Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017 Please review the above anatomy of the heart. THINGS TO REMEMBER There are 3 electrolytes that affect cardiac function o Sodium, Potassium, and Calcium When any of these electrolytes are out of the normal range you may see changes in your cardiac rhythm and in your rhythm strip analysis Normal contraction of the atria and ventricles is sequential Automaticity - ability to automatically generate electrical impulse o The SA node is normally in control and is called the pacemaker of the heart because it possesses the highest level of automaticity. If the SA node fails to generate electrical impulses at its normal rate or stops functioning entirely, or if the conduction of these impulses is blocked, pacemaker cells in secondary pacemaker sites can assume control as pacemaker of the heart but at a much slower rate. In general the farther away the impulse originates from the SA node, the slower the rate. This does not apply to Sinus Bradycardia; which originates from the SA node at a slower pace than normal
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THINGS TO REMEMBER...If you see a lethal arrhythmia(v-fib, asystole, v-tach without a pulse, etc.) call a CODE BLUE and Start your ACLS Protocol If you have a change in rhythm, notify
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Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Please review the above anatomy of the heart.
THINGS TO REMEMBER
There are 3 electrolytes that affect cardiac function
o Sodium, Potassium, and Calcium
When any of these electrolytes are out of the normal range you may see changes in
your cardiac rhythm and in your rhythm strip analysis
Normal contraction of the atria and ventricles is sequential
Automaticity - ability to automatically generate electrical impulse
o The SA node is normally in control and is called the pacemaker of the heart because it possesses the
highest level of automaticity. If the SA node fails to generate electrical impulses at its normal rate or
stops functioning entirely, or if the conduction of these impulses is blocked, pacemaker cells in
secondary pacemaker sites can assume control as pacemaker of the heart but at a much slower rate. In
general the farther away the impulse originates from the SA node, the slower the rate.
This does not apply to Sinus Bradycardia; which originates from the SA node at a slower pace
than normal
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Electrical Conduction
Sinus Atrial (SA) Node; High in the R atrium. Pacemaker of the heart. Rate of automaticity @ 60-
100bpm
Atrio-Ventricular (AV) Node; low R atrium near the Tricuspid valve.
o Slows conduction from atria-ventricles through Bundle of His, allowing time for atria to empty
blood into ventricles. Impulse rate of 40-60 bpm. Back up pacemaker to SA node
Bundle of His-Purkinje Fibers; Upper portion of the septum connects the AV node with the two bundle
branches. Purkinje fibers are hair like fibers along the endocardial surface of both ventricles. Impulse
rate 20-40 bpm.
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Sequence of Excitation
Impulse passes from SA node ►AV node ► ventricles via the atrioventricular bundle (Bundle of
His) Bundle of His splits into two pathways in the interventricular septum (Bundle Branches, R & L)
R & L Bundle branches carry the impulse towards the apex of the heart
Purkinje fibers carry the impulse to the heart apex and ventricular walls
Segment Representation P wave: represents depolarization/spread of electrical impulse through R & L atria
PR interval: conduction of impulse through AV node, Bundle of His, R/L Bundle Branch
& Purkinje fibers
QRS: Depolarization of ventricles (Q, R & S waves)
ST segment: represents early repolarization of ventricles
J point: where QRS stops and ST segment begins T wave: represents ventricular repolarization
QT interval: Represents total Ventricular activity
Artifact: distortion of ECG tracing by non cardiac electrical activity like:- loose electrode, patient movement, muscle activity, interference
o Depolarization: cardiac cell is stimulated and positively charged. Greater Na/Ca in the cell o Repolarization: returning back to its resting state of negative charge.
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
The 6 Step Approach to strip interpretation
Rate? ( Tachy, Brady, normal SR)
Rhythm? (Regular or Irregular)
P-R Interval Normal? (0.12-0.20 sec)
QRS complex normal? (0.04-0.11sec or ≤ 0.11) (does
every QRS look the same?)
QT Interval (0.36 – 0.44) Usually gender/age dependent
P-wave upright & preceding every QRS? (one P-
wave for every QRS) (does every P wave look the same?)
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Interventions
If you see a lethal arrhythmia(v-fib, asystole, v-tach
without a pulse, etc.) call a CODE BLUE and Start your
ACLS Protocol
If you have a change in rhythm, notify your supervisor,
gather pertinent data (i.e. history, recent labs, prior
rhythm, medications) and notify the physician if
applicable
Measurements
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Let’s Review some rhythm strips
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Cardiac Telemetry Self Study: Part One Cardiovascular Review 2017
Let’s Practice!
For each practice strip, use the 6 step approach to identify: Rate, Rhythm,
P-R interval, QRS complex, QT interval, and P-wave characteristics