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Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician
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Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Dec 18, 2015

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Page 1: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Monash University-2009Alcohol and Other Drugs

Presenter: Effie Moraitis

Senior Clinician

Page 2: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Topics covered today:

What is a drug? – some definitions Theoretical models of drug use Harm minimisation Dependence Syndrome Classification of drugs and their effects AOD assessment – some important points Stages of Change model Effects of Alcohol and Marijuana use. Especially

Neurological impact. Withdrawal Symptoms Treatment Options

Page 3: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Making sense of AOD issues:

What is a drug? Who uses drugs? Theoretical models of drug use Harm minimisation Dependency, tolerance and

withdrawal Patterns of drug use

Page 4: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

What is a drug? Definitions:

World Health Organisation:“Any substance which when taken into the

body, alters its function physically and/or psychologically, excluding food, water and oxygen” (cited in McCallum 1994 p 90 – WHO 1994)

“Any substance the people consider to be a drug, with the understanding that this will change from culture to culture and from time to time” (Krivanek 1995 p 2)

Page 5: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Who Uses Drugs?

Drug use occurs across cultures, suburbs, genders and class systems.

Common thought is that people in lower socioeconomic communities use more drugs than those from affluent communities.

True or False?

Page 6: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Theoretical Models of drug use:

Moral ModelKey assumption:

using drugs is morally wrong and anti-social

Intervention:spiritual direction, gaol, providing an environment that promotes pro-social values

Page 7: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Theoretical Models of drug use:

Disease Model (Medical Model)Key assumptions:

Some people have a natural predisposition to drug useDependency is controlled by physiological / genetic factorsDependency will inevitably result in a loss of control and progression of their condition

Page 8: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Theoretical Models of drug use:

Disease Model con’tInterventions:

- Total abstinence- Self-help (eg: 12 step programs)- Supporters of this model suggest that a person addicted to a drug / s will be unable to control their substance use.

Page 9: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Theoretical Models of drug use:

Social learning model:Key assumptions:

Focuses on the interaction between the environment, the individual and the drug. Drug use is learntIntervention:Learning new coping strategiesMay use cognitive restructuring techniques

Page 10: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Theoretical Models of drug use:

Public Health Model:Key assumptions:Looks at the availability of the drug, cost, the properties of the substance, individual factors and socio-political factors (advertising, economic gains from drug use, peer pressure)Interventions:Education, political actions, legislation

Page 11: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Harm minimisation approach:

Has underpinned Australia’s drug strategy since 1985

Harm minimisation accepts that the use of drugs is a part of life and that on many occasions, drug use is non-problematic. It also recognises that drug use can cause harm amount the people that use and the wider community. Harm minimisation seeks to reduce drug related harm

Page 12: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

National Drug Strategic Framework:

The National Drug Strategy focuses on three core elements of harm reduction

- demand reduction (prevention)- supply reduction (law enforcement)- harm reduction (education, information

Page 13: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Dependence, Tolerance and Withdrawal:

Dependence:Maladaptive pattern of substance use, leading

to clinically significant impairment or distress.

The substance is often taken in larger amounts or over a longer period than intended

There is a persistent desire or unsuccessful efforts to cut down or control substance use.

Page 14: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Dependence, Tolerance and Withdrawal:

Tolerance:The need for increased amounts of the

substance to achieve intoxication or the desired effect.

This may vary- across individuals- across substances- across physiological systems

Page 15: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Dependence, Tolerance and Withdrawal:

Withdrawal:Maladaptive behavioural change (which may

be the opposite to the acute effect of the substance

Withdrawal syndromes may change according to the substance

The same or closely related substances may be taken to relieve or avoid withdrawal symptoms

Page 16: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Patterns of consumption and types of drug use:

Controlled use Experimental use Social / recreational use Circumstantial use / situational Intensive use Dependency

Page 17: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Drug Classifications:

Drugs are often classified in two ways: Legal status Central nervous system effects

Classifications:DepressantsStimulants

Hallucinogens

Page 18: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

An AOD assessment:

What are some important points to cover in an AOD

assessment?

Page 19: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Assessment – some points:

Psychosocial history Substance use history – type of substances

used, frequency, quantity, when and how they use, circumstances

Medical history and current medications Psychiatric history Four Ls – Liver, Lover, Livelihood, Legal

Page 20: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Stages of Change Model:

DiClementi & Prochaska (1986) Illustrates that change is a process,

and change is a process that can take time

Is a useful tool in identifying where people are at in their change process

People can go forwards or backwards in this model.

Page 21: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Six stage model: Pre-contemplative stage

Person does not see they have a problem Contemplative stage

Person is weighing up cost / benefits to change Preparation stage

Person is preparing to change Action stage

Person is actively pursuing change Maintenance stage

Person is maintaining the change Relapse stage

Person returns to previous levels of drug use

Page 22: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Alcohol: What is it? Alcohol is ethyl alcohol or ethanol. It is a

natural product of fermenting sugars. It’s usually made from grains such as hops, barley, rice and/or fruits. It can also be made from other plants.

The concentration of alcohol varies widely according to the type of alcoholic drink. Hence different standard drink variations.

ROA – Oral/Swallowed

Page 23: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Side Effects

Feelings of relaxation, lowered inhibition, increased sociability.

In higher doses alcohol can cause dizziness, nausea, slurred speech, slower reflexes, sleepiness, dehydration and bad judgement.

In even higher doses it can cause blackouts, organ failure, liver damage, coma and in extreme cases death.

Page 24: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Neurological Side Effects

When Alcohol hits your lips your whole body is affected.

Within the lining of your mouth a small percentage of alcohol is absorbed. It irritates the mouth lining as well as the oesophagus, acting like an anaesthetic.

From there…

Page 25: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Neurological side effects con’t.

Alcohol travels to your stomach This is where it’s absorbed into the

bloodstream. Then it continues to the small intestine and

from here it it is completely absorbed into the bloodstream. Alcohol can reach the small intestine within 5 minutes.

At this point the alcohol can reach every cell in the body

Page 26: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Effects of alcohol on the body

Alcohol shares many properties with water. It is highly soluble in water and travels through the body as water does.

In it’s circulation through the body, the alcohol reaches the brain.

The feelings of intoxication now begin. They are dependant on concentration of alcohol in the body and how fast it reaches the small intestine, the strength, whether there has been food consumption, age gender, body size..

Page 27: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Effects con’t

The liver metabolises 90% of the alcohol in your body. The rest is eliminated by perspiration, or via kidneys and lungs.

The remaining alcohol continues its circulation throughout the body.

Where to from here?

Page 28: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Effects con’t

Alcohol’s effect on the brain is abnormal, as the brain is usually protected from chemicals and drugs by the “blood/brain barrier” which acts as a filter.

The simple molecular structure of alcohol allows the penetration into the brain. This occurs in the frontal lobe.

Page 29: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Effects con’t

At this point there is a loss of reason, caution and inhibitions.

At the Parietal Lobe there is a loss of fine motor skill, slower reaction, reflex time and shaking.

In the Temporal lobe occurs the slurred speech as well as impaired hearing.

At the Occipital Lobe blurred vision and judgement, and loss of vital functions

Page 30: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Chronic Alcohol Consumption

When people consume large quantities of alcohol, they develop a Thiamine Deficiency(Vitamin B1).

This causes the neurological disorder called Wernicke-Korsakoff Syndrome. Wernicke’s encephalopathy and Korsakoff’s psychosis are the acute and chronic phases of this condition especially affecting short term memory.

An absence of Thiamine results in an inadequate supply of energy to the brain.

Page 31: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Chronic Consumption con’t

In chronic heavy alcohol consumers, the frontal lobes of the brain shrink. This is probably partly due to loss of water and partly due to cell death.

The lobes may expand again if the person stops drinking, but evidence of cell death remains in impaired function.

Page 32: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Did You Know?

When people become intoxicated it is common to feel warmth, however this is misleading. Alcohol acts as a vasodilator, dilating surface blood cells. This actually expands blood vessels causing people to lose body heat.

Page 33: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Withdrawal Symptoms of Alcohol

Sweating, facial flushing Tremors Agitation Palpitations, hypertension Poor appetite, nausea, vomiting, diarrhoea Poor sleep, anxiety Cravings, strong desire to drink Poor concentration Headaches

Page 34: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

More Serious Symptoms

Severe hypertension Seizures Hallucinations, delirium Arrhythmias Precipitation/ exacerbation of underlying

medical or psychiatric disorders Mood swings 

Page 35: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Illnesses caused by alcohol

Sleep and sexual disorders Psoriasis of the liver Psychotic and mood disorders Foetal Alcohol Syndrome Depression Heart failure Wernicke-Korsakoff Syndrome

Page 36: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Marijuana: What Is It?

Marijuana also known as Cannabis, is a green, brown, or grey mixture of dried, shredded leaves, stems, seeds, and flowers of the hemp plant. You may hear marijuana called by street names such as pot, herb, weed, grass, boom, Mary Jane, gangster, or chronic. There are more than 200 slang terms for marijuana.

Sinsemilla (sin-seh-me-yah; a Spanish word), hashish ("hash" for short), and hash oil are stronger forms of marijuana.

Page 37: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

The Classification

All forms of marijuana are mind-altering. In other words, they change how the brain works. They all contain THC (delta-9-tetrahydrocannabinol), the main active chemical in marijuana. They also contain more than 400 other chemicals. Marijuana's effects on the user depend on the strength or potency of the THC it contains (5). THC potency of marijuana has increased since the 1970s but has been about the same since the mid-1980s.

Page 38: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Effects of Marijuana Impaired perception Diminished short-term memory Loss of concentration and coordination Impaired judgement Increased risk of accidents Loss of motivation Diminished inhibitions/Increased heart rate

 

Page 39: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Effects of Marijuana con’t

Anxiety, panic attacks, and paranoia Hallucinations/Delusions Damage to the respiratory,

reproductive, and immune systems Increased risk of cancer Psychological dependency

Page 40: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Neurological Effects of Marijuana

When someone smokes Marijuana, THC rapidly passes through the bloodstream.

This carries the chemical to organs throughout the body, including the brain

Page 41: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Neurological effects con’t.. Cannabinoids is an active ingredient of

Marijuana. The most psychoactive cannabinoids chemical in Marijuana that has the biggest impact on the brain is tetrahydrocannibol, or THC. THC is the main active ingredient in marijuana because it affects the brain by binding to and activating specific receptors, known as cannabinoid receptors. "These receptors control memory, thought, concentration, time and depth, and coordinated movement. THC also affects the production, release or re-uptake (a regulating mechanism) of various neurotransmitters

Page 42: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Neurological Effects of Marijuana con’t..

Neurotransmitters are chemical messenger molecules that carry signals between neurons. Some of these affects are personality disturbances, depression and chronic anxiety. Psychiatrists who treat schizophrenic patients advise them to not use this drug because marijuana can trigger severe mental disturbances and cause a relapse.

Page 43: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Memory Loss

When one's memory is affected by high doses of marijuana, short-term memory is the first to be triggered. Marijuana's damage to short-term memory occurs because THC alters the way in which information is processed by the hippocampus, a brain area responsible for memory formation. One region of the brain that contains a lot of THC receptors is the hippocampus, which processes memory.

Page 44: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Emotional Impairment

Marijuana also impairs emotions. When smoking marijuana, the user may have uncontrollable laughter one minute and paranoia the next. This instant change in emotions has to do with the way that THC affects the brain's limbic system. The limbic system is another region of the brain that governs one's behaviour and emotions.

Page 45: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Cognitive Impairment

The chemicals in Marijuana bring cognitive impairment and troubles with learning for the user. Smoking [marijuana] causes some changes in the brain that are like those caused but cocaine, heroin, and alcohol.

Page 46: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Withdrawal Symptoms of Marijuana

Anxiety, agitation, restlessness, irritability Nausea Dysphoria, lethargy Cravings, strong desire to use Sleep disturbances (including vivid dreams,

nightmares, insomnia) Sweating Headaches Mood disturbances 

Page 47: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Illnesses caused by marijuana

Cannabis is linked with Mental Health Disorders. If there is a predisposition in a persons family history

of a mental health disorder, Marijuana can trigger it to occur.

Short Term Memory loss

Research has begun on potential Learning difficulties experienced by children whose mothers used Marijuana during pregnancy and breastfeeding.

Page 48: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Treatment Options Treatment for any drug is more effective when

tailored to the specific individual requirements. It can involve a combination of methods

including: Medication and GP/Psychiatric Involvement Individual Counselling Group Therapy Home Based Withdrawal Residential Withdrawal Long Term Rehabilitation And more…

Page 49: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

Treatment Options con’t

In Victoria there are over 1,000 Alcohol and Other Drug Treatment Services.

Inclusive in these are 24/7, free and immediate Counselling, Information and referral services specifically for anyone who has any Alcohol and Other Drug related concerns. These services are anonymous and confidential (within confidentiality limitations).

Page 50: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

24/7 Services

DirectLine: 1800 888 236 for consumers and significant others who are experiencing Alcohol and Other Drugs related concerns.

DACAS: 1800 812 804 Drug and Alcohol Clinical Advisory Service for Health Professionals.

This service has 24/7 Addiction Specialist Medical Consultants.

Page 51: Monash University-2009 Alcohol and Other Drugs Presenter: Effie Moraitis Senior Clinician.

24/7 Services con’t

CounsellingOnline: www.counsellingonline.org.au A web-Based Counselling, Information, Referral and Support service 24/7 for consumers and significant others specifically related to Alcohol and Other Drug concerns. This is a National Service.

All services are staffed by Professional Counsellors