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Page 1: Sleep Medicine for Ortho-Pedo Residents
Page 2: Sleep Medicine for Ortho-Pedo Residents

Optimum Development of the Teeth, Jaws and Face: Yet Another Reason to Breastfeed

Kevin Boyd, M.Sc. (Nutrition), D.D.S.

Hypothesis: Ancestral (Paleo) regimens of Infant and Early Childhood Feeding (IECF) were/are protective against skeletal malocclusion pre-Industrial/Westernized cultures

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The Problem:

SDB/OSA/Neuro-cognative Impairment:

The Solution:

Early Identification and Collaboration:

- compromised naso-respiratory function

-unhealthy sleep architecture (PSG)

-Sleep Medicine

-Evolutionary Medicine

-Evolutionary Dentistry

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1. Sleep is not a luxury, sleep is as necessary to survival and well-being as food and water

Take Away Points:

2. There is a bi-directional association(e.g., perio/T2DM)between certain craniofacial phenotypes(high-vaulted palate, posterior crossbite, retrognathia, hyper-divergent

growth) and clustering with other known risk factorsfor SDB/OSA (snoring, ATH, bedwetting, night terrors, restless legs/active sleep, etc.)

3. preventive strategies include OMT, infant feeding/diet counseling; Tx alternatives to CPAP/surgery include RME, BB-O

4. collaborativeopportunities(responsibility?) exist for orthodontists, pediatric dentists, GP’s and RDH’s to identify/screen at-risk kids for SDB/OSA….refer (ENT’s, OMT’s, Sleep specialists) and Tx p.r.n.

e.g., The AAPMD

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In conclusion:-abnormal craniofacial morphology, but not excess body fat, was associated with SDB in children 6–8 years of age.-patients with dental malocclusions, deviant craniofacial features and tonsillar enlargement should always be examined as regards to their sleeping habits, snoring and pauses in breathing during sleep.-children with tonsillar hypertrophy, cross bite and convex facial profile could be candidates for

early intervention and orthodontic treatment to prevent the progression of SDB in coming years.

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Childhood Sleep Disorder Breathing: A Dental Perspective

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Narrowed posterior pharyngeal space associated with most bi-maxillary retrognathic (modern) skulls

Hyoid bone inferior position (to MP) associated with most bi-maxillary retrognathic (modern) skulls

Obtuse naso-labial angle associated with most bi-maxillary retrognathic (modern) skulls

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Lateral cephalographs of 3 children with chronic mouth breathing: images show different grades of airway

obstruction relative to adenoid size. A, Grade 1 in a girl 12 years 3 months old. B, Grade 2 in a boy 4 years 4

months old. C, Grade 3 in a boy 4 years 9 months old who also exhibits the typical morphologic and dental characteristics of long face syndrome.

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-In the deciduous dentition, a distinctive occlusal and skeletal pattern of Class II maloccluson exists. In addition to concomitant diagnostic dental relationships in the sagittal plane (distal step, Class II deciduous canine relationship, excessive overjet), transverse interarch discrepancy due to a narrower maxillary arch is a constant feature of early Class II malocclusion. Skeletal findings in children with Class II malocclusion typically include significant mandibular retrusion and shorter total

mandibular length.

-the clinical signs of Class II malocclusion are evident in the

deciduous dentition and persist into the mixed dentition.

Am J Orthod Dentofacial Orthop. 1997 May;111(5):502-9.

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3. preventive strategies include OMT, infant feeding/diet counseling; Tx alternatives to CPAP/surgery include RME, BB-O

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Hypothesis: Ancestral (Paleo) regimens of Infant and Early Childhood Feeding (IECF) were/are protective against skeletal malocclusion pre-Industrial/Westernized cultures

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“ …(malocclusion) is a relatively new phenomenon in the human population and we do not find it in skeletons until after the seventeenth century. ” -Peter Gluckman

“… jaw anomalies (malocclusions wherein the teeth cannot fit properly in the jaw) are relatively new to European populations. Well-preserved skeletons from the 15th and 16th centuries show almost no malocclusion in the population….”

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“….there is much circumstantial evidence that jaws and faces do not grow to the same size that they used to precisely because of our softer, more processed diets.” Daniel E. Lieberman

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Abingdon Cemetery

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Is the basis for McNamara’s “Ideal” anthropologically informed?

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The Angle Orthodontist: 54 (1): 5-17 1984

Angle Orthod. 54(1): 5-17 1984

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Pierre-Robin pt. –retrusive mandible…retrusive maxilla!

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ILLINOIS SLEEP SOCIETY CONFERENCE 2012

Advancing Mandibles and Maxillas with Biobloc-Orthotropics: A Non-Surgical Approach to Increasing Posterior Pharyngeal Airway Space in Pediatric OSA Patients

Kevin Boyd, DDS

Kevin L. Boyd, M Sc, DDS

Courtesy of Brian Hockel, DDS

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2/15/2010 BIOBLOC STAGE-1 Tx 2/21/2012 BIOBLOC STAGE- 3 Tx

Example: Assign an arbitrary control value for airway radius of 1.0. A 50% reduction in airway radius would mean that the new airway radius would be 0.5. Now, according to Poiseuille, that gives us...R = 1/(0.5)4R = 1/0.0625 = 16Therefore, resistance to airflow is increased 16-fold with a decrease in airway diameter (and radius) of 50%.

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Stephen Sheldon, MD CMHCorrelating PSG data with Biobloc TX response

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Original Article Changes of pharyngeal airway size and hyoid bone position following orthodontic treatment of Class I bimaxillary protrusionQingzhu Wanga; Peizeng Jiab; Nina K. Andersonc; Lin

Wangd; Jiuxiang LineABSTRACT

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Although dentists and orthodontia recognize the importance of evaluating and treating OSA, they have yet to realize howwell-positioned they are for the prevention of sleep-

disordered breathing (SDB).

Another possible explanation for our findings is that oral cavity features such as high palates, narrow dental arches, and retruded chin all are additional risk factors for SDB in children38

38. Kushida CA, Efron B, Guilleminault C. A predictive morphometric model for the obstructive sleep apnea syndrome. Ann Intern Med. 1997;127:581–587

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