Book Reading-Heart Disea se Braunwald Chapter 4 Physical Examination of the Heart and Circulation (I) Presenter R4 吳吳吳 Superviser P 吳吳吳
Dec 22, 2015
Book Reading-Heart DiseaseBraunwald
Chapter 4 Physical Examination of the Heart and Circulation (I)
Presenter R4 吳明昇Superviser P 蔡良敏
The General PE
• General appearance—skin color, truncal obesity, long extremities
• Respiration—orthopnea, Cheyne-Stokes (periodic), JVE
• Position– sit quietly(angina), sitting upright (CHF), moving about(AMI), leaning forwards (pericarditis)
Head and Face
• Expressionless face, periorbital puffiness, loss of lateral eyebrows, large tongue and dry sparse hair Myxedema
• Ear lobe crease frequent in CAD • De Musset sign (bobbing of head with each
heart beat) severe AR• Facial edemaTV disease or constrictive p
ericarditis
Eyes
• External ophthalmoplegia and ptosis Kearns-Sayre syndrome complete AV block
• Exophthalmos and starehyperthyroidism cause of high CO heart failure
• Blue scleraosteogenesis imperfecta aortic dilatation, AR, dissection and MVP
• Pulsation of eyeball or earlobe (Pulsatile exophthalmos) severe TR
Eye Fundi
• HTN• Infective endocarditis Roth spots• Papilledema malignant HTN and cor pul
monale with severe hypoxia• Hypercholesterolemia beading of retinal
artery• Embolic retinal occlusion RHD, LA myx
oma, atherosclerosis of aorta
Skin and Mucous membranes
• Central cyanosis R to L shunt• Peripheral cyanosis CHF and PAOD• Bronze pigmentation of skin and loss of axillary a
nd pubic hair hemochomatosis cause of cardiomyopathy
• Jaundice Pulmonary infarction, congestive hepatomegaly, cardiac cirrhosis
• Lentigines PS or HCM
Skin and Mucous membranes
• Xanthoma over sc or tendon suspect hyperlipoproteinemia cause of premature atherosclerosis
• Hereditary telangiectases (skin, mucosa, GI tract and airway) of lung cause of R to L shunt
Extremities
• ArachnodactylyMarfan syndrome• Systolic flushing of nail bedsQuincke sign A
R(widened pulse pressure)• Clubbing of fingers and toescentral cyanosisc
yanotic heart or hypoxic pulmonary disease• Unilateral clubbing aortic aneurysm• Differential cyanosis PDA with reverse shunt• Osler nodes, Janeway lesions, splinter hemorrhage
IE• Edema, bilateral or unilateral
Chest
• Barrel-shaped chest suspect emphysema, chronic bronchitis and cor pulmonale
• Bulging of right upper sternum aortic aneurysm• Pectus excavatum (Funnel chest) or pectus carinat
um (Pigeon chest) Marfan syn.• Kyphoscoliosis induce cor pulmonale• Rales and wheezing BS pulmonary edema
Abdomen
• Painful hepatomegaly due to right heart failure hepatojugular reflex
• Pulsation over liver severe TR or constrictive pericarditis
• Palpable kidney suspect polycystic kidney disease cause of HTN
• Systolic bruit over umbilicus or flank renovascular HTN
• Aortic aneurysm palpable below umbilicus
Jugular Venous Pulse(internal jugular vein)
• It was evaluated in 45 degree position• Upper normal limit 4cm above sternal angle (9
cm CVP)• Abdominal-jugular reflex press periumbilical area for 10-30 s normal < 3cm elevation and only transiently Abnormal right heart failure or TR, if not elevated PAWP or CVP
Jugular Venous Pulse(internal jugular vein)
Jugular Venous Pulse(internal jugular vein)
• Kussmaul signparadoxical rise in JVP during inspiration constrictive pericarditis and sometimes in CHF and TS
• Prominent a wave RVH, pulmonary hypertention and TS
• Cannon a wave AV dissociaton• Absent a wave atrial fribrillation• A steeply rising H wave restrictive cardiomyop
athy, constrictive pericarditis, RV infarction
Jugular Venous Pulse(internal jugular vein)
• Rapid and deep y with rapid rise to H wave ( W-shaped) constrictive pericarditis
• Prominent X descent cardiac tamponade
• Prominent c-v waveTR
• Equal a and v wave ASD
Arterial Pulse
• Carotid arterymost accurate representation of central aortic pulse
• Brachial arterymost suitable for evaluating the rate of rise of pulse, contour, volume, and consistency
Normal Arterial Pulse
Abnormal Arterial Pulse
• Bisferiens pulse: AR, AR + AS, HCOM• Dicrotic pulse: cardiac tamponade, severe HF, hyp
ovolemia shock• Pulsus alternans(alternate > 20mmHg) LV failu
re • Pulsus bigeminusVPC related • Pulsus paradoxus cardiac tamponade, emphyse
ma, asthma, hypovolemic shock, pulmonary embolism
• Pulsus tardus slow upstroke• Pulsus parvuslow amplitude
Abnormal Arterial Pulse
AR-widen pulse pressure
• Corrigan or Water-hammer pulse• Pistol shot sound (Traube sign): systolic murmur• Duroziez sign: diastolic murmur• Quincke sign• Hill sign: SBP in low ex- arm > 20mmHg• Becker sign: visible pulsation in retina• Mueller sign: pulsating uvula
Arterial Pulse in Vascular disease
• Normal aorta is palpable above umbilicus
• A palpable aorta below umbilicus suspect aortic aneurysm
• Absent dorsalis pedis and posterior tibial artery 2% normal aberrant course
• 50% stenosis artery bruits
The Cardiac ExaminationInspection
• Respiration pattern• Collateral vein• Pectus excavatum (funnel chest): Marfan sy
n., homocystinuria, Ehlers-Danlos syn., Hunter-Hurler syn., MVP.
• Cardiac pulsation thrusting apex >2cmLV enlarge lateral to midclavicular lineLV enlarge
The Cardiac ExaminationPalpation
• In 30 degree, supine and lateral decubitus position• Left Ventricle Apical thrust (PMI) >10cm from the midsternal line or >3cm in diamet
er LV enlargement Double systolic outward thrust HCOM Systolic retraction of chest (Broadbent sign) co
nstrictive pericarditis Presystolic expansion reduced LV compliance
(accompany with S4)
The Cardiac ExaminationPalpation
• Right ventricle
palpable systolic movement in left parasternal area RVH or enlargement
Thrills accompany with load harsh low to median frequency murmur
Cardiac Auscultation
• Aortic area R 2nd ICS
• Pulmonary area L 2nd ICS
• Tricuspid area L 4th ICS
• Mitral area Apex
• Bell lower pitch sound, slightly to firmly
• Diaphragm high pitch sound, firmly
Cardiac AuscultationHeart Sound
Heart sound
• S1: closure of MV—Apex
closure of TV—left lower SB Widely split of S1: RBBB Single S1: LBBB Load S1: Rapid heart rate, short PR, MS
Cardiac AuscultationHeart Sound
• Normal splitting of S2 in inspiration, S2 split into A2 and P2
• Abnormal splitting of S2 Wide physiological splittingdelay P2 or early
A2 (RBBB or MR) Paradoxical splittingLBBB or RV pacemaker Narrow physiological splitting pulmonary
hypertension Fixed splitting: ASD
Cardiac AuscultationHeart Sound
• Early systolic soundsAortic or pulmonary ejection sounds (AS, bicuspid AV, PS)
• Mid- to late systolic sounds (click) MVP• Early diastolic soundsMS (opening snap),
pericardial knock (constrictive pericarditis), MR knock(with poor LV compliance), atrial myxoma(polp)
• Mid- to late diastolic sounds S3 or S4
Heart sound
• S3 is generated during ventricle rapid filling (normal < 40Y)
LV dysfunction, AR, increase rate or volume of ventricle filling
• S4 is generated during atrial contribution to ventricle filling (may be normal in elderly?)
HTN,AS, HCM, ischemic heart, acute MR