reduces capillary
plugging Topical Therapy absorb drainage Treat dermatitis protect periwound skin Prevent recurrence