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Tayangan Case 1

Jun 04, 2018

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Mirza Heltomi
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    DIFFICULTY OF DIAGNOSISCEPHALOPELVIC DISPROPORTION

    AT A PRIMIGRAVIDAWITH SEVERE PREECLAMPSIA

    By: Yudha Pranata

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    cephalopelvic

    disproportion

    failure to

    progress

    CERVICALEFFACEMENT

    AND DILATATION

    DESCENT OFPRESENTING PART

    INTRODUCTION

    ABNORMAL

    PROGRESS OFLABOR

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    PREGNANCY INDUCED

    HYPERTENSION

    IS ONE OF THE FACTOR THAT CAUSEDMATERNAL AND PERINATAL MORBIDITY AND MORTALITY

    C-SECTION DUE TO DYSTOCIA

    CONTRIBUTED 1/3 OF THE TOTALC-SECTION RATE

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    IDENTITY

    NAME

    AGE

    ADDRESS

    EDUCATION

    OCCUPATION

    MEDICAL RECORD

    DATE OF ADMISSION

    : Mrs. H: 21 y.o

    : Cibeunying Bandung

    : Senior High School

    : House wife

    : 0504 xxxx: August 10 th, 2005 at 16.00

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    ANAMNESIS Refered by :

    Mid Wife A- RB Al-Islam Bandung

    Letter of explanation :G1P0A0 term parturien 2nd stage +CPD + SEVERE PREECLAMPSIA(BP: 150/100 mmHg)

    Chief complain :Baby wasnt delivered yet after 2hours bear down

    G1P0 A0

    HIGHBLOOD

    PRESSURE

    15 HOURS

    BEFORE

    ADMISSION

    LABOR PAINTerm pregnantBaby wasnt delivered yetafter 2 hours bear down

    Know hypertension 5 hours beforeadmission (160/110 mmHg)History of hypertension (-)Blurred vision, severe cephalgia, epigastricpain (-)

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    Amnionic membrane (-) 6 hours before admission

    clear, febris (-)

    Fetal movement (+)

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    OBSTETRIC HISTORY

    1. This Pregnancy

    Additional anamnesis : Marital history :

    , 21 y.o, senior high school, house wife

    , 22 y.o, STM, private employee

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    Contraception : -

    Last Menstrual Period : Nov, 5th 2004

    Estimed birth pregnancy : August, 12th 2005

    Prenatal care : midwife 11x,

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    PATIENTS HOME VISIT

    Lived with her parents and husband in semi permanenthouse 5 x 7 m2

    Mother was only housewife, her husband was privateemployee

    300 m from nearest midwife

    5 times PNC at Padasuka PHC6 times at other midwife

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    PHYSICAL EXAMINATION

    General Condition : composmenthis, goodBlood Pressure : 160/110 mmHgPulse rate : 80 x/mntRespiration Rate : 20 x/mntTemperature : 36,50CBody Weight : 57 kgs

    Body Height : 145 cmsLiver And Spleen : hard to assessOthers : within normal limits

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    EXTERNAL EXAMINATION

    Fundal height : 32 cm above the symphisis

    Abdominal circumference: 102 cm

    Fetal position : Head U back at left 3/5

    Fetal heart rate : 136-140 x/mnt

    Uterine Contraction: Once in 3-4 minutes, 40second, strong

    Estimated fetal weight: 2800 grams

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    INTERNAL EXAMINATION

    Vulva/vaginal : No abnormalities

    : Complete

    Amnionic membrane : (-), residual fluid (+)

    Head : St -1, caput (+) as big as egg

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    PELVIC EXAMINATION

    Promontorium : not palpable

    Linea innominata : palpated 1/3 1/3

    Sacrum : Concave

    Spina ischiadica : not prominent

    Pubic Arcus : >90 o

    Side- wall : straight

    Pelvic : good

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    LABORATORY FINDINGS

    Hemoglobins : 12,6 gr% Ureum/Creatinin : 22/1,48 mg/dl

    Leucocytes : 24.400/mm3 Ur/Kr : 18/0,81

    Ht : 38 % Blood Glucose : 85 mg/dl

    Trombocytes : 312.000/mm3 Asam urat : 4,3 mg/dl

    Urine : ++ Na : 138 mEq/dl

    SGOT/SGPT : 23/23 U/L K : 2,6 mEq/dl

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    DIAGNOSIS

    G1P0A0 term pregnant 2 nd stage of labor +

    severe preeclampsia +

    cephalopelvic disproportion

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    PLAN OF MANAGEMENT

    KaEn 1B infussion, cross match, blood reserve

    MgSO4 40% intramuscular injection (RB Al-Islam)

    MgSO4 20% intravenous, then MgSO4 40%

    intramuscular for maintenance dose

    ECG, thoracal Ro, complete laboratory result Internal and neurology consult

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    Planned to perform C-section due to cephalopelvicdisproportion

    Informed consent

    Anaesthesiologist consult

    Contact perinatologist

    Observation General condition, vital sign, fetal heartrate, uterine contraction

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    Internal department consult

    Dk/ - G1P0A0 term parturition 2nd stage of labor + CPD- severe preeclampsia

    Advis : Low dietary salt Metyldopa 3x500 mg titration dose

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    Neurologist consult

    Dk/ - 2nd hypertension in pregnancy

    Advis : Blood pressure regulation according to Internal

    Department Consult if there was focal neurological deficit

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    Observation

    Time Uterine

    contraction

    FHR

    (x/mnt)

    BP

    (mmHg)

    PR

    (x/mnt)

    RR

    (x/mnt)Information

    16.00-17.00 3-41x/45 S 136-140 160/110 80 20 - Admission testBaseline 140-150 bpmVariability > 5 bpm Akseleration (+)Deceleration (-)- Informed consent- BP resucitation- Internal and

    neurological consult

    17.00-18.00 3-41x/45S 140-144 170/110 84 24

    18.00-18.15 3-41x/45S 144-148 160/160 88 24

    Internal Examination at 18.15:v/v : no abnormalities

    : complete Amniotic membrane : (-)

    Head : St -1, caput (+) as big as egg

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    D/ G1P0A0 term parturition 2nd stage of labor +cephalopelvic disproportion + severe preeclampsia

    T/ Planned to perform C-section due to cephalopelvicdisproportion Anaesthesiologist consultContact perinatologist and operation theatreObservation vital sign, FHR, uterine contraction

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    The patient wasbrought to EMGoperating theatre

    18.20

    18.30

    The patient arrived atEMG operating theatreUC : 3-41x/40 S FHS: 136-140 bpm

    Cesareansection began.

    18.40

    A male baby was born by headluxation

    BW:2770 gr, BL:49,3 cmHead circumference: 34 cm

    18.50 19.40

    18.55

    The umbilical cord wasdelivered by gentlytraction of the cord

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    Preoperative diagnosis :

    G1P0A0 term parturition 2nd stage of labor + cephalopelvicdisproportion + severe preeclampsia

    Postoperative diagnosis

    P1A0 term delivery by C-section due to cephalopelvicdisproportion + severe preeclampsia

    Type of surgery : SCTP + IUD insertion

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    DISCUSSION

    1 H t di g h l l i

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    Inadequateuterus

    contraction

    Birth canal

    resistention

    Progresif progress ofcervical effacement anddilatation

    Fetal descent

    1. How to diagnose cephalopelvic

    disproportion on this patient?

    Abnormal

    Labor progress

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    Tabel 1. Clinical findings in woman with ineffectivelabor

    Inadequate cerviks dilatation or fetal descentProtracted labor-slow progress Arrested labor-no progress

    Inadequate expulsive effortFetopelvic diproportion

    Excessive fetal sizeInadequate pelvic capacity

    Malpresentation or position of the fetusRuptured membrane without labor

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    American College ofObstetricians and

    Gynecologists - ACOG (1995)

    Abnormalities

    caused dystocia

    Abnormalities of the powers

    Abnormalities involving the passenger

    Abnormalities of the passage

    Uterine contraction andmother power to beardown

    attitude, size and fetal abnormalities

    Pelvic bone andsoft tissue abnormalities

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    Failure to progress

    Dystocia

    Abnormally slow progressof labor

    Cephalopelvicdisproportion

    http://www.doereport.com/enlargeexhibit.php?ID=614
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    http://www.doereport.com/enlargeexhibit.php?ID=614
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    Normally pelvic

    Normally size

    CEPHALOPELVIC DISPROPORTION

    FETAL MOTHER

    Contracted pelvic

    Excessivefetal size

    COMBINATION

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    PELVICTYPE

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    CEPHALOPELVICDISPROPORTION

    Absolute Relative

    Big fetal head or

    Small pelvic bone

    Normally pelvic capacityBut asynclitism (+)

    Extension (+)

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    TRUE DISPROPORTIONMOLDING WITHOUT

    DESCENT OF PRESENTING PART

    VAGINAL DELIVERY WAS IMPOSSIBLE

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    ABDOMINOVAGINAL EXAMINATION( MULLER SIGN )

    ABDOMINAL EXAMIINATION

    ( OSBORN SIGN )

    X-RAY OR USG PELVIMETRY

    EXAMINE THEDISPROPORTION

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    PELVIC EXAMINATION WAS GOOD FETAL WAS NORMAL IN SIZE ABDOMINAL PALPATION WAS 3/5 INTERNAL EXAMINATION WAS AT STATION -1 COMPLETELY CERVICAL DILATATION CAPUT WAS (+)

    IN THIS CASE

    DISPROPORTION CAUSED BYMALPRESENTATION OR BY

    MALPOSITION

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    Tabel 1. Clinical findings in woman with ineffectivelabor

    Inadequate cerviks dilatation or fetal descentProtracted labor-slow progress Arrested labor-no progress

    Inadequate expulsive effortFetopelvic diproportionExcessive fetal sizeInadequate pelvic capacity

    Malpresentation or malposition of the fetusRuptured membrane without labor

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    MA L PRENTATION : NO N VERTEX PRESENTATION

    BREECH PRESENTATION 3% BROW PRESENTATION 1/1500 DELIVERY FACE PRESENTATION 1/500 DELIVERY

    MA L POSITION : A B NORMA L LY VERTEX POSITION

    TO THE MATERNA L PELVIC

    OCCIPITOLATERAL OCCIPITOPOSTERIOR

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    TYPE OF PRESENTATION

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    DIAGNOSIS OF SEVERE PREECLAMPSIA

    Diastolic blood pressure > 110 mmHg Urinary protein >2 g/24 h or > 2+ Serum creatinin > 1.2 mg%

    with oliguria < 400 ml/24 h

    Trombocytopenia < 100.000 /mm3 Increase LDH levels Increase liver enzym Cephalgia with visual and cerebral disturbance Epigastric pain Pulmonal oedema with cyanosis HELLP syndrome

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    MgSO4 Continuous intravein infusion

    Initial dose : 4 g (20 cc MgSO4 20%) in 100 cc RL for15-20 minutesMaintenance dose : 10 g (50 cc MgSO4 20%) in 500 ccRL 1-2 g/h (20-30 gtt/minutes)

    Intermitten intramuscularInitial dose : 4 g (20 cc MgSO4 20%) iv

    with 1 g/minutesMaintenance dose : 4 g (10 cc MgSO4 40%) every 4 h

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    C section in this case

    No progress of labor No descent of presenting part Presenting part was still high

    Spontaneous conversion to the face or vertekspresentation was rare

    No indication for assisted delivery by vacuum or

    forcipal extraction

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    PROGNOSIS FOR THE NEXT DELIVERY

    ASNM : CPD 1/250 DELIVERY AJPH : MORE THAN 65 % MOTHER WHO

    HAD BEEN DIAGNOSED WITH CPD,

    WERE ABLE TO DELIVERVAGINALLY IN NEXT PREGNANCIES

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