I. INTRODUCTION Passage of a urinary stone is the single most common cause of acute ureteral obstruction and affects as many as 12% of the population. The pain may be some of the most severe pain that humans experience, and complications of stone disease may result in severe infection; renal failure; or, in rare cases, death (http://emedicine.medscape.com/article/381993-overview). Semins et al performed a study regarding how increased body mass index (BMI) may affect the risk of kidney stone disease. They found that in patients with a BMI greater than 30 kg/m 2 , there was a significantly increased likelihood of kidney stone formation. Renal calculi occur in 5-12% of the American population, and they are bilateral in 10-15% of patients. The prevalence of urinary lithiasis is as high as 2-3% in the general population. A slightly lower prevalence of urinary stones is found in less developed countries, possibly because of diets lower in protein. Passage of a renal stone is the most common cause of acute ureteral obstruction. When this occurs, pressure in the collecting system and renal blood flow acutely increase, followed by decreased blood flow after 1-2 hours. Hematuria usually occurs. This can be intermittent or persistent and microscopic or gross. However, as many as 10% of patients with acute stones may not have hematuria. Acute ureteral obstruction by stone causes severe, colicky (intermittent) 1
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Case Study Non-Fixing Right Kidney Secondary to Obstructing Distal Ureterolithiasis With Nephrolithiasis
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I. INTRODUCTION
Passage of a urinary stone is the single most common cause of acute ureteral
obstruction and affects as many as 12% of the population. The pain may be some of
the most severe pain that humans experience, and complications of stone disease
may result in severe infection; renal failure; or, in rare cases, death
(http://emedicine.medscape.com/article/381993-overview). Semins et al performed a
study regarding how increased body mass index (BMI) may affect the risk of kidney
stone disease. They found that in patients with a BMI greater than 30 kg/m2, there
was a significantly increased likelihood of kidney stone formation. Renal calculi
occur in 5-12% of the American population, and they are bilateral in 10-15% of
patients. The prevalence of urinary lithiasis is as high as 2-3% in the general
population. A slightly lower prevalence of urinary stones is found in less developed
countries, possibly because of diets lower in protein.
Passage of a renal stone is the most common cause of acute ureteral
obstruction. When this occurs, pressure in the collecting system and renal blood flow
acutely increase, followed by decreased blood flow after 1-2 hours. Hematuria
usually occurs. This can be intermittent or persistent and microscopic or gross.
However, as many as 10% of patients with acute stones may not have hematuria.
Acute ureteral obstruction by stone causes severe, colicky (intermittent) flank pain
that can radiate throughout the groin, testicles, back, or periumbilical region. Some
patients with renal calculi may have no symptoms at all. Stones smaller than 4 mm
pass spontaneously in approximately 80% of patients. Stones that are 4-6 mm pass
in approximately 50% of patients, whereas stones larger than 8 mm pass in only
approximately 20% of patients. Occasionally, recurrent infection may result in
pyelonephritis or abscess. Stones can cause renal scarring, damage, or even renal
failure if they are bilateral. In 10% of patients, stones recur within 1 year. This
percentage increases to 50% within 10 years.
Urinary stones occur more often in white populations than in black
populations. They are also more prevalent in highly developed countries, possibly as
a result of a higher protein diet. Males are at a greater risk than females, with a
Bleeding between periods: “wala mi kabalo” (“we don’t know”)
Pregnancy HX: 4x been pregnant
Episiotomy: “normal mi tanan” (“we are all delivered normally”)
Lochia: none
Complication of pregnancy: “wala” (none)
Surgeries: “mattress-tumor sa mattress (dugay nah)”
Hormonal therapy/calcium use: “wala” (none)
Practice SBE: “wala” (none)
Discharges: “wala” (none)
Last PAP smear: “wala man mi nag pacheckup”
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(“we never been to some check-ups”)
Method of birth control: “wala sila ana” (“they don’t practice such”)
Others/Comments: Patient has not undergone any examination on his genitals
Objective
Breast examination: “wala” (none)
Vaginal warts/lesions: none
XIII. SOCIAL INTERACTIONS
Subjective
Marital status: “Minyo ko” (Married)
Yrs in relationship: “43 years”
Living with: “with apo and family” (“with grandchildren and family”)
Concerns/stresses: “sa kwarta jud karon” (“for now, it would be money matters”)
Extended family: “oo” (“yes”)
Other support person: “anak” (“children”)
Role within family structure: mother
Report of problems related to illness/condition: “dili na kayo makatabang sa balay
tungod aning sakit ni mama” (“she’s unable to help us with the household chores
because of her present condition”)
Others/comments: The main concern of the patient is about money matters and her
present condition.
XIV. Teaching/ Learning
Subjective
Dominant language (specify): “bisaya” (“visayan”)
Literate: “oo” (“yes”)
Educational Level: Elementary Graduate
Health beliefs/ practices: “wala” (none)
Familial risk factors:
Diabetes: none
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TB: none
Heart Disease: none
Stroke: none
High BP: none
Epilepsy: none
Kidney Disease: renal failure(Paternal side).
Cancer: none
Mental Illness: none
Use of alcohol: “panalagsa. Kung naai party” (“sometimes, and when there is a party”)
Others/comments: drink alcohol of about 1-2 glasses.
XV - Body Map
(Illustrate in the body map how your patient looks like, e.g. tubes inserted, bruises, surgical incisions, physical abnormalities, affected areas. Mark with a small “X” where it is located or draw it on the body map and then label it in the space provide.)
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Slightly stooped
Productive cough w/ yellowish phlegm
Skin turgur: decreased elasticity
Dry and pale mucous membranes
Breath sound: crackles
Weak grasp
Shallow breathing
Moving slowly and avoiding physical actions
Oily scalp
Slight body odor noted
IVF: D5LR 1 L @ 30 gtts/min infusing well @ right handDate ordered:12/12/201000
surgical wound @
the right lower
quadrant of the
abdomen (@
mcburney’s point)
with sutures intact
with minimal
serous blood
discharges.
Pain: gnawing and achingIntensity: 8 of the
pain scale 1-10
and 10 as the
highest and 1 as
the lowest
B. History Present illness:
5 months: prior to admission patient had sudden onset of right flank pain radiating to the right upper quadrant as well as pain in urination. Patient was admitted to a local hospital in Initao and admitted which in ultrasound revealed nephrolithiasis.
3 months: prior to admission patient was again admitted in a local hospital in Initao because of blood in urination and was initially diagnosed to have Ureterolithiasis and was referred to this institution.
2 months: prior to admission patient consulted in this institution(NMMC). Patient’s kidneys were examined and the results was: Nonfunctioning kidneys secondary ureterolithiasis with nephrolithiasis.
2 weeks: prior to admission, patient’s s.o. said: gahi iyang kilid gihilantan siya ug gitakigan. gidala namo siya sa ospital sa initao para magpa check up pero nag ingon ang nurse “balik pagka huwebes kai nay private doctor from CDO “. Pagbalik namo pagpa.check-up, gi UTZ siya. 3 days after mi nagpa.UTZ, ingon ang doctor nga naa daw stone sa right niya nga kidney. Gi-adto dayon mi sa Mercury Hospital sa Iligan, pagtan-aw sa result, ingon ang doctor nga kelangan daw operahan kai dako daw pud ang bato.
10 days: prior to admission, pt’s urine was tested and revealed nephritis and infection. Patient’s blood was also tested and revealed nephritis and hemoconcentration.
5 days: prior to admission the complained for pain felt “ Ga inum siya ug tambal para mawala ra. Ginadaladala raman gud na niya” as verbalized by SO.
1 day: prior to admission the patient consulted the local hospital for the schedule of the surgery due to the pain that the patient felt. The pain got worst but can still tolerate and relieved by rest. The patient can still do light activity.
2-3 hours: prior to admission the patient was unable to walk straighten up his body due to pain and already needs assistance.
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C. Laboratory Results
Radiographic ReportPatient: NQPlate no. 2010-11-1619Tentative Diagnosis: HydronephrosisDate Examined: October 28, 2010Parts Examined: Kidneys Requested by: Dr. EFindings:
IV UROGRAMScout film shows a non-obstructive gastrointestinal gas pattern/ Psoas shadows are distinct. An ovoid Calcific density measuring 3.2 x 2.1 cms in the Right hemiabdomen between L2 & L3 and another irregular Calcific Density seen measuring 1.1 x 0.7 cms in the Right iliac wing. The visualized osseous structures are unremarkable. Serial films following intravenous administration of contrast opacification of the left Pelvocalyceal System which exhibits no dilatation, but filling defect. The ureter likewise opacifice and runs a normal course with normal caliber. There is no nephrogram effect noted on the Right Kidney. There is also opacification of the right pelvocalyceal system and ureter even in delayed 24-hour study. The bladder adequately distends which shows an remarkable mucosal pattern with filling defect .Post void study shows a uncomplete emptying of the bladder.Impression:
o Nonfunctioning kidneys secondary ureterolithiasis with nephrolithiasis.
Chest – PA, upright viewo There is no evidence of active parenchymal infiltrates.
o Heart is not enlarged
o Aorta, Trachea, Diaphragm and Sulci are unremarkable.
Impression:No radiographic abnormality in chest.
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St. Ignatius Urine AnalysisDate: 12/1/2010 11:06AM
Normal values
Interpretation
MacroscopicColor Straw Yellow-amber NormalTransparency Turbid Clear to
slightly turbidNormal
pH 4.0 4.8-8.0 Acidic: indicates accumulation of solutes in the urine in relation to the calculi formation.
Specific Gravity 1.010 1.005-1.030 NormalChemicalProtein Trace Negative Indicates nephritisSugar Negative Negative NormalMicroscopic- CellPUS cells Loaded/HPF Few Indicates InfectionRBC 1-3/HPF 0-2/HPF NormalMicroscopic- EpitheliumSquamous Few None or few NormalBacteria Moderate None Indicates Infection
St. IgnatiusClinical ChemistryDate: 12/1/2010
Result Normal Values InterpretationRBS 106.5 80-140 NormalCreatinine 1.0 0.5-1.3 NormalBUN 29.0 10-30 NormalSodium 157.0 135-145 Nephritis,
hemoconcentrationPotassium 4.1 3.5-5.0 Normal
Ultrasound KUB December 1, 2010 Interpretation:Urinary retention, 153.7 ml (66%)Nephrolithiasis, right kidney with regression in size and numbersNephrolithisasis, left kidney with regression
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St. IgnatiusHematology ReportDate: 12/1/2010
Patient value Normal values InterpretationTotal WBC 5.9 5-10 NormalTotal RBC 4.10 3.69-5.9 NormalHemoglobin 10.6 11.7-14.0 Decrease Hemoglobin
infection, nephrolithiasis, and urinary tract obstruction. Various cancers of the kidney
exist; the most common adult renal cancer isrenal cell carcinoma. Cancers, cysts, and
some other renal conditions can be managed with removal of the kidney,
or nephrectomy. When renal function, measured by glomerular filtration rate, is
persistently poor, dialysis and kidney transplantation may be treatment options.
Although they are not severely harmful, kidney stones can be a pain and a nuisance.
The removal of kidney stones includes sound wave treatment, which breaks up the
stones into smaller pieces which are then passed through the urinary tract. One
common symptom of kidney stones is a sharp pain in the medial/lateral segments of the
lower back.
Location
In humans, the kidneys are located in the abdominal cavity, and lie in a retroperitoneal
position. There are two, one on each side of the spine. The asymmetry within the
abdominal cavity caused by the liver typically results in the right kidney being slightly
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lower than the left, and left kidney being located slightly more medial than the right. The
left kidney is approximately at the vertebral level T12 to L3, and the right slightly lower.
The right kidney sits just below thediaphragm and posterior to the liver, the left below
the diaphragm and posterior to the spleen. Resting on top of each kidney is an adrenal
gland. The upper (cranial) parts of the kidneys are partially protected by the eleventh
and twelfth ribs, and each whole kidney and adrenal gland are surrounded by two layers
of fat (the perirenal and pararenal fat) and the renal fascia. Each adult kidney weighs
between 125 and 170 grams in males and between 115 and 155 grams in females. The
left kidney is typically slightly larger than the right.
Structure
The kidney has a bean-shaped structure, each kidney has concave and convex
surfaces. The concave surface, the renal hilum, is the point at which the renal
artery enters the organ, and therenal vein and ureter leave. The kidney is surrounded by
tough fibrous tissue, the renal capsule, which is itself surrounded by perinephric
fat, renal fascia (of Gerota) and paranephric fat. The anterior (front) border of these
tissues is theperitoneum, while the posterior (rear) border is thetransversalis fascia.
The superior border of the right kidney is adjacent to the liver; and the spleen, for the
left border. Therefore, both move down on inhalation.
The kidney is approximately 11–14 cm in length, 6 cm wide and 4 cm thick.
The substance, or parenchyma, of the kidney is divided into two major structures:
superficial is the renal cortex and deep is the renal medulla. Grossly, these structures
take the shape of 8 to 18 cone-shaped renal lobes, each containing renal cortex
surrounding a portion of medulla called a renal pyramid (of Malpighi). Between the renal
pyramids are projections of cortex called renal columns (of Bertin). Nephrons, the urine-
producing functional structures of the kidney, span the cortex and medulla. The initial
filtering portion of a nephron is the renal corpuscle, located in the cortex, which is
followed by a renal tubule that passes from the cortex deep into the medullary pyramids.
Part of the renal cortex, a medullary ray is a collection of renal tubules that drain into a
single collecting duct.
The tip, or papilla, of each pyramid empties urine into a minor calyx, minor calyces
empty into major calyces, and major calyces empty into the renal pelvis, which becomes
the ureter.
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Blood supply
receive blood from the renal arteries, left and right, which branch directly from
theabdominal aorta. Despite their relatively small size, the kidneys receive
approximately 20% of thecardiac output.
Each renal artery branches into segmental arteries, dividing further into interlobar
arteries which penetrate the renal capsule and extend through the renal columns
between the renal pyramids. The interlobar arteries then supply blood to the arcuate
arteries that run through the boundary of the cortex and the medulla. Each arcuate
artery supplies several interlobular arteries that feed into the afferent arterioles that
supply the glomeruli.
The interstitum (or interstitium) is the functional space in the kidney beneath the
individual filters (glomeruli) which are rich in blood vessels. The interstitum absorbs fluid
recovered from urine. Various conditions can lead to scarring and congestion of this
area, which can cause kidney dysfunction and failure.
After filtration occurs the blood moves through a small network of venules that converge
into interlobular veins. As with the arteriole distribution the veins follow the same
pattern, the interlobular provide blood to the arcuate veins then back to the interlobar
veins which come to form the renal vein exiting the kidney for transfusion for blood.
Histology
Innervation
The kidney and nervous system communicate via the renal plexus, whose fibers course
along the renal arteries to reach the kidney. Input from the sympathetic nervous
system triggers vasoconstriction in the kidney, thereby reducing renal blood flow. The
kidney is not thought to receive input from the parasympathetic nervous system.
Sensory input from the kidney travels to the T10-11 levels of the spinal cord and is
sensed in the corresponding dermatome. Thus, pain in the flank region may be referred
from the kidney.
Functions
The kidney participates in whole-body homeostasis, regulating acid-base
balance, electrolyteconcentrations, extracellular fluid volume, and regulation of blood
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pressure. The kidney accomplishes these homeostatic functions both independently
and in concert with other organs, particularly those of the endocrine system. Various
endocrine hormones coordinate these endocrine functions; these
include renin, angiotensin II, aldosterone, antidiuretic hormone, andatrial natriuretic
peptide, among others.
Many of the kidney's functions are accomplished by relatively simple mechanisms of
filtration, reabsorption, and secretion, which take place in the nephron. Filtration, which
takes place at the renal corpuscle, is the process by which cells and large proteins are
filtered from the blood to make an ultrafiltrate that will eventually become urine. The
kidney generates 180 liters of filtrate a day, while reabsorbing a large percentage,
allowing for only the generation of approximately 2 liters of urine. Reabsorption is the
transport of molecules from this ultrafiltrate and into the blood. Secretion is the reverse
process, in which molecules are transported in the opposite direction, from the blood
into the urine.
Excretion of wastes
The kidneys excrete a variety of waste products produced by metabolism. These
include the nitrogenous wastes urea, from proteincatabolism, and uric acid, from nucleic
acid metabolism.
Acid-base homeostasis
Two organ systems, the kidneys and lungs, maintain acid-base homeostasis, which is
the maintenance of pH around a relatively stable value. The kidneys contribute to acid-
base homeostasis by regulating bicarbonate (HCO3-) concentration. The kidneys have
two important roles in the maintaining of the acid-base balance: to reabsorb bicarbonate
from and to excrete hydrogen ions into urine
Osmolality regulation
Any significant rise in plasma osmolality is detected by the hypothalamus, which
communicates directly with the posterior pituitary gland. An increase in osmolality
causes the gland to secrete antidiuretic hormone (ADH), resulting in water reabsorption
by the kidney and an increase in urine concentration. The two factors work together to
return the plasma osmolality to its normal levels.
ADH binds to principal cells in the collecting duct that translocate aquaporins to the
membrane allowing water to leave the normally impermeable membrane and be
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reabsorbed into the body by the vasa recta, thus increasing the plasma volume of the
body.
There are two systems that create a hyperosmotic medulla and thus increase the body
plasma volume: Urea recycling and the 'single effect.'
Urea is usually excreted as a waste product from the kidneys. However, when plasma
blood volume is low and ADH is released the aquaporins that are opened are also
permeable to urea. This allows urea to leave the collecting duct into the medulla
creating a hyperosmotic solution that 'attracts' water. Urea can then re-enter the
nephron and be excreted or recycled again depending on whether ADH is still present
or not.
The 'Single effect' describes the fact that the ascending thick limb of the loop of Henle is
not permeable to water but is permeable to NaCl. This means that a countercurrent
system is created whereby the medulla becomes increasingly concentrated setting up
an osmotic gradient for water to follow should the aquaporins of the collecting duct be
opened by ADH.
Blood pressure regulation
Long-term regulation of blood pressure predominantly depends upon the kidney. This
primarily occurs through maintenance of theextracellular fluid compartment, the size of
which depends on the plasma sodium concentration. Although the kidney cannot
directly sense blood pressure, changes in the delivery of sodium and chloride to the
distal part of the nephron alter the kidney's secretion of the enzymerenin. When the
extracellular fluid compartment is expanded and blood pressure is high, the delivery of
these ions is increased and renin secretion is decreased. Similarly, when the
extracellular fluid compartment is contracted and blood pressure is low, sodium and
chloride delivery is decreased and renin secretion is increased in response.
Renin is the first in a series of important chemical messengers that comprise the renin-
angiotensin system. Changes in renin ultimately alter the output of this system,
principally the hormones angiotensin II and aldosterone. Each hormone acts via multiple
mechanisms, but both increase the kidney's absorption of sodium chloride, thereby
expanding the extracellular fluid compartment and raising blood pressure. When renin
levels are elevated, the concentrations of angiotensin II and aldosterone increase,
leading to increased sodium chloride reabsorption, expansion of the extracellular fluid
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compartment, and an increase in blood pressure. Conversely, when renin levels are
low, angiotensin II and aldosterone levels decrease, contracting the extracellular fluid
compartment, and decreasing blood pressure.
Hormone secretion
The kidneys secrete a variety of hormones, including erythropoietin, calcitriol,
and renin. Erythropoietin is released in response to hypoxia(low levels of oxygen at
tissue level) in the renal circulation. It stimulates erythropoiesis (production of red blood
cells) in the bone marrow.Calcitriol, the activated form of vitamin D, promotes intestinal
absorption of calcium and the renal reabsorption of phosphate. Part of the renin-
angiotensin-aldosterone system, renin is an enzyme involved in the regulation
of aldosterone levels.
Kidney StonesA kidney stone is a solid piece of material that forms in the kidney out of substances in the urine. Normally, urine contains chemicals that prevent or inhibit the crystals from forming. However, these inhibitors do not seem to work for everyone. Calcium (Oxalate or Phosphate)The most common kidney stone type contains calcium in combination with either oxalate or phosphate. These are called calcium oxalate stones or calcium phosphate kidney stones, respectively. Calcium is a normal part of a healthy diet and makes up important parts of the body, such as bones and muscles. Calcium that is not used by the bones and muscles goes to the kidneys. In most people, the kidneys flush out the extra calcium in the urine. However, when calcium stays in the kidneys, it joins with other waste products to form a calcium kidney stone. StruviteA struvite stone, also known as an infection stone, may form after there is an infection in the urinary system. This type of kidney stone contains the mineral magnesium and the waste product ammonia. Uric AcidA uric acid stone may form when there is too much acid in the urine. If you tend to form uric acid stones, you may need to cut back on the amount of meat that you eat.
CystineCystine is one of the building blocks that make up muscles, nerves, and other parts of the body. Although cystine stones are rare, they may form when there is too much cystine buildup in the urine. The disease that causes this kidney stone type runs in families.
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IV. PATHOPHYSIOLOGYA. Narrative Pathophysiology
The urinary calculi (Urolithiasis) are calcifications in the urinary system. There are two primary causative factors related to the formation of these stones such as urinary stasis and supersaturation of urine with poor soluble crystalloids. There are also 2 primary predisposing factors that are related to the case such as age and family history. Age is one factor because it is most common in adults age 40 and older, though kidney stones may occur at any age Family history is another predisposing factor because there is an increase of the disease associated in her paternal side. On the other side the precipitating factors are diet, low intake of water, lifestyle, metabolic disturbances, and urinary stasis. Diet is a factor because increase intake of purines, oxidates, calcium supplements and animal proteins can increase occurrence of the disease process. Low intake of water can increase solute concentration occurs because of fluid depletion or an increased solute load. This increased concentration leads to precipitation of crystals, such as calcium, uric acid, and phosphate. Another factor would be metabolic disturbances. It is because it influences the solubility of certain crystals, with some crystal types precipitating readily in acid urine and some in alkaline urine. Abnormal pH levels occur in renal tubular acidosis with the administration of carbonic anhydrase inhibitors, in the presence of urea-splitting bacteria, and in severe, chronic diarrhea. The last precipitating factor would be urine stasis. Stasis of urine from the bladder neck obstruction, continent urinary diversion, and immobilization increases the risk for development of stones because the crystals in unmoving urine precipitate more readily. It all starts with the uric acid, ammonia phosphate, and calcium oxalate stone material deposition on proximal renal tubule. There would then be supersaturation of urine by stone forming constituents and nephrocalcinosis on the proximal tubule. Due to the supersaturation of urine there would then be nidation of crystals of foreign bodies from the supersaturated urine. On the other side, Due to the nephrocalcinosis on the proximal tubule the patient then manifested the Randall’s plaque in relation to the low back flank pain. The stones then progress to the loop of henle and there would be more stones accumulating and would be increasing in size. An increase in stone would then attract itself to the blood vessel wall surface and would finally erode. The process progresses with the stone formation in the kidneys. Due to certain factors such as low intake of water and improper diet, the stone increases its size. Stone matrix progresses due to the mucoproteins that bind to the mass of the stone that forms multiple urinary calculus. On the other hand, due to the stone formation in the kidneys the stones that were accumulated would then descend from the kidneys to the ureter. Growth of these stones continues by aggregation to form larger particles. One of these particles may travel down the urinary tract until it is trapped at some narrow point where for stone formation occurs. It then increases the increases the pressure in the ureter that would then be severely stretched and would then loose its ability to undergo peristaltic contractions. Due to the urethral walls looses its ability to undergo contractions the urethral walls would then bleed and hematuria would again be manifested by the patient. There would also be slowed passage of urine due to the loosed contractions. Due to the bleeding of the urethral walls there would then be invasion and infection of urea splitting bacteria that had increased the production of white blood cells and was manifested with pyuria and fever. The continuous bleeding would result to the scarring
45
of the urethral walls and would then again increase the stasis of urine in the ureters. The increased stasis would then accumulate the urine substances and nucleation occurs, in which crystals are formed from super saturated urine. Due to these occurrences there would then be stone formation in the bladder and ureters that is manifested with decreased urine output. The process then proceeds with the destruction of normal function of the kidney. One the function of our kidneys is blood formation, due to the destruction of the normal functioning of the kidneys there would then be a decrease in production of erythropoietin. Due to this anemia occurs due to the decrease production of red blood cells including oxygen carrying components called hemoglobin.
(Was not manifested) the disease progresses to a more severe stage. Renal Failure occurs with the increased sympathetic nervous system activity related to dysfunction of autonomic nervous system. There would be an increased activity of renin angiotensin aldosterone system that increases the systemic vascular resistance. It would then also increase the renal absorption of sodium, chloride and water related to a genetic variation in the pathways by which kidneys can handle. There would then be increase in the viscosity of blood as well as increased cardiac workload and would then progress to cardiac failure and finally DEATH.
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V. MEDICAL MANAGEMENT
A. General Management
Ideal Management Actual Management
Intake and Output – to determine
any abnormalities of the function of
the urinary and digestive system.
Vital Signs Monitoring – to monitor
the progress of the patient’s
condition
IV Therapy- for hydration and
replacement of electrolytes to
maintain fluid and electrolyte
balance
Diet as tolerated- for the patient to
get the appropriate amount of
vitamins and minerals needed.
Ranitidine(500mg IVTT q 4 hours
RTC X 24)- treatment for post
surgical ulcer formation
Paracetamol(300mg IVTT q 4
hours RTC X 24) – treatment for
fever
Tramadol (500mg 5/100 IVTT q 6
hours RTC) – Treatment of
moderate or moderately severe
pain felt at the Surgical incision site
Metoclopramide(10mg IVTT stat)-
treatment for post operative
nausea and vomiting
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Diphenylhydramine(50mg 5/100
IVTT PRN)- treatment for allergic
reactions and to prevent motion
sickness
Cefuroxime(750mg IVTT q 8 ANS
(-))- perioperative prophylaxis
Oxygen administration via nasal
cannula- it is given to provide
sufficient oxygen for the body of
the patient.
Blood Transfusion- it is to supply
enough blood for the body due to
blood loss during operation.
Ideal Laboratory Examinations Actual Laboratory Examinations
1. Complete Blood Count
-Include hemoglobin and
hematocrit measurements,
erythrocyte (RBC) count, Leukocyte
(WBC) count, differential white cell
count.
-Basic screening test and one of
the most frequently ordered blood test.
-a common blood test that
evaluates the three major types of
cells in the blood: red blood cells,
white blood cells, and platelets.
-ordered as part of a routine
check-up, or if you are feeling more
tired than usual, seems to have an
1. Complete Blood Count
- Used to determine infection and other diseases. It is used to determine if there are any bleeding problems.
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infection, or has unexplained bruising
or bleeding.
2. Urinalysis
-Ordered to diagnose a number
of medical conditions including
diabetes, urinary tract infections, and
kidney diseases. This is done to detect
any cells, urine crystals, mucus, and
other substances, and to identify any
bacteria or other microorganisms that
might be present.
3. Creatinine levels – to assess for
renal function
2. Urinalysis
-used to determine if there are infections, bleeding and disease.
Ideal Imaging Studies Actual Imaging Studies
1. Chest X-Ray
-used to visualize the lung fields
to check for any scarring or
accumulation of fluid in the lungs.
2. Intravenous Urogram
-The IVU is an x-ray procedure using contrast medium that shows the size,
placement and function of the parts of the renal tract, which is made up of the kidneys, the ureters (tubes from the kidneys to the bladder) and the bladder.
This test is sometimes used to locate the site and identify the underlying problem when the flow of urine is obstructed. IVU can also show abnormal connections between the urinary tract and the skin or other organs.
3. Ultrasonography of kidney, ureter
and bladder
- high-frequency sound waves
are
transmitted from a transducer to the
kidneys and perirenal structures. The
resulting echoes are displayed on a
monitor as anatomic images. Renal
ultrasonography can be used to detect
abnormalities or clarify those detected
by other tests. It's especially useful in
cases in which excretory urography is
ruled out. Unlike excretory urography,
this test isn't dependent on renal
function and therefore may be useful in
patients with renal failure.
cause of the obstruction of the flow in the urinary tract.
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Ultrasonography of the ureter, bladder,
and gonads also may be used to
evaluate urologic disorders.
Ideal Pharmacotherapy Actual Pharmacotherapy
1.Nonopioid Analgesic
-drugs that have principally analgesic,
antipyretic, and anti-inflammatory
actions. They are milder forms of
analgesics.
2.Thiazide diuretic
-promotes calcium resorption from the
renal tubules, thereby preventing
excess calcium loads in the urine.
3. Vitamin B6(pyridoxine), magnesium
oxide, or cholestyramine.
- for calcium oxalate stones treatment
1.Ranitidine(500mg IVTT q 4 hours RTC
X 24)- treatment for post surgical ulcer
formation
2.Paracetamol(300mg IVTT q 4 hours
RTC X 24) – treatment for fever
3.Tramadol (500mg 5/100 IVTT q 6 hours
RTC) – Treatment of moderate or
moderately severe pain felt at the CTT
insertion site
4.Metoclopramide(10mg IVTT stat)-
treatment for post operative nausea and
vomiting
5.Diphenylhydramine(50mg 5/100 IVTT
PRN)- treatment for allergic reactions and
to prevent motion sickness
6.Cefuroxime(750mg IVTT q 8 ANS (-))-
perioperative prophylaxis.
Ideal Surgical Management Actual Surgical Management
1. Shock Wave Lithotripsy- It may be performed on an outpatient basis, either with intravenous sedation or general anesthesia, and the duration of treatment is usually less than one hour. Recent advances have been made in the
1. Nephrectomy - The surgery done was nephrectomy to remove the whole non functioning right kidney which had kidney stones made up with calcium.
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understanding of stone fragmentation and the etiology of renal injury which have made SWL more effective and safer. Small variations in the technique with which SWL is applied can affect optimal treatment outcomes. For example, a recent literature review and meta-analysis have found that slowing the rate of SWL delivery to 60 shock waves per minute fragments stones more effectively than treatment at a rate of 120 shock waves per minute.
2. Laser Lithotripsy-can be used to fragment stones in the bladder, ureter or kidney via a laser fiber which is passed through an endoscope [Figure 2]. Typically, a Holmium:YAG laser is used, which operates in the near infra-red area of the electromagnetic spectrum with a wavelength of approximately 2140 nanometers. The Holmium laser is one of the safest intracorporeal lithotripsies available for stone fragmentation. The depth of tissue penetration of the laser light is 0.5 to 1.0 mm, and the laser may be used in patients receiving anti-coagulation medications. Typical fiber diameter sizes for ureteroscopy
55
range from 200-400 microns. The rigidity of the fibers can limit the degree of deflection of the flexible ureteroscope, making the treatment of stones in the lower pole of the kidney especially challenging. The upper limit of stone size that can be efficiently treated with laser lithotripsy is approximately 1.5cm. Laser lithotripsy is usually performed as an outpatient procedure with the patient receiving a general anaesthetic. The operating room time varies based on the stone burden being treated and the surgical approach employed, but typically it lasts one to two hours.
3. Rigid Lithotrites- is a minimally invasive treatment alternative to open surgery for patients with large or complex renal calculi. PNL is performed in the operating room using general anesthesia, with the patient typically in the prone position, although supine PNL has been described. A rigid nephroscope is placed into the collecting system of the kidney via a small incision in the flank. Intracorporeal lithotripsy is then performed by inserting a rigid lithotrite through the endoscope and placing it in contact with the
56
stone. Lithotripters using ultrasonic energy are most commonly utilized during PNL and can efficiently fragment and remove the majority of stone types. Continuous suction irrigation is used to evacuate stone fragments quickly and to maintain clear visibility. However, hard calculi such as cystine and calcium oxalate monohydrate stones are less efficiently fragmented and removed with ultrasonic technology. For these stones, pneumatic lithotripters are often used for fragmentation as these devices readily break stones of any composition. The chief disadvantage of pneumatic lithotripters, however, is their inability to concurrently evacuate stone debris while fragmenting the stone. Rather, manual fragment removal is needed by using stone graspers, which may be a time-consuming process.
4. Nephrectomy -The surgery is performed with the patient under general anesthesia. The surgeon makes an incision in the side of the abdomen to reach the kidney. Depending on circumstances, the incision can also be made midline. The ureter and blood vessels are disconnected, and the kidney is then removed. The
RANITIDINE 500mg IVTT q 4 hours RTC x 24Classification: histamine 2 receptor blocking drugIndication Mechanism of action Contraindication Side effects Nursing precaution
Post surgery antacid to prevent ulcer formation
Competitively inhibits gastric acid secretion by blocking the effect of histamine on histamine 2 receptors.
Cirrhosis of the liver, impaired renal or hepatic function
Headache, abdominal pain, constipation, diarrhea, nausea and vomiting
Tell the patient that he may experience side effects brought about by the drug and if such are intolerable he must report them
Oral care if vomiting occurs
Monitor patient continually if giving IV medications
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PARACETAMOL 300mg IVTT q 4 hours RTC X 24Classification: acetaminophen, anti pyretic and analgesicIndication Mechanism of action Contraindication Side effects Nursing precaution
Fever Inhibits prostaglandins in CNS, but lacks anti-inflammatory effects in periphery; reduces fever through direct action on hypothalamic heat regulating center
Renal insufficiency, anemia, liver failure. Clients with pulmonary and cardiac diseases are more susceptible to acetaminophen toxicity
Advise patient to take medication exactly as directed and not to take more than recommended amount.
Avoid alcohol intake.
Caution patient to check the label in the OTC drugs. Advise to avoid taking more than one product containing acetaminophen at a time to prevent toxicity.
Advise patient to consult health care professional if discomfort or fever is not relieved by routine doses of this drug.
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METOCLOPRAMIDE 10mg IVTT statClassification: Anti emeticIndication Mechanism of Action Contraindication Side Effects Nursing Precaution
Treatment for post operative nausea and vomiting when nasogastric tube is undesirable
Blocks dopamine receptors in the chemoreceptor trigger zone of the CNS. Stimulates motility of the upper GI tract and accelerates gastric emptying.
Hypersensitivity; possible GI obstruction or hemorrhage; history of seizure disorder; Parkinson’s disease
Dose may be given slowly over 1-2 min. Rapid administration causes transient but intense feeling of anxiety and restlessness followed by drowsiness advise patient to notify health care professional immediately if involuntary movements of eyes, face, or limbs occursadvise patient to avoid concurrent use of alcohol and other CNS depressants while taking this medication May cause drowsiness. Caution patient to avoid driving or other activities requiring alertness until response to medication is known.
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DIPHENYLHYDRAMINE 50mg 5/100 IVTT PRNClassification: antihistamine Indication Mechanism of
ActionContraindication Side Effects Nursing Precaution
Relief of allergic symptoms caused by histamine release. Prevention of motion sickness.
Antagonizes the effects of histamine at H21 receptor sites; does not bind to or inactivate histamine. Significant CNS depressant and anti cholinergic properties.
Hypersensitivity, acute attacks of asthma; lactation; known alcohol intolerance
May cause drowsiness. Caution patient to avoid driving or any activities which requires alertness until response of drug is known.
Perform oral care since it can cause dry mouth. Frequent oral rinses and candies can decrease dryness.
Advise patient to avoid use of alcohol and other CNS depressants concurrently with this medication.
Instruct patient taking diphenylhydamine in OTC preparation to notify health care professional if symptoms persist after 7 days.
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TRAMADOL 500mg 5/100 IVTT q 6 hours RTCClassification: analgesicIndication Mechanisms of actions Contraindication Side effects Nursing precaution
Moderate to moderate severe pain
Binds to mu opioid receptors. Inhibits re uptake of serotonin and norepinephrine in the CNS
Hypersensitivity; patients who are acutely intoxicated with alcohol, sedatives, hypnotics and centrally acting analgesics; pregnancy and lactation; severe hepatic impairment
May cause dizziness and drowsiness. Caution patient to avoid driving or any activities which requires alertness until response to drug is known.
Advise patient to change positions slowly to minimize orthostatic hypotension.
Caution patient to avoid concurrent use of alcohol and other CNS depressants with this medication.
Encourage patient to turn, cough and breathe deeply every 2 hours to prevent atelectasis.
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CEFUROXIME 750mg IVTT q 8 ANS (-)Classification: anti infective, second generation cephalosporinsIndication Mechanism of action Contraindication Side effects Nursing precaution
Perioperative prophylaxis
Binds to cell wall membrane causing cell death. Bactericidal action to susceptible bacteria.
Hypersensitivity to cephalosporins; serious hypersensitivity to penicillins
Seizures, diarrhea, cramps, nausea, vomiting, rashes, phlebitis at IV site, bleeding and neutropenia
Advise patient to report signs of superinfections( furry overgrowth on the tongue and vaginal itching) and allergies.Instruct patient to notify health care professional if ever fever and diarrhea develop, especially if stool contains blood, pus and mucus. Advise patient not to treat diarrhea without consulting health care profession.
“ Gasakit akong likod ( pointing at the surgical sight)”, as verbalized by the patient
Objective: Pain scale of 8/10 Grimacing Guarding behavior Requesting help while
walking
Acute pain related to surgical incision secondary to surgical procedure
SHORT TERMAfter 3 minutes of nursing interventions the patient will be able to
Report alleviation of pain with a pain scale from 8/10 to 3/10 as evi.denced by absence of grimed face and guarding behavior.
After 30 minutes of nursing interventions the patient will be able to
Demonstrate the use of relaxation skills and diversional activities as indicated such as: deep breathing techniques and imagery technique.
Demonstrate control of pain, feel more relaxed and interact more openly.
1. Instruct bed rest, allowing patient to assume comfortable position
2. Guide and instruct patient in relaxation technique such as deep breathing
3. Provide comfort measures such as therapeutic touch and communication
4. Teach the patient to socialize with family and SNs
5. Provide adequate rest periods
6. Eliminate additional stressors or sources of discomfort by providing quiet and calm environment
7. Assist in position changes
1. To promote rest and reduce fatigue
2. To promote generalized relaxation and to decrease sensation of pain
3. To promote nonpharmacological pain management
4. Diversional activities promote generalized relaxation and to decrease sensation of pain
5. To prevent fatigue6. To promote
nonpharmacological pain management
7. To prevent development of pressure sores
Short term goals were met
Patient was able to report pain was minimized; participate in the plan of care; feel more relaxed and interact more openly.
Patient was able to demonstrate a decrease in pain from 8/10 to 3/10 as evidenced by the absence of grimed face and guarding behavior.
Long term goals that were partially met:
1. Patient was able to report pain was relieved
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LONG TERM
Upon discharge the patient will be able to:
Experience absence of pain on surgical site
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Nursing Care Plan 2Cues Diagnosis Planning Interventions Rationale Evaluation
Sujective:
Pain: “motukar ra man ang sakit, mga 15-30min”
Location: “sa iyang likud dapit daw”
“sakit jud siya nga mangluya si mama”
Associated s/s: “lantan, takig, gainit ang tiyan”
Itchiness “katol kayo ako samad pero dili nako ginakamot, ginahikap-hikap lang nako sa kilid”
Objective: Pain scale: 8 out of scale
of 1-10 Pain description: sharp Right flank pain noted Surgical incision noted on
right lower back area
Impaired Skin Integrity related to surgical incision
Short Term:Within 16 hours, client will be able to:
1. Verbalize measures in preventing infection correctly.
2. Demonstrate proper wound care and dressing.
3. Demonstrate improved behaviour like diet modification and early mobilization.
4. Verbalize importance of following therapeutic plan.
Independent1. Note changes in skin color,
texture, and turgor. Assess areas of least pigmentation for color changes like sclera, conjunctiva, nailbeds, buccal mucosa, tongue, palms, soles of feet
4. Inspect skin on a daily basis, describing changes observed including remeasuring the wound.
5. Instruct patient to keep area clean and dry, demonstrating how to dress wounds aseptically, and support the incision ( ex. Splinting when coughing), prevent infection, and stimulate circulation to surrounding areas.
6. Use appropriate barrier dressings, wound coverings, drainage appliances, and skin-protective agents for open/draining wounds
7. Instruct SOs in monitoring for
Assess for abnormalities that may affect patient well-being
Assess for abnormalities that may affect patient well-being
Assess for abnormalities that may affect patient well-being
To monitor progress of wound healing.
Assists body’s natural process of repair
To protect wound and surrounding tissues
Moisture potentiates skin breakdown
After intervention, client was able to:
2. Verbalize and demonstrate proper infection prevention measures and skin care.
3. Demonstrate wound and dressing care properly.
4. Modify diet to increase in oral fluid intake and protein-rich balanced diet.
5. Display clean, non-infected and non-complicated wound.
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moist or wet linens and replace them with clean and dry ones.
8. Reposition patient every 2 hours involving client through client education and on desired positions in conjuction to other activities.
9. Encourage early ambulation
10. Encourage adequate nutritional intake and increase in fluids and protein intake.
11. Discuss importance of early detection on skin changes and complications.
12. Discuss the medical management and preventive care measures to client and SO
13. Reinforce measures in avoiding infection like proper wound care, avoiding moisture in wound site etc.
Dependent:8. Give Cefuroxime 750mg
IVTT q8h ANST (-)RTC per doctor’s order
9. Give Tramadol 500mg 5/100 IVTT q6h RTC per doctor’s order
Enhances understanding and cooperation
Promotes circulation and reduces risks associated with immobility.
To provide positive nitrogen balance to aid in healing and to maintain general good health
To monitor abnormalities and promote cooperation
To promote understanding, commitment to plan of therapy and optimizing outcomes
Reinforce prevention of infection and encourage cooperation
Short TermWithin 16 hrs duty @ NMMC, client will be able to:
1. Identify risk factors for occurrence of infection correctly.
2. Identify and demonstrate correctly the interventions that prevent/reduce risk of infection.
Long Term1. After 16hours of
duty at NMMC, client will be able to demonstrate lifestyle changes to promote safe environment like proper wound care and dressing, proper hygiene and hand washing, and intake of balanced diet.
INDEPENDENT1. Discuss different risk factors
that may contribute to infection and its prevention measures.
2. Instruct patient to keep area clean and dry, demonstrating how to dress wounds aseptically, and support the incision ( ex. Splinting when coughing), prevent infection, and stimulate circulation to surrounding areas.
3. Use appropriate barrier dressings, wound coverings, drainage appliances, and skin-protective agents for open/draining wounds
4. Demonstrate tepid sponge bath for fever
5. Instruct SOs in monitoring for moist or wet linens and replace them with clean and dry ones.
6. Reposition patient every 2
To broaden client knowledge on risk factors causing infection thus promoting cooperation on prevention measures through self monitoring and change in unhealthy habits
Assists body’s natural process of repair
To protect wound and surrounding tissues
Relieve fever and promote comfort from heat and
Improve circulation Moisture potentiates skin
breakdown
Enhances understanding and cooperation
After nursing interventions, client was able to:
1. Be free of cough, and fever by v/s within ranges of: T=36.5-3C, P=60-100bpm,R=1-20cpm,BP between 90/60mmHg and 140/90mmHg.
2. demonstrate deep breathing and coughing exercises correctly.
3. demonstrate proper hand washing and tepid sponge bath, proper wound and dressing care.
4. Performs early ambulation and frequent
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hours involving client through client education and on desired positions in conjuction to other activities.
7. Encourage early ambulation
8. Encourage adequate nutritional intake and increase in fluids and protein intake.
9. Discuss importance of early detection of skin changes and complications.
10. reinforce health teachings on keeping wound and dressing clean and dry, proper wound splinting, proper hand washing, frequent change in positions and adequate intake of nutritional foods with increase proteins and fluids
11.Assess and document skin conditions, noting inflammation and drainage.
Promotes circulation and reduces risks associated with immobility.
To provide positive nitrogen balance to aid in healing and to maintain general good health
To monitor abnormalities and promote cooperation and understanding, commitment to plan of therapy and optimizing outcomes
Encourage self-care and reinforce importance of non-pharmacologic intervention on infection prevention
Can be a source of infection and may reveal infection (drainage, inflammation), monitor for early signs of infection and early management and prevention of condition
Informing the client and SO increases self-esteem on
changes in position as tolerated.
5. Performs correct wound splinting.
6. Increase in oral fluid and protein-rich food intake, and intake of balanced diet.
7. Identify risk factors for occurrence of infection correctly.
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12.Note signs and symptoms of sepsis (systemic infection): fever, chills, diaphoresis altered level of consciousness, positive blood cultures. Then instruct client and SO to monitor for s/s of: fever, chills, diaphoresis and altered level of consciousness like confusion.
13.Maintenance of sterile technique in performing invasive procedures.
14.Encouraging deep breathing, coughing exercises, position change.
15.Maintain adequate hydration, void frequently
16.Perform regular perineal care and proper drying
17.Emphasize importance of taking antibiotics as directed (e.x dosage, therapy length)
Dependent:10.Give Paracetamol 300mg
IVTT q4h RTC x 24 per
having role in care and increases cooperation in therapy from gained knowledge on monitoring Signs of systemic infection, knowledge by client and SO aids in improve nursing care and monitoring
To prevent spread of microorganisms, prevention on cross-contamination and defense against nosocomial infection
For mobilization of respiratory secretions.
To avoid UTI
To reduce UTI risk cause by moisture and poor hygiene.
Premature discontinuation of treatment when client begins to feel well may result to infection
To relieve fever
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doctor’s order11.Give Cefuroxime 750mg
IVTT q8h ANST (-)RTC per doctor’s order
To treat infection
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VII. PROGNOSIS
CRITERIA:
Good Poor
A. Response of the patient regarding the presence of the
pain after its management
B. Physiologic response of the body to disease process
C. Relief of symptoms associated with the disease
condition
D. Performance of the daily living of the patient during
E. Compliance of the patient to the medication and/ or
therapy
F. Adequacy of rest periods and sleep
G. Consumption of the patient with nutrition
H. Patient’s significant others’ behaviour regarding the
health teaching given by the health caregiver and the
physician
I. Attitude
J. Duration of Illness
K. Precipitating Factor
L. Nature of Problems
M. Predisposing Factors
N. Family Support
O. Level of Consciousness
CALCULATIONS:
Formula: amount # of (good/poor) x 100 = % (Percentile)
15
Amount of: Percentile
Good = 12 80%
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Poor = 3 20%
INTERPRETATION:
Since, patient is cooperative and its precipitating factors are modifiable, patient’s condition may have a good prognosis. Based on the criteria, 12 out of 15 or 80% accounts for good prognosis,3 out of 15 or 20% accounts for poor prognosis which shows that the patient is compliant to her medications and will have a good recovery.
VIII. DISCHARGE PLANM- Medication Instruct patient to follow right dose and timing of medication
and not to replace any medication without consultation to physician.
Report any adverse effects and drug/food-drug interactions to the physician.
Instruct patient to follow orders for take home medications upon discharge as prescribed by the physician.
E- Exercise Assist significant others on how to help the patient maintain physical activity, an exercise as well as how to prioritize activities and establish a balance between activities.
Perform exercises and activities tolerated by client.Encourage patient to do active range of motion such as rotating and
flexion of arms and legs.Instruct significant others to be watchful for any risk that may cause
injury to the patient.T- Treatment Perform proper wound care by maintaining aseptic technique to
prevent infection.Keep dressing intact and avoid wound exposure to prevent unwanted
harboring of microorganisms.Strict medication regimen.Discuss drug therapy to the patient and significant others.Instruct patient to keep updated with any follow-up
examinations/therapy/check-ups after discharge.Assure patient and significant others that they will be able to perform
home-based treatment properly. H- Health Teaching Indicate enough bed rest
Explain importance of medication complianceDiscuss to the patient and significant others the indication of the
prescribed medications, outcomes, dosages, contraindications and side effect.
Wash hands before and after performing wound care.O- Out-Patient; Follow-up
Inform the patient about when, where and what time is the next check-up. The next check-up is usually one week after discharge.
Instruct patient and significant others to report to the physician and/or health care personnel when unusualities are noted.
Stress out to the family to seek immediate consultation if adverse reaction of drugs occurs.
74
Encourage them to carry out follow up diagnostic regimen.D- Diet Eat meals 3 times a day.
Discuss importance of taking vitamins and minerals.Instruct patient to ensure increase fluid intake at least 8 to 10 glasses
per day.S- Spirituality Encourage her to develop both the trust and faith in God.
Encourage prayers to the client in order for him to express his thoughts, endeavors, and feelings according to her beliefs.
IX. CONCLUSION
The proponents of this case study have gathered all relevant details
regarding the case herein about a 64 year old female diagnosed of
Nonfunctioning kidney secondary to urolithiasis and nephrolithiasis ,
admitted at NMMC. The health team involved in the case provided in conjunction
to the medical treatments, overall nursing management set to alleviate the
client’s condition and support the patient’s over all well being. The necessary
health teachings were also given to the patient and her watcher in order to
alleviate the signs and symptoms of the disease condition.
Implementation of relevant and effective nursing interventions to relieve
signs and symptoms were strictly observed so as to prevent or at least reduce
the risks of complications.
From what the group can surmise however the limitations on the
effectiveness of the provided nursing interventions rest on the grounds of the
presence of irreversible and inalterable factors such as advanced age and
preexisting medical conditions.
With this subsequent information, the persons involved in this study have
realized the significance of this case, its management and its overall impact on
affected clients. In line with this, the proponents worked hand in hand with each
other to help convey what has been researched and planned about the case.
X. RECOMMENDATION
In nursing practice, we recommend student nurses to regularly check and
monitor closely patient’s vital signs especially with patients like NQ that have
advanced age and are diagnosed with other medical conditions which may
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aggravate the disorder, over all condition of the patient and prompt adherence to
safety precautions considering that the client is at risk for injury. Also follow up
patient’s medication supply for prompt timing of administration.
It is recommended for the healthcare team to:
Participate the client, family, significant others and members of the healthcare
team to establish client-centered goals directed toward promoting and restoring
the clients optimum state of health, preventing illness and providing rehabilitation.
Support client’s and family’s decisions regarding care.
Promote an environment conducive to maintenance or restoration of the client’s
ability to carry out activities of daily living.
Provide for continuity of care in the management of the disorder
Demonstrate caring behaviors in providing nursing care.
Assist other personnel to develop skills in providing nursing care.
Manage an environment that promotes client’s self-esteem, dignity, safety, and
comfort.
Student nurses and other health care providers should read further
number of books regarding Nonfunctioning kidney secondary to
urolithiasis and nephrolithiasis, be updated about the disease and
incorporate it with desired plan of care. This is to prevent patients with
Nonfunctioning kidney secondary to urolithiasis and nephrolithiasis from
developing severe complications, preserve patients’ health and promote
wellness.
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XI. BIBLIOGRAPHY
77
XII. APPENDIXA. Doctors Order
12/13/20106am >to operating room on call12/13/2010 post-op order4pm >to PACU S/P Nephrectomy right under EA-GETA
>monitor V/S q15mins until stable then q1hour>NPO>O2 inhalation @ 5LPM via face mask>IVF: PNSS 1L @ 30gtts/min>IVTTF: PNSS 1L@ 30gtts/min
>Meds: Cefuroxime 750mg IV q8hours (-)ANSTRanitidine 500mg IV q8hoursMetoclopromide 10g IV PRN for vomitingParacetamol 300mg IV q6 RTC starting tomorrow 6amDiphenhydramine 50mg slow IV push for pruritus
>I & O q1 refer if < 50cc/hr>Morphine precaution, offer if BP <90/60, HR < 50bpm, RR < 12cpm>watch out transfusion reaction>suction secretions PRN>refer accordingly>thank you
5:55PMBP: 130/90 >May transport pt. to wardHR: 110RR: 20O2 Sat: 96%12/14/20107:30 am >pt. CBC now(-) fever >change dressing
>continue IVF and meds>Change dressings>V/S q 4hours>I & O q shift>refer accordingly.
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B. NURSES NOTES
12/12/10 3-113 pmD> received on bed
>Scheduled for Nephrectomy Right on Dec . 13, 2010 A > Vital signs taken and recorded
>Intake and output measured and recordedDAT >Served and consumed share with fair appetite
>encouraged verbalization of feelings of discomfort>encouraged relaxation technique and deep breathing exercises>adequate rest periods provided>kept watched for any unusualities>needs attended
R >on bed resting>endorsed
12- 12-10 11-711pm D > Received asleep on bed
>Poor eye contact A > Instructed to do deep breathing exercises
>encouraged to verbalize feelingsNPO >instructed and maintined
>for nephrectomy right on 12-13-10 with consent> Morning care done> Hemodialysis started with D5LR regulated at 30gtts/min
R > on bed resting>off to OR on Call
12-13-10 7-3 D > Received on bed
>with D5LR 1L @ 900cc level regulated @ 30gtts/min A >V/S taken and recorded
>visited by Dr.E with orders carried out by NOD> for Nephrectomy Right today with consent signed.
NPO >maintained>still to secure blood for OR use>I&O measured and recorded>kept watched for any unusualities>to OR on call
11:00am> brought to OR per stretcher with above IVF going on12/13/10 7-3 OR notes12:15pm>received from ward per stretcher, awake, conscious and coherent with IVF going on D5LR 1L
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100cc level infusing well on Left arm.>consent signed for nephrectomy>ashered to operating table safely and comfortably in supine position>hooked to monitor with initial V/S of BP:140/70mmHg,PR:90bpm,RR:20cpm,SP O2:100%
12:30 >oxygenation given via face mask>with IVF D5LR 1L consumed and followed up D5LR 1L infusing well.
12:35 >Epidural anesthesia inducted by Dr.L12:40 >general anesthesia inducted by Dr.L
>with IVF D5LR consumed and followed-up with Volvulen 500cc infusing well @ left arm
12:45 >Endotracheal tube inserted>foley catheter French 14 inserted aseptically and attached to urobag draining well.>placed on Left Lateral position with support in placed>final Skin Prep done aseptically>draped accordingly>counting of sponges, instruments and needles done and recorded.
1:25 pm>operation started by Dr.L assisted by Dr.M and Dr.L>bleeding clamped and cauterized
1:45pm>with IVF of Volvulen 500cc consumed and followed-up with PNSS 1Linfusing well on Left arm2:40pm>blood transfusion started @ pts blod type A+ with serial no. 2K10-12-7912 segment
no.648X7600 as side drip.>endorsed to 3-11 shift nurse with complete counting of sponges and instruments
12/13/10 OR notes 3-113:00pm >received on OR table on Left Lateral position ongoing operation in general anesthesia
>with IV fluid of PNSS 1L @ 500cc level regulated @ KVO rate>with side drip of ongoing blood transfusion of 1”U” PRBC with Serial no. 2K10-12-7912 blood type “A” regulated @ 30gtts/min>with endotracheal tube attached to 4LPMO2>with epidural catheter in place>With foley catheter attached to urobag infusing well
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>initial counting of sponges, sharps, and instruments complete – surgeon informed>suturing done – by layer>operation ended>epidural cath rulled out by Dr.L
4:35 >extubated>post-op wound dressing-applied>post-op care done
4:43 >transported to PACU with same IVF ongoing; with ongoing blood transfusion>with foley catheter>with post-op wound dressing applied>endorsed to PACU NOD with complete documents attached.
3-11 PACU nurses notes
4:40pm >received from OR per stretcher, conscious and coherent, not in respiratory distress, with PNSS
1L @ 10gtts/min @30cc level @ Left arm infusing well.>V/S checked and recorded>BP monitored and recorded>status post nephrectomy Right under general anesthesia>O2 inhalation @ 6L/min, face mask administered>hooked to O2 Sat monitor>with post-op wound dressing dry and intact>kept flat on bed, safely and comfortably>With foley catheter to urobag drainage with Pus>post anesthetic care done
NPO >maintained>with PRBC “U” type “A+” with serial #2k10-12-7912 segment #698Y7660 transported after proper crossmatching and blood typing @ 30gtts/min going on>endorsed to A2F1
OR memo OR surgical safety checklist Sponge count Anesthesia record PACU monitoring
12/14/10 7-37:15amD>received awake from PACU per stretcher accompanied by relatives
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>with post operative wound @ right flank >with ongoing IVF of D5LR @ KVO rate infusing well >with ongoing blood transfusion of PRBC 1 unit A+ @ 20cc level with serial
#2k10-12-7912 on side drip of IVF
>with foley catheter attached to urobag draining well with yellow colored urine A>ushered to bed safely and comfortably
>vital signs taken>above blood product consumed and followed up with PRBC 1 unit with serial
number 2K10-12 1884 segment #48456913 regulated @ 30gtts/min infusing well >observed for early transfusion reaction
NPO >maintained>kept dry and comfortable>endorsed resting in bed with ongoing blood transfusion
Dec 14, 2010 3-11 shift3:00pmD> Received awake lying on bed with #3 D5LR @ 30gtts/min @ 800 cc
>with postoperative dressing on Right flank clean and dry A >needs attendedClear Liquids> maintained
> turned to sides, back kept dry>kept rested>observed for unusualities>encouraged ambulation
R >well rested
11-711pm D > Received asleep on bed
>with IVF of D5LR I L @ 850 cc level regulated @ 30 gtts/min>with post-op wound
A>instructed to keep post op dressing dry and intact @ all timesClear liquids >liquid in moderate amount with aspiration precaution
>nursing care done>on bed resting>observed
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C. Intake and Output / Vital Signs
DATE TIME BP PR RR TEMP. Intake IVF OutputDec. 11, 2010