Nursing Care Plan Client: Bartolabac, Fidela Hospital No.: 0-800-22500- 773 Age: 66 years old Room No.: C414 Impression: cough, dyspnea Physician: Dr. R. Go Diagnosis: Community acquired pneumonia – high risk in acute respiratory failure Nurse’s Signature: R.C.R. UCSN Clinical Portrait Pertinent Data Assessment Upon assessment patient X, 66 years old, a Roman Catholic from 0671 Sitio Anagan, Apas, Cebu City, was seen lying on bed conscious and a febrile with the S.O. at the bedside. Patient had an IVF of D5NSS infused at right arm/hand running at 30 gtts/min + dopamine at 10gtts/min. A NGT was inserted and was aided with a mechanical ventilator. Pulse oximeter was connected at the right hand and FBC-UB was noted. Significant Findings History of Present Illness A case of Mrs. Fidela B. Bartolabac, 66 years old, born on April 24, 1942 at Berida, Leyte, Roman Catholic and is widowed for 3 years, residing at 0671 Sitio Amagan, Apas, Cebu City 6000 was admitted on November 29, 2008 at 1:44 pm at Chong Hua Hospital. Mrs. Bartolabac has Bronchial Asthma taking Seretide and Ventolin inhaler for treatment but is poorly compliant. She was taking vitamins. Family history of diabetes.
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Nursing Care Plans for Community Acquired Pneumonia 2009
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Nursing Care Plan
Client: Bartolabac, Fidela Hospital No.: 0-800-22500-773Age: 66 years old Room No.: C414Impression: cough, dyspnea Physician: Dr. R. GoDiagnosis: Community acquired pneumonia – high risk in acute respiratory failure Nurse’s Signature:
R.C.R. UCSN
Clinical Portrait Pertinent Data
Assessment
Upon assessment patient X, 66 years old, a Roman Catholic from 0671 Sitio Anagan, Apas, Cebu City, was seen lying on bed conscious and a febrile with the S.O. at the bedside. Patient had an IVF of D5NSS infused at right arm/hand running at 30 gtts/min + dopamine at 10gtts/min. A NGT was inserted and was aided with a mechanical ventilator. Pulse oximeter was connected at the right hand and FBC-UB was noted.
Significant Findings
Patient was tachypniec with vital sighs of BP= 90/60 mm/Hg. HR of 117 bpm and RR of 31 cpm. Roles positive at binasal assessment and occasional wheeze BLF. Patient was tachycardic with regular murmurs. Positive cyanosis on finger nail beds of
History of Present Illness
A case of Mrs. Fidela B. Bartolabac, 66 years old, born on April 24, 1942 at Berida, Leyte, Roman Catholic and is widowed for 3 years, residing at 0671 Sitio Amagan, Apas, Cebu City 6000 was admitted on November 29, 2008 at 1:44 pm at Chong Hua Hospital.
Mrs. Bartolabac has Bronchial Asthma taking Seretide and Ventolin inhaler for treatment but is poorly compliant. She was taking vitamins. Family history of diabetes.
Mrs. Bartolabac used to for about 30 packs per year and drinks alcohol occasionally. She was positive of pulmonary tuberculosis and was poorly taking anti TB medications for 2 years. Months prior to admission weight loss was positive and occasional chest pains noted
both hands. With history of PTB.
Nursing Review of Systems Gordon's 11 Functional Health Patterns
1. Health perception/ Health management
Patient had history of Pneumonia and Pulmonary Tuberculosis. Doctors prescribed her with medicines but the patient was not able to maintain it due to financial problem. She never complained about her condition though she experienced short of breathe and cough. Her family decided to admit her to Chong Hua hospital when she cannot tolerate it anymore and when cyanosis was observed by the family member.
She used Wachichao plant for her maintenance.
2. Nutrition/ Metabolic
Patient is choosy about her food. She usually eats rice and liver barbeque. She loves to eat fruits as long as they have. She seldom eats meat. She eats vegetables once in a while. In the morning, she eats bread and ate breakfast and lunch (brunch) at around 11:00AM
Few weeks before admission, she just want to eat porridge for her meal. Now, she has Nasogastric
Chief Complaints
Cough, dyspnea
Vital Signs Taken Upon Admission
T= 36.2◦C R= 31 cpmP= 117 bpm BP= 90/60 mmHg
Laboratory results
X-Ray12/14/08
Conclusion
1. Chronic inflammation process in both lung fields2. Modified silhouette3. Atherosclerosis of the thoracic aorta4. Calcified trachoebronchial tree5. Generalized osteopenia/osteoporosis
CBC12/13/08
Result reference unitWBC 6.28 4.8-10.8
10^3/ul
tube attached.
3. Elimination
Patient had normal elimination pattern but then, few days after her admission, she experienced constipation but had good urine output. Now, she defecates daily and maintain good urine output.
4. Activity/ Exercise
Patient usually does sedentary activities at home, like eating, watching television, talking with some friends, and sometimes does sweeping.
On first contact with the patient, seen patient lying on bed, weak, and with limited movements.
Eight days after admission, patient had great improvement, she can now smile, do sign language, move her extremities and with glow on her face. Though she still needs assistance, she can turn to sides now with ease.
5. Cognitive/ Perceptual
Patient needs to be oriented with the time and date though she is aware that she is currently admitted in the hospital. She is responsive (through gestures), coherent, and can relate to conversations. She even smile with jokes and wave her hands when
someone she used to see visited her. She speaks in a very low voice at present because the Endotracheal tube was just removed.
6. Sleep/Rest Patterns
Patient usually sleeps between 8:00PM – 9:00PM and wakes up around 5:00AM. She had afternoon nap everyday.
Now that she is admitted, she could hardly sleep because of her condition. Her vital signs need to be monitored hourly. She is also disturbed by her cough.
7. Self Perception/ Self Concept
Though the patient looses weight, she doesn’t look under weight at all. She is just weak because she is sick. She looks accommodating and friendly despite of her condition.
8. Role/ Relationship
Patient lives in her own house with her daughter and two grandchildren. They have close family ties. She is open to them with her feelings. She is fond of talking. She spends most of her time at home with her family.
The patient has five children, two boys (deceased) and three girls. Her husband died long time ago.
10. Coping/ stress tolerance
The patient is open to her family about her problems. But then, with regards to her sickness, she never complained about it. She kept it to herself as long as she can tolerate it.
11. Values and beliefs
Patient is a Roman Catholic but didn’t go to church. She didn’t join any religious community.
Nursing Problems
1.) Ineffective airway clearance2.) Impaired physical mobility3.)Risk for aspiration4.)Risk for impaired skin integrity5.) Impaired verbal communication
Nursing Diagnosis
1. Ineffective Airway Clearance related to increased
sputum production as evidenced by cough.2. Impaired Physical Mobility related to restrictive
devices3. Impaired Verbal communication r/t attachment to
mechanical ventilator.4. Risk for infection related to depressed immune
system5. Risk for aspiration r/t tube feedings and
secretions.
Acid fast stain
Specimen – sputum
Report: no acid fast bacilli seen
Nursing Care Plan
Cues Nursing Diagnosis
Scientific Basis
Goal and Outcome Criteria
Nursing Actions Rationale Evaluation
S: “Giubo ug kutasan ako mama ug maglisod siya ug ginhawa” As verbalized by the clients daughter.
O: 1.Received patient lying on bed, conscious, coherent
Actual
Ineffective Airway
Clearance related to increased sputum
production as
evidenced by
cough.
A cough is a protective reflex that cleanses the lower airways by an explosive expiration. Inhaled particles, accumulated mucus, inflammation or presence of a foreign
After 8 hours of nursing intervention, client’s airway is free of secretions as evidenced by eupnea and clear lung sounds after coughing or
To perform nursing care to help patient improved Airway
Independent;
1. Assess respiratory movements and use of accessory muscles.
Use of accessory muscles to breathe indicates an abnormal increase in work of breathing. (Gulanickl et. al.: 2007,480).
Goal Partially met
After 8 hours of nursing intervention, client’s airway was free of secretions as evidenced by eupnea and clear lung
afebrile, tachypneic and with mechanical ventilator support.
2. Change in respiratory status.
3. Patient demonstrate persistent coughing and dyspnea
4. Abnormal lung sounds
5. With pulse oximeter attached - T= 36.2oC
- P= 81
body initiates the reflex by stimulating the irritant receptors in the airway. The cough consists of inspiration, closure of glottis, and vocal cord, contraction of glottis, causing sudden, forceful expiration that removes the offending matter. The effectiveness of the cough depends on the depth
suctioning.
Specifically:
1. Client will maintain a stable breathing.
2. Client’s mucus will be thin and scant.
3. Client’s breath sounds are clear.
2. Assess cough for effectiveness and productivity.
3. Observe sputum color, amount, and odor and report significant changes.
Dependent:
1. Monitor pulse oximeter and ABGs.
Patients may have ineffective cough due to fatigue or thick tenacious secretions. (Gulanickl et. al.: 2007,480).
A sign of infection is discolored sputum. An odor may be present. (Gulanickl et. al.: 2007,480).
Hypoxemia may result from impaired gas exchange from build up of secretions.
sounds after coughing or suctioning.
Specifically:
1. Client maintained a stable breathing.
2. Client’s mucus wasthin and scant.
bpm- R= MV- BP=
90/55 mmHg.
of the inspiration and the degree to which the airway narrow, increasing the velocity of the expiratory gas flow. Cough occurs frequently in healthy individuals.
A persistent cough
indicates presence of disorder or a disease. An acute
non productive cough often
indicates
2. Monitor chest x-
ray reports.
Collaborative:
1. Consult the respiratory therapist for chest physiotherapy and nebulizer treatments, as appropriate and ordered.
ABGs provide data about carbon dioxide levels in the blood. (Gulanickl et. al.: 2007,480).
These determine progression of disease process. (Gulanickl et. al.: 2007,480).
Chest physiotherapy includes the techniques of postural drainage and chest percussion to loosen and mobilize
bronchitis or viral
pneumonia. A persistent
cough is commonly
caused by a tumor,
congestion, or
hypertensive airways. A cough
that produces purulent sputum usually
indicates infection, whereas a cough that produces
non purulent
sputum is non specific and merely indicates
2. Assist with bronchoscopy and thoracentesis, as appropriate.
secretions in smaller airways that cannot be removed by coughing or suctioning. A nebulizer may be used to humidify the airway to thin secretions to facilitate their removal. (Gulanickl et. al.: 2007,481).
Bronchoscopy is done to obtain lavage samples for culture and sensitivity and to remove mucous plugs; thoracentesis is done to drain associated pleural effusions.
irritation. (McCance, 2000:1150
3. Anticipate possible need for intubation if patient’s condition deteriorates.
4. Administer medications such as antibiotics and expectorants for productive coughs. Administe r inhaled bronchodilators and inhaled steroids, as prescribed, to open airway and decrease inflammation.
(Gulanickl et. al.: 2007,481).
Intubation may be needed to facilitate deep suctioning efforts and to provide source for augmenting oxygenation. (Gulanickl et. al.: 2007,481).
A variety of medications are available to treat specific problems. (Gulanickl et. al.: 2007,481).
Nursing Care Plan
Cues Nursing Diagnosis
Scientific Basis Goal and Outcome Criteria
Nursing Actions
Rationale Evaluation
S- Patient pointing her throat.-no verbalizationsO- • NGT inserted •Mechanical ventilator noted •Suction machine at
PotentialRisk for
aspiration r/t tube feedings
and secretion
s.
Crackles indicate static pulmonary secretions that need to be mobilized. This also includes accumulation of saliva on the airways .When this obstructs the airway the pulmonary tissues beyond the collapses and
After 8 hours of nursing intervention the patient will be able to maintain a patent airway
Specifically the patient and s.o.
To perform nursing care to prevent aspiration
Independent:1. Monitor level of consciousness.
A decreased level of consciousness is a prime factor for aspiration ( Gulanick/Mayers:2008
goal met:
After 8 hours of nursing intervention the patient was able to maintain a patent airway.
bedside. •patient pointing on her neck.
massive atelectosis results. (Smeltzer,Bare,,Hinkle cheever;2008,534)Pulmonary complications from NGT intubation occur because coughing and cleaning of the pharynx is impaired , because gas build up can irritate the phrenic nerve and because tubes may dislodged, retracting the distal and above the esophagogastric sphincter places the patient of risk for aspiration(Smeltzer,Bare,Hinkle,Cheever;2008,1180-1181)
will be able to:
1. Feel relief with concerns of secretions
2. Will be able to do basic suctioning procedures.
3. Have a secured NGT placement.
4. Have no abnormal breath sounds upon assessment.
2. Auscultate bowel sounds to evaluate bowel motility and assess for abdominal distention and firmness.
3. Position patient in an elevated upper body or side lying.
4. In Patients with artificial airways.
• Perform oral suctioning as needed.
5. In patients with NGT
pp18)
• Decreased gastrointestinal mobility increases the risk of aspiration because foods and fluids accumulate in the stomach(Gulanick/Mayers:2008;pp19
• This decreases the risk of aspiration by promoting the drainage and secretions away from the airway. (Gulanick/Mayers:2008pp.19)
•Reduces oro- pharyngeal secretions and reduces aspiration rising. ((Gulanick/Mayers:2008
Specifically the patient and s.o. was able to:
1. Feel relief of concerns about secretions.2. Able to do basic suctioning procedures.3. Have a secured NGT placement 4.Have no abnormal breathe sounds upon assessment5. Have a normal
5. Have normal breathing pattern.
• Check placement of tube before feeding by color or aspirate or listening for bubbling sounds upon air induction.
Collaborative:
6.Collaborate with respiratory therapist, as needed to determine cuff pressure(tubes)
7. Collaborate with the dietitians about having
pp.19)
•A placed tube may erroneously deliver tube feeding into the airway. ((Gulanick/Mayers:2008pp.19)
•On ineffective or over inflated cuff can increase the risk for aspiration. ((Gulanick/Mayers:2008pp.19)
breathing pattern.
blenderized diet for the patient.
Dependent:
8. Suction hourly as ordered by the physician.
9. Administer drugs in
appropriate preparation
as ordered by the physician.
• Appropriate mixture of food as well as balanced meal provides nutrients needed. ((Gulanick/Mayers:2008pp.63)
•To eliminate secretions (Doenges 63)
• Drugs in tablet forms must be crushed during administration (Doenges 63)
Nursing Care Plan
Cues Nursing Diagnosi
s
Scientific Basis
Goal and Outcome Criteria
Nursing Actions
Rationale Evaluation
S: Patient wrote on a piece of paper “gusto na ko mulakaw”
Actual
Impaired Physical Mobility related
to restrictiv
In place mechanical devices
are common to non
ambulatory patients
After 8 hours of nursing intervention the patient will be relieved from discomfort
To perform nursing care to help patient exercise in bed
1. Assess
>Restricted movement affects the
Goal met
After 8 hours of nursing intervention the patient was relieved from discomfort
O: 1.Received patient lying on bed, conscious, coherent afebrile, tachypneic and with mechanical ventilator support.
2. Pulse oximeter at right hand
3. IVF infused at right hand
4. Weak muscles
e devices
but in cases where
patients are able to walk,
the devices would
likely limit their
activities provided
that machines are easily altered by movemen
t examples would be
casts, neck
support and
ventilator. (Microsoft ® Encarta ® 2007)
Specifically
1. Patient will be free of complications of immobility, as evidenced by intact skin, absence of thrombophlebitis, & normal bowel pattern.
2. Patient will perform exercises in bed
3. Patient will move allowed body parts for
exercise.
patient’s ability to perform ADL’s effectively and safely on a daily basis.
2 Assess ability to perform ROM to all joints.
3 Encourage & facilitate early ambulation & other ADL’s when possible.
4 Provide positive reinforcement during activity.
ability to perform most ADL’s .
>This provides baseline measurement for the future evaluation and guides therapy.
>The sooner the patients becomes mobile, less chance that debilitation will occur.
>Patients may be reluctant to move or initiate new activity due to fear of falling. A positive approach allows
Specifically
1. Patient was free of complications of immobility, as evidenced by intact skin, absence of thrombophlebitis, & normal bowel pattern.
2. Patient was able to perform exercises in bed
3. Patient moved allowed body parts for exercise.
5 Evaluate patient’s performance in doing ADL’s.
6 Assess
the learner to feel good about learning accomplishments.
> Evaluating performance helps in improving once abilities & maximizing activities.Even patients who are temporarily immobile are at risk for effects of immobility such as skin breakdown, muscle weakness, thrombophlebitis, constipation, pneumonia, & depression.
> Regular
patient or caregiver’s knowledge of immobility & its complication.
7 Assess skin integrity
8 Assess elimination pattern.
9 Turn & position the patient every 2 hours or as needed.
10 Evaluate
examination of skin (especially over bony prominences) will allow for prevention or early recognition & treatment of pressure sores.
> Turning the patients optimizes circulation to all tissues & relieves pressure.
> Immobility promotes constipation
> It helps in evaluating patient’s outcome from nursing interventions.
the patient free of complications of immobility.
11 Assess patient’s difficulty in walking.
12 Encourage walking exercise interspersed with rest periods
13 Involve client/ SO in care, assisting them to learn ways of managing deficits.
14 Instruct client/ SO in
> It helps in determining factors that contributed to patient’s difficulty in moving
> To reduce fatigue.
> To enhance safety for client & SO/ caregivers.
> To reduce risk of falls
safety measures as individually.(eg. maintaining safe travel pathway, proper lightning.
> It helps in determining
patient’s outcome to be
effective or not.
Drug Study
Generic Name
Brand Name
Classification
Mechanism of Action
Indication
Contraindication
Side Effects
How Suppli
ed
Dosage/
Frequency
Nursing Intervention
Omeprazole
Omepron
Proton pump
Thought to be a gastric
Short term
Lactation. Use as
Headache,
IV infusio
40mgOnce a
1.Consider dosage
inhibitor pump inhibitor in
that it blocks the final
step of acid production inhibiting the H+ an/K+ ATPase
system at the
secretory surface of the gastric
parietal cell. Both basal
and stimulated
acid secretions
are inhibited.
Serum gastrin
levels are increased during the first 1 to 2
treatment of
active duodenal ulcer. Short
term (4-8
weeeks) treatme
nt of erosive
esophagitis
diagnosed by
endoscopy.
Maintain healing
of erosive
esophagitis.
Treatment of
heartburn and other
maintenance therapy
for duodenal
ulcer disease.
OTC use in those who
have trouble of
pain swallowing food, are vomiting blood, or excreting bloody or
black stools.
abdominal
pain, diarrhea, N&V, URTI,
dizziness, rash.
n day adjustment in those with impaired hepatic function especially when used for maintaining clients with erosive esophagitis.2.List reason for therapy, triggers, frequency, characteristics of S&S, other agents trialed.3.Record abdominal assessment, radiographic/endoscopic findings, and H. pylori result.4.Administer one hr before giving meal.5. Report any changes in urinary
weeks of therapy and
are maintained
at such levels during the course of therapy.
(Spratto et.al.:2008:1
159)
symptoms
associated with GERD.
elimination, pain, discomfort, or persistent diarrhea.6. Avoid activities that require mental alertness until drug effects realized; may cause dizziness.7. For short term use only, drug inhibits total gastric acid secretion. Side effects of prolonged therapy and suppression of acid secretion alter bacterial colonization and lead to hypoclorhydria and hypergastrenemia which may cause an increase risk for gastric
tumors.
Drug Study
Generic Name
Brand
Name
Classification
Mechanism of Action
Indication Contraindication
Side Effects
How Suppli
ed
Dosage/
Frequency
Nursing Intervention
Piperacillin
sodium and
Tazobactam
sodium
Zosyn,
Peptaz
Antibiotic,
Penicillin
A combination
of Piperacillin sodium and Tazobactam sodium, a
beta-lactamase inhibitor.
Tazobactam inhibits beta-
lactamases, thus
ensuring activity of piperacillin
against beta-
lactamase-producing
microorganis
(1)Appendicitis complicated by rupture or abscess and peritonitis caused by piperacillin-resistant, beta-lactamase producing strains of Escherichia coli, Bacteroides fragillis. (2)Community Acquired Pneumonia
Hypersensitivity to
penicillins, cephalosporins, or beta-lactamase inhibitors.
Diarrhea, constipation, N&V, dyspepsi
a, headache, rash, rhinitis,
dyspnea, abdominal pain.
IV infusio
n
4.5 gm IV q 8 hours
1.For IV administration or infusion, reconstitute the powder for injection with 5 ml suitable diluent/gram piperacillin. IV diluents that can be used include 0.9% NaCl, sterile water for injection, dextran 6% in saline,D5W,KCl 40mEq, bacteriostatic saline/parabens, bacteriostatic water/parabens
ms. Thus Tazobactam
broadens the
antibiotic spectrum or piperacillin
to those bacteria normally
resistant to it. (Spratto
et.al.:2008:1269)
of moderate severity caused by piperacillin-resistant, beta-lactamase producing strains of Haemophilus influenzae. (3)Moderate to severe nosocomial pneumonia caused by piperacillin-resistant, beta-lactamase producing strains of Acinetobacter baumanii.
(4)
, bacteriostatic saline/benzyl alcohol,bacteriostatic water/benzyl alcohol.2.Note reasons for therapy, type, location, characteristics of S&S.3. List any sensitivity to penicillins, cephalosporins, beta-lactamase inhibitors, or other allergens.4. List drug prescribed to ensure none interact unfavorably. Use of heparin and oral anticoagulants may require dosage adjustments.
rash, diarrhea, GI upset, lack of response or worsening of
condition.
Drug Study
Generic Name
Brand
Name
Classification
Mechanism of
Action
Indication Contraindication
Side Effects
How Suppli
ed
Dosage/ Frequen
cy
Nursing Intervention
clopidogrel
wintop
Anti-platelet
drug
Inhibits platelet
aggregation by
inhibiting binding of adenosin
e diphosphate (ADP)
to its platelet receptor
and susbsequent ADP
meditative
activation of
glycoprotein
Reduction of MI,
stroke and ucercular death in patients
with atheroscler
osis documented by recent stroke, MI
or established peripheral
arterial disease
Lactation, active
pathological bleeding such us
peptic ulcer or
intracranial hemorrhage
Appendage
disorders,
headache, chest pain, flu-
like sympto
ms
Tablets 75 mg
Clopidogral 75
mg i tab, PO,OD
>Do not cofuse with antidepreesant > Document otheros dehoric event or established peripheral arterial disease requiring therapy.> Asses for active bleeding as with ulcers or intracranial bleeding.> list all drugs prescribed/consumed esp. OTC.> Consider 5 rights in giving meds.
GPII/IIa complex. Effect on receptors
is irreversib
le thus platelets
are affected
for remainder of their lifespan. (MIMS.co
m)
> Explain the purpose of the medication.> obtain baseline V/S.> Document the
procedure.
Drug Study
Generic Name
Brand Name
Classification
Mechanism of
Action
Indication
Contraindication
Side Effects How Supplied
Dosage/ Frequen
cy
Nursing Interventio
n
Acytylcystien
Flulmucil
sachet
Mucolytics
Decrease the
production of
mucus at respiratory tracts
by stimulatin
g the productio
n of glutathiance thus decreasin
g the viscosity
of secretion
s.
Acute and
chronic respiratory tract affection
s with abundant mucus secretio
ns.
Contraindicated to
patients having
asthma. Patients with
history of peptic ulcer.
Urticaria bronchospasm, nausea,
vomiting.
100 mg /200mg sachetInhalation 100mg/ml
Syrup 100mg/5ml x 150
ml
200 mg 1 sachet + socc
h20 NGT bid
asses drug expiration date>asses for drug tolerance characterized >obtain baseline v/s>take the drug with meals>advice increase fluid intake >consider patients safety>evaluate
effectiveness of drug>chart procedure
Drug Study
Generic Name
Brand Name
Classification
Mechanism of
Action
Indication Contraindication
Side Effects
How Suppli
ed
Dosage/ Frequen
cy
Nursing Interventio
n
Simvastatin
Vidastat
Dyslipidemic Agent
Reduction of low density
lipprotien cholesterol in that following inhibition
of the HWG-COA
reductase
activity, the LDL receptor activity on the liver is
increased and this
In CHD: Reduce risk of death and non fatal MI. Reduce risk of strike and transient ischemic attacks. In hypelipidemia: an adjuct to the diet to reduce elevated total-c, LDL-C , apolipoprotien B and TG in patients with primary hyper choles terolemia, hanozygous
Active liver disease or
unexplained persistent
elevations of serum trans
aminoses parphyria, pregnancy lactation
Constipation
dyspepsia
flatulence
Tablet 10
mg,20 mg, 30 mg,40
mg
Vidastat 40g I
tab NGT OD q8hr
Asses drug expiration date >consider the 5 rights of drug administration.>obtain baseline data >dissolve solution thoroughly> encourage high fiber diet, fluid.> inform patient about the mechanism
leads to increase
d removal of LDD
cholesterol.
(KIMS.com)
familial hyrecholesterogous or mixed hyperlipidemia.
(MIMS.co
of drugs>chart the procedure>evaluate effectiveness of drugs.
Drug Study
Generic Name
Brand Name
Classification Mechanism of
Action
Indication Contraindication
Side Effects
How Supplied
Dosage/ Frequen
cy
Nursing Interventio
n
Midozalam
Dormicum
Benzodiazepiness sedative
hyponotics
Inhibits sympath
etic nervous system
activation and
initiates sedation hyponoti
cs
Disturbances of sleep rhythm, insomnia esp.difficulty in falling asllep either initially or after premature awakening.Sedation
in premed before
surgical or
Premature infants
myasthenia gravis
Insomnia in
psychosis severe depressi
on
Ampule 5mg/1ml5mg/5ml,15 mg/3ml
Film coated tablet 15 mg
IVTT 2.8 mg OD
1. Assess level of sedation and level of consciousness through out and for 2-6 hour following administration2. Monitor blood pressure, pulse and respiration continuously during administrat
diagnostic procedure
s, induction
and maintena
nce of anesthesi
a. (MIMS.co
m)
ion 3. Administer IM doses deep into muscle4. In form the patient that this medication will decrease mental recall of the procedure5. Instruct patient to inform health care professional prior to administration if pregnancy is suspected
>asses drug expiration date>asses for drug tolerance characterized >obtain baseline v/s>instruct the patients to remain in a stable position for 2-3 hrs. >advice
increase fluid intake >consider patients safety>evaluate effectiveness of drug>chart procedure
Drug Study
Generic Name
Brand Name
Classification
Mechanism of Action
Indication Contraindication
Side Effects
How Suppli
ed
Dosage/ Frequen
cy
Nursing Interventio
n
rebamipide
Mucusta
ANTACIDS Rebamipide is a
mucosal protective agent and
is postulated to increase
gastric blood flow, prostaglan
din biosynthes
is and decrease
free oxygen radicals.
For acute gastritis
and exacerbati
on of chronic
gastritis, gastric ulcers
Lactation. Rash, pruritus,
constipation,
diarrhoea, nausea.
Tablet100 mg
Mucosta 1 tab
OD TID
1. Chedk renal studies to check renal function is normal2. Check the pattern of bowel elimination.3. Record the gastric pain being experienced.4. report for coffee ground
stools5. if the patient is pregnant has edema or hypertensive, use low sodium antacids
Drug Study
Generic Name
Brand
Name
Classification
Mechanism of Action
Indication
Contraindication
Side Effects
How Supplied
Dosage/ Frequen
cy
Nursing Interventio
n
prednisone
pred corticosteroids
The anti-inflammatory effect is
due to inhibition
of prostaglan
din synthesis, the drug
also inhibits
accumulation of
machrophages and
leukocytes at sites of inflammati
on and
Allergis and
edematous
respiratory and
neoplastic
diseases
Gastric & duodenal
ulcers, systemic fungal &
certain viral infections, glaucoma,
psychoses or severe
psychoneuroses; live vaccines;
hypersensitivity to
glucocorticoids.
Insomia, nosia and vomiting, GI upset, fatique,
dizziness, muscle
weakness, increased hunger/thirst, joint
pain, decreased diabetic control.
tablets:1mg, 5mg, 10mg, 20
mg, 50 mgOral
solution: 5mg/5ml;
syrup 5mg/5ml;
20 mg 1 tab NGT
BID
1. note reasons for therapy, type, onset, characteristics of signs and symptoms, clinical presentation2. monitor CBC, ESR, electrolytes, BP, blood sugar, weights and mental
inhibits phagocytos
is and lysosomal enzyme release.
status.3. with COPD provide rescue doses and instruct client how and when to use.4. with chronic pain, titrate dose to assess for relief
Drug Study
Generic Name
Brand Name
Classification
Mechanism of Action
Indication
Contraindication
Side Effects How Suppli
ed
Dosage/ Frequen
cy
Nursing Intervention
Ivabradine HCl
coralan
AntianginaIvabradine
Ivabradine is a pure heart rate-lowering agent, acting by selective and specific inhibition of the cardiac pacemaker /current that controls the spontaneous diastolic depolarisation in the sinus node and regulates
Chronic angina pectotis
Resting heart rate <60 bpm prior to
treatment, cardiogenic shock, acute MI, severe
hypotension (<90/50 mmHg), severe hepatic
insufficiency, sick sinus syndrome, SA block,
severe heart failure,
pacemaker-
Luminous phenomena (phosphenes), blurred
vision, bradycardia
, 1st degree AV
block, ventricular extrasystol
es, headache, dizziness.
film-coated tablet 5 mg, 7.5 mg
5 mg 1 tab NGT
OD
1. Assess the client for gastrointestinalupset and peripheral edema.2. Assess pain and limitation of movement ; note type, location & intensity prior to & at the peak following administration.
heart rate. The cardiac effects are specific to the sinus node with no effect on intra-atrial, atrioventricular or intraventricular conduction times, nor on myocardial contractility or ventricular repolarisation.
dependent patient, unstable
angina, 3rd degree AV-
block. Concomitant
potent CYP3A4
inhibitors. Pregnancy &
lactation.
3. Monitor vital signs and check for peripheral edema.d. Don’t breastfeed while taking this drug.4. Discontinue drug and notify physician if signs and symptoms of hypersensitivity occur.5. Caution the patient to avoid concurrent use of alcohol with this medication.
Drug Study
Generic Name
Brand Name
Classification
Mechanism of
Action
Indication Contraindication
Side Effects
How Supplie
d
Dosage/ Frequen
cy
Nursing Interventi
on
Co amoxicl
av
Agcomen
Amoxicillin and
enzyme inhibitor
Prevents the
plorefiration of of infecting bacteria
by inhibitng synthesis
of bacterial capcule. (mims.co
m)
Bacterial infections caused by amoxicillin-resistant β-lactamase producing
strains.These include
actinomycosis, biliary
tract infections, bronchitis,
endocarditis,
gastroenteritis, typhoid
& para typhoid
Pregnancy & lactation. Elderly & neonates.
Severe renal impairment.
Diarrhea, hepatitis
& cholestati
c jaundice. Erythema multiform
e, Stevens-Johnson syndrome, toxic
epidermal
necrolysis &
exfoliative
dermatiti
tablet 625 mg
625 mg 1 tab
NGT q 8 hrs
a. Assess for allergy to penicillin.b. Instruct to take the entire quantity of drug exactly as prescribed even after she feels better.c. Encourage to increase fluid intake.
Stimulates beta 2 receptors on the bronchi. Causes less tachycardia and is longer acting. (Woods, Sratto, 28)
Management of
reversible bronchosp
asm associated
w/ obstructive
airway diseases
eg bronchial asthma, COPD
Hypertrophic
obstructive cardiomyop
athy or tachyarryth
mia. Hypersensitivity to soya lecithin or
related food products (for MDI).
Headache,pain,
influenza, chest pain;
nausea. Bronchitis, dyspnea, coughing,
pneumonia, bronchospa
sm, pharyngitis,
sinusitis, rhinitis.
Pulmoneb
solution 2.5 mL
1 neb now
1. Obtain record and baseline vital sings2. Assess the presence of palpitations and dysrhythmias3. Perform assessment of the patient’s mental status4. Warm patient about
possible paradoxical bronchospasm5. use cautiously to patients with CV disorders, hypertension, renal disease and diabetes.
IVF STUDY
Student’s Name: Group 4 Section R Date of Submission: January 8, 2009Area: Chong Hua Hospital Cliinical Instructor: Benita Edelia D.
AgramonPatient’s Name: Patient X Doctor: Dr. R. GoRoom/Bed No.: C-414 Date of Admission: December 13, 2008Age: 66 years old Hospital No.: 0-800-22500-773Status: Widowed Diet: Liquid diet
Type of solutio
n
Classification
Content Mechanism of
action
Indications Contraindications
How supplie
d
Dosage Nursing Responsibiliti
es
0.9% Sodium chlorid
e solutio
n
Isotonic Each 1000 mL contains 900mg of Sodium ChlorideOsmolarity:308 mOsm/L
Electrolytes in 1000
Replaces sodium
and chloride
and maintains
levels (Lippincott Williams & Wilkins:
2005, 879).
Fluid and electrolyte
replacement in
hyponatremia caused by electrolyte loss or in
severe salt depletion.
Contraindicated in patients with conditions in which sodium chloride administration is detrimental. >contraindicated in patients
with with increased,
normal, or only
I.V. 1 liter
IVF PNSS 1 liter @
30 gtts/mi
n
1. Monitor electrolyte levels (Lippincott Williams & Wilkins: 2005, 879).2. Explain use and Administration of drug to patient and family
mL:Sodium… 154 mmolChloride…154 mmol
slightly decreased electrolyte
levels.
(Lippincott Williams & Wilkins: 2005, 879).3.Tell Patient to report adverse reactions promptly (Lippincott Williams & Wilkins: 2005, 879).4.Regulate flow rate as ordered (Lippincott Williams & Wilkins: 2005, 879).6.Check on skin integrity for redness, edema, swelling and pain (Lippincott Williams & Wilkins: 2005,
879).7.Do not let the bottle be consumed totally to prevent air embolism (Lippincott Williams & Wilkins: 2005, 879).8.Check mfor any bubbles present on the I.V. line (Lippincott Williams &
Wilkins: 2005, 879).
Type of solution
Classification Content Mechanism of action
Indications Contraindications
How suppli
ed
Dosage Nursing Responsibiliti
es
Dopamine
Sympathomimetic, direct acting and
indirect acting
Synthetic
Dopamine
Dopamine is the
immediate precursor of epinephrine in the body. Exogenously administered,
it produces direct
stimulation of beta-1
receptors and variable (dose-
dependent) stimulation of
alpha receptors
(peripheral vasoconstricti
on). Will cause a
release of
Adjunct to standard measures
to improve:
BP, Cardiac output, urine
output, in treatment of shock,
unresponsive to fluid replaceme
nt.
Pheochromocytoma,
uncorrected tachycardia, ventricular
fibrillation, or arrhythmias.
Pediatric clients.
IVF 500 ml
PB Dopamine IV @
30 gtts/min
1. Dilute just prior to administration, solution stable for 24 hr at room temperature, protect from light.2. To prevent fluid overload, may use more concentrated solutions with higher doses.3. Administer using an electronic infusion device. Carefully reconstitute
norepinephrine from, as its storage sites. These action
results in increased
myocardial contraction,
CO, and SV as well as
increased renal blood
flow and sodium
excretion. Exert little effects on DBP and
induces fewer
and calculate dosage.4. When discontinuing, gradually decrease dose, sudden cessation may cause marked hypotension.5.Monitor VS, I&O, and ECG; titrate infusion to maintain SBP as ordered.6. Be prepared to monitor CVP and PAWP. Report ectopy, palpitations, anginal pain, or vasoconstriction.
7. Explain to the family that drug administered IV to improve cardiac function thus increasing BP and improving urine output.8. Report any
chest pain, increase
SOB, headaches,
or IV site pain.
Type of solutio
n
Classification
Content Mechanism of action
Indications
Contraindications
How supplie
d
Dosage Nursing Responsibiliti
es
D5NSS Hypertonic Each 1000 mL contains 1000mg of Sodium ChlorideOsmolarity:500 mOsm/L
Electrolytes in 1000 mL:Sodium… 354 mmolChloride…354 mmol
The solution has a higher sodium concentration than to the intracellular area thus by virtue of osmosis the water is taken out of the cell the cell shrinks. This increases the plasma volume of
Rehydrate; has free
water, salt and calories
Contraindicated in patients with conditions in which sodium chloride administration is detrimental. >contraindicated in patients
with with increased,
normal, or only slightly
decreased electrolyte
levels.
I.V. 1 liter
IVF D5NSS 1 liter @ 30
gtts/min
1. Monitor electrolyte levels (Lippincott Williams & Wilkins: 2005, 879).2. Explain use and Administration of drug to patient and family (Lippincott Williams & Wilkins: 2005, 879).3.Tell Patient
the blood. to report adverse reactions promptly (Lippincott Williams & Wilkins: 2005, 879).4.Regulate flow rate as ordered (Lippincott Williams & Wilkins: 2005, 879).