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DR.TARIG MAHMOUD MD SUDAN HAIL UNIVERSITY KSA Post partum collapse
24

postpartum collapse

Jul 19, 2015

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Page 1: postpartum collapse

DR.TARIG MAHMOUD

MD SUDAN

HAIL UNIVERSITY KSA

Post partum collapse

Page 2: postpartum collapse

Maternal collapse is defined as an acute event involving the cardiorespiratory systems and/or brain, resulting in a reduced or absent conscious level (and potentially death).

Page 3: postpartum collapse

causes

Shock- eg postpartum haemorrhage.

Pulmonary embolus

Amniotic fluid embolus

Cardiac causes

Intracranial events

Drug overdose/toxicity

Biochemical causes –e.g. hypoglycaemia

Page 4: postpartum collapse

Primary PPH Secondary PPH

Loss of MORE than or EQUAL to 500mL blood from the genital tract within 24 hours of delivery

Loss of MORE than or EQUAL to 500mL blood from the genital tract between 24 hours and 12 weeks post delivery

Postpartum haemorrhage

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PPH CAUSES:

1.Uterine atony

2.Retained placenta

3.Genital tract laceration

4.Coagulopathy

5.Acute inversion of uterus

Page 6: postpartum collapse

REMEMBER

4“T”s

Uterine atony

Retained placenta and/ormembranes

Clotting disorder

Tone

Tissue

Trauma

Thrombin

Injury to vagina, perineum and uterine tears at C/S

Page 7: postpartum collapse

1.Uterine atony

Predisposing conditions:

Multiparity.

Over distension of uterus (Macrosomia,polyhydramnios).

Prolong labour (uterine inertia).

Fibroid.

Placenta previa.

Oxytocin induce labour.

Page 8: postpartum collapse

2.Retained placenta

Prevent a uterus from contracting .

Causes:

Placenta separated but undelivered.

Placenta partly or wholly attached.

Placenta accreta.

Page 9: postpartum collapse

3.Genital tract laceration

Perineal or vaginal tears.

Causes:

Instrumental delivery (Cervical & vaginal tears).

Extension of episiotomy .

Uterine rupture.

Page 10: postpartum collapse

4.Coagulopathy

Causes:

Von Willebrand's Disease, Platelet disorder.

Placenta abruption (retro placental clot leadsto consumption of clotting factors).

Unidentified dead fetus.

Amniotic fluid embolus (amniotic fluidentering maternal circulation)

Page 11: postpartum collapse

5.Acute inversion of uterus

Caused by traction on the umbilical cord before placenta has separated.

Associated factors:

-Fundal placenta

-Short cord

-Morbidly adherent placenta

Lead to Cardiovascular collapse & shock.

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Mangment of PPH

ABC (fluid resuscitation).

Feel abdomen-uterus poorly contracted rub the fundus gently (see wether uterus contract and bleeding arrested).

Set up IV line & IV infusion of 40 units of syntocinon.

Bimanual compression

potent drugs can also be used these include ergometrine, prostaglandin F2α or misoprostol.

The bladder should be catheterized as an empty bladder aids uterine contraction.

Page 13: postpartum collapse

Send blood for group and cross matching. Examine placenta to see if it is complete(If

uncompleted, sent to theatre for manual remove).

If complete(Examine vagina and cervix in lithotomy position for laceration)

No laceration(allow further measures (eg. uterine tamponade,radioloical occlusion)

Exploratory Laporotomy(iliac artery ligation) or B lynch (sutures which are placed in double loop surround the uterus & aim to squeeze whole uterus) .

Hysterectomy correction of clotting factors using fresh frozen

plasma, platelets and cryoprecipitate.

Page 14: postpartum collapse

Bimanual compression

Page 15: postpartum collapse

B lynch

Page 16: postpartum collapse

Pulmonary embolism

Thrombosis is the most common cause ofmaternal death in the UK.

The most common presentation is of mildbreathlessness, or inspiratory chest pain, in awoman who is not cyanosed but may beslightly tachycardic (90 bpm) with a mildpyrexia (37.5ºC).

massive PE may present with suddencardiorespiratory collapse.

Page 17: postpartum collapse

Diagnosis and management:

o PE can be a cause of sudden cardiorespiratorycollapse.

o In this situation, diagnosis and managementshould occur simultaneously.

o Urgent resuscitation using the structured ABCapproach is needed.

o If PE is suspected, anticoagulation should beinstituted.

Page 18: postpartum collapse

Amniotic fluid embolism

caused by amniotic fluid entering the maternal circulation.

This causes acute cardiorespiratory compromise and severe DIC.

Difficult to diagnose in life, and is typically diagnosed at postmortem, with the presence of fetal cells (squames or hair) in the maternal pulmonary capillaries.

Page 19: postpartum collapse

Diagnosis and management:

Symptoms occurring just before the collapse may be helpful in diagnosis.

• breathlessness.

• chest pain.

• feeling cold.

• lightheadedness.

• restlessness, distress and panic.

• pins and needles in the fingers.

• nausea and vomiting.

Page 20: postpartum collapse

The prognosis is poor, with around 30 per cent of patients dying in the first hour and only 10 per cent surviving overall.

Management is supportive, requiring intensive care and there are no specific therapies available.

Page 21: postpartum collapse

Cardiac causes

The main cardiac events are myocardial infarction, aortic dissection and cardiomyopathy.

signs and symptoms such as central chest or interscapular pain, a wide pulse pressure, and a new cardiac murmur.

Management by ABC approach and seek help of cardiologist.

Page 22: postpartum collapse

Intracranial events

Intracranial haemorrhage is complication of uncontrolled hypertension, but can also result from ruptured aneurysms and arteriovenous malformations.

severe headache often precedes maternal collapse.

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Drug overdose/toxicity

In obstetric practice, the two main drugs that can give rise to overdose or toxic problems are magnesium sulphate and local anaesthetic agents.

The antidote to magnesium toxicity is 10 ml 10% calcium gluconate given I.V .

Intralipid 20%should be used in cases of collapse secondary to local anaesthetic toxicity.

Page 24: postpartum collapse

Thank You for