GROUP 5 MEMBERS 1. Alex Mbewe 2. Monica Banda 3. Rosella Munyenyembe 4. Andrew Moyo 5. Nelson Munthali 6. Mtisunge Wandale 7. Jacqualine Ntaba
Dec 19, 2014
GROUP 5 MEMBERS1. Alex Mbewe2. Monica Banda3. Rosella Munyenyembe4. Andrew Moyo5. Nelson Munthali6. Mtisunge Wandale7. Jacqualine Ntaba
PRESENTS SHIGELLOSIS
By ALEX ‘ SIAL’ MBEWE
BROAD OBJECTIVE
By the end of this presentation, learners should acquire knowledge on how to manage a patient with shigellosis.
SPECIFIC OBJECTIVESO Definition of shigellosisO Causes of shigellosisO Types O How it is spreadO PathophysiologyO Clinical manifestationsO Medical managementO Nursing managementO Complications
Definition
OThis is an acute bacterial infection of the lining of the intestines (especially large intestines)
CausesShigellosis is caused by a group
of bacteria called shigella (gram- negative organism)
Types
1. Shigella sonei – also called group D. it is responsible for most of the cases
2. Shigella flexineri Also called group B
3. Shigella dysenteriae Can lead to outbreaks in developing
countries
SPREADO Shigellosis is spread through fecal-
oral routeO People with shigellosis release it
through the stoolsO It spreads from one infected person
to contaminate water or food or directly to another person.
O Outbreaks are associated with poor sanitation, contaminated food or water and crowded living conditions
O Common among travelers in developing countries and workers or residents of refuge camps
CLINICAL MANIFESTATIONS
OUsually develop about 1-7 days (average 3 days) after you come into contact with the bacteria
OAcute (sudden) abdominal pain or cramping
OAcute (sudden) feverOBlood, mucus or pus in stoolsOCrampy rectal painONausea and vomiting
OWatery diarrhoeaOAbdominal tendernessODehydration with fast heart rate
and low BpO Loss of appetite
Diagnostic tests
OStool cultureOWhite blood cells in stoolsOElevated blood cell count (FBC)
PATHOPHYSIOLOGYO Once ingested, the bacteria survives the
gastric environment of the stomach and progresses to large intestines
O There, they attach to and penetrate the epithelial cells of the intestinal mucosa.
O After invasion, they multiply intracellulary and spread to neighboring epithelial cells, resulting in tissue destruction.
O It produces toxins that can attack the lining of the large intestines, causing swelling, ulcers on the intestinal wall and bloody diarrhoea.
Pathophysiology cont….
OSeverity of diarrhoea sets apart shigellosis from regular diarrhoea and it is usually associated with bloody or pus stained diarrhoea.
MEDICAL MANAGEMENTO The goal is to replace fluids and
electrolytesO Advise patient on dietO Self measure to avoid dehydration like
drinking electrolyte solution to replace fluids e.g. ORS
O Antibiotics only in severe cases e.g. ampicillin and ciprofloxacin 250mg BD IV-they shorten the length of illness
O Antidiarrhoea agents e.g. Loperamide 2mg BD
O I.V fluids 2-3 litres/24hrs e.g. R/LO Stop taking diuretics
NURSING MGTOASSESSMENT - History of stool pattern and
associated symptomsO FrequencyODurationOCharacterOConsistency of stools
O history of medication use of other drugs known to cause
diarrhoea e.g. laxativesOSocial history
NURSING MGT CONT……
OFamily historyORecent travel, stress, health
and family history of illnessOEating habits, appetite, food
intolerance especially milk and other dairy products
Objective data Lethargy Sunken eye balls Fever Pallor Dry mucous membranes Poor skin turgor Parienal irritational Malnutrition Concentrated urine
Physical examinationOVital signs and weight measurementOPatients’ skin is inspected for signs
of dehydrationOPoor turgor and dryness and area of
breakdown of the skinOAbdomen
• Distension• Bowel sounds• Palpate for tenderness
Nursing diagnosisODiarrhoea r/t acute infectious
process evidenced by frequent loose and liquid stools
OFluid and electrolyte imbalance r/t diarrhoea and vomiting
ONutritional imbalance; less than body requirements r/t loss of appetite, nausea, vomiting evidenced by weight loss
OAltered thermoregulation hyperthermia r/t to the infection as evidenced by rise of temperature to 38 degrees celsius
OAltered comfort (abdominal pain) r/t increased peristalsis evidenced by patient’s verbalization and facial expression
ORisk for anemia related to blood in stools
ORisk for altered skin integrity related to dehydration
ORisk for Hypovolemic shock r/t loss of fluids due to diarrhoea
Interventions O Commence IV fluids as ordered e.g. R/L – to
replace lost fluids and correct electrolyte balanceO Catheterize – to monitor input and output and balance fluidsO Enforce strict IP measures to avoid cross infectionO Provide small and frequent food to normalize
nutritional status and reduce peristalsis movement
O Administer prescribed antipyretics e.g. panadol 1g tds po. This will act on the prostagrandin of the hypotharamus hence it will reduce fever.
O Administer analgesics e.g. panadol 1g po tds to reduce pain .
complicationsO Intestinal perforationODehydrationOHypoglycemiaOComaORectal prolapseOHypovolemic shockOBacteremiaOPeritonitis
ReferencesO Lewis S.M., Heitkemper M.M and Dirksen
S.R. (2010). Medical surgical nursing assessment and management of clinical problems.(7thed) St Louis:C.V.Mosby
O Smeltzer S.C., Bare B.G and Hinke J.L (2010). Brunner & suddarth’s textbook of medical surgical nursing.(12th ed). Philadelphia:J.B Lippincott
O www.mayoclinic.com