I. INTRODUCTION Cerebrovascular disease is a group of brain dysfunctions related to disease of the blood vessels supplying the brain. Hypertension is the most important cause; it damages the blood vessel lining, endothelium, exposing the underlying collagen where platelets aggregate to initiate a repairing process which is not always complete and perfect. Sustained hypertension permanently changes the architecture of the blood vessels making them narrow, stiff, deformed, uneven and more vulnerable to fluctuations in blood pressure. A stroke is caused by the interruption of the blood supply to the brain, usually because a blood vessel bursts or is blocked by a clot. This cuts off the supply of oxygen and nutrients, causing damage to the brain tissue. The most common symptom of a stroke is sudden weakness or numbness of the face, arm or leg, most often on one side of the body. Other symptoms include: confusion, difficulty speaking or understanding speech; difficulty seeing with one or both eyes; difficulty walking, dizziness, loss of balance or coordination; severe headache with no known cause; fainting or unconsciousness. The effects of a stroke depend on which part of the brain is injured and how severely it is affected. A very severe stroke can cause sudden death. The 1990 Global Burden of Disease (GBD) study provided the first global estimate on the burden of 135 diseases, and cerebrovascular diseases ranked as the second leading cause of death after ischemic heart disease. During the past decade the quantity of especially routine mortality data has increased, and is now covering approximately one-third of the world’s population. The increase in data availability provides the possibility for updating the estimated global burden of stroke. Data on causes of death from the 1990s have shown that cerebrovascular diseases remain a leading cause of death.
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I. INTRODUCTION
Cerebrovascular disease is a group of brain dysfunctions related to disease of the blood vessels supplying the brain. Hypertension is the most important cause; it damages the blood vessel lining, endothelium, exposing the underlying collagen where platelets aggregate to initiate a repairing process which is not always complete and perfect. Sustained hypertension permanently changes the architecture of the blood vessels making them narrow, stiff, deformed, uneven and more vulnerable to fluctuations in blood pressure.
A stroke is caused by the interruption of the blood supply to the brain, usually because a blood vessel bursts or is blocked by a clot. This cuts off the supply of oxygen and nutrients, causing damage to the brain tissue.
The most common symptom of a stroke is sudden weakness or numbness of the face, arm or leg, most often on one side of the body. Other symptoms include: confusion, difficulty speaking or understanding speech; difficulty seeing with one or both eyes; difficulty walking, dizziness, loss of balance or coordination; severe headache with no known cause; fainting or unconsciousness.
The effects of a stroke depend on which part of the brain is injured and how severely it is affected. A very severe stroke can cause sudden death.
The 1990 Global Burden of Disease (GBD) study provided the first global estimate on the burden of 135 diseases, and cerebrovascular diseases ranked as the second leading cause of death after ischemic heart disease.
During the past decade the quantity of especially routine mortality data has increased, and is now covering approximately one-third of theworld’s population. The increase in data availability provides the possibility for updating the estimated global burden of stroke.
Data on causes of death from the 1990s have shown that cerebrovascular diseases remain a leading cause of death.
In 2001 it was estimated that cerebrovascular diseases (stroke) accounted for 5.5 million deaths world wide, equivalent to 9.6 % of all deaths Two-thirds of these deaths occurred in people living in developingcountries and 40% of the subjects were aged less than 70 years.
Additionally, cerebrovascular disease is the leading cause of disability in adults and each year millions of stroke survivors has to adapt to a life with restrictions in activities of daily living as a consequence of cerebrovascular disease. Many surviving stroke patients will often depend on other people’s continuous support to survive.
II. OBJECTIVES
GENERAL OBJECTIVES
1. To be able to discuss the effect, signs and symptoms of the disease,
Cerebrovascular Disease.
2. How to diagnose, prevent and the treatment should the nurse give for
the patient full recovery.
SPECIFIC OBJECTIVES
1. To be able to discuss patients background ( lifestyle, history of the
past illness, family health history) to show how may this effect on the
occurrence of this disease.
2. To be able to discuss the anatomy and the physiology of the heart, for
you to be able to understand where the infection takes place.
3. To be able to discuss the pathophysiology of cardiovascular diseases
and also to know and understand the etiology of the disease.
4. To be able to discuss the patient activities of daily living. To know if
there’s a factor that triggers the disease
5. To be able to discuss, nursing care plan for our patient.
6.To be able to discuss, the medication / drugs that the patient taken
and the diagnostic test that being perform for the patient.
7. Lastly, to be able to discuss our discharge plan for fully recovery of
our patient.
III. PATIENT’S PROFILE
IV. PHYSICAL ASSESSMENT
GENERAL SURVEY
Mr. X was lying semi-fowler’s on bed, conscious, coherent, afebrile with monitoring devices.
A. VITAL SIGNS
Date Shift Time
Temp BP RR PR Intake
Output
07/18/09
7am-1pm
36.8 210/100
58 20
B. HEADPink papillary conjunctiva, no nuchal rigidity and no carotid bruit.
C. NEUROLOGIC STATUS-Oriented to time, person and place.
CRANIAL NERVES ASSESSMENT
CN I- can smellCN II- (2-3) ERTLCN III, IV, VI- EDM, intactCN V- (+) corneal reflexCN VII- no facial asymmetryCN IX- (+) gag reflexCN XI- can shrug shoulderCN XII- tongue at midline
D. PULMONARY SYSTEM
-Respiratory rate was 58 cpm-SCE, no vesicular breath sounds.-AP, Apical beat at the 6th ICS anterior axillary line normal
sounds.
E. GASTROINTESTINAL SYSTEMFlabby, NaBS, no abdominal bruit, (-) edema,(-) cyanosis.
F. MUSCULOSKELETAL SYSTEM
The patient manifested good posture and moved voluntarily; he had symmetrical musculature on both sides of the body. Weakness was noted.
G. GENITO- URINARY SYSTEM
Patient voided 60 – 350 cc per shift as weighed and yellow in color.
Three cavities, called the primary brain vesicles, form during the early
embryonic development of the brain. These are the forebrain
(prosencephalon), the midbrain (mesencephalon), and the hindbrain
(rhombencephalon).
The telencephalon generates the cerebrum (which contains the
cerebral cortex, white matter, and basal ganglia).
The diencephalon generates the thalamus, hypothalamus, and
pineal gland.
The mesencephalon generates the midbrain portion of the brain
stem.
The metencephalon generates the pons portion of the brain stem
and the cerebellum.
The myelencephalon generates the medulla oblongata portion of
the brain stem
Figure
1
The four divisions of the adult
brain.
The cerebrum consists of two cerebral hemispheres connected by
a bundle of nerve fibers, the corpus callosum. The largest and most
visible part of the brain, the cerebrum, appears as folded ridges
and grooves, called convolutions. The following terms are used to
describe the convolutions:
A gyrus (plural, gyri) is an elevated ridge among the
convolutions.
A sulcus (plural, sulci) is a shallow groove among the
convolutions.
A fissure is a deep groove among the convolutions.
The deeper fissures divide the cerebrum into five lobes (most
named after bordering skull bones)—the frontal lobe, the parietal
love, the temporal lobe, the occipital lobe, and the insula. All but
the insula are visible from the outside surface of the brain.
A cross section of the cerebrum shows three distinct layers of
nervous tissue:
The cerebral cortex is a thin outer layer of gray matter. Such
activities as speech, evaluation of stimuli, conscious thinking,
and control of skeletal muscles occur here. These activities
are grouped into motor areas, sensory areas, and association
areas.
The cerebral white matter underlies the cerebral cortex. It
contains mostly myelinated axons that connect cerebral
hemispheres (association fibers), connect gyri within
hemispheres (commissural fibers), or connect the cerebrum
to the spinal cord (projection fibers). The corpus callosum is a
major assemblage of association fibers that forms a nerve
tract that connects the two cerebral hemispheres.
Basal ganglia (basal nuclei) are several pockets of gray
matter located deep inside the cerebral white matter. The
major regions in the basal ganglia—the caudate nuclei, the
putamen, and the globus pallidus—are involved in relaying
and modifying nerve impulses passing from the cerebral
cortex to the spinal cord. Arm swinging while walking, for
example, is controlled here.
The diencephalon connects the cerebrum to the brain stem. It
consists of the following major regions:
The thalamus is a relay station for sensory nerve impulses
traveling from the spinal cord to the cerebrum. Some nerve
impulses are sorted and grouped here before being
transmitted to the cerebrum. Certain sensations, such as
pain, pressure, and temperature, are evaluated here also.
The epithalamus contains the pineal gland. The pineal gland
secretes melatonin, a hormone that helps regulate the
biological clock (sleep-wake cycles).
The hypothalamus regulates numerous important body
activities. It controls the autonomic nervous system and
regulates emotion, behavior, hunger, thirst, body
temperature, and the biological clock. It also produces two
hormones (ADH and oxytocin) and various releasing
hormones that control hormone production in the anterior
pituitary gland.
The following structures are either included or associated with the
hypothalamus.
The mammillary bodies relay sensations of smell.
The infundibulum connects the pituitary gland to the
hypothalamus.
The optic chiasma passes between the hypothalamus and the
pituitary gland. Here, portions of the optic nerve from each
eye cross over to the cerebral hemisphere on the opposite
side of the brain.The brain stem connects the diencephalon to the spinal cord. The brain stem resembles the spinal cord in that both consist of white matter fiber tracts surrounding a core of gray matter. The brain stem consists of the following four regions, all of which provide connections between various parts of the brain and between the
brain and the spinal cord
Figure
2
Prominent structures of the brain
stem.
The midbrain is the uppermost part of the brain stem.
The pons is the bulging region in the middle of the brain
stem.
The medulla oblongata (medulla) is the lower portion of the
brain stem that merges with the spinal cord at the foramen
magnum.
The reticular formation consists of small clusters of gray
matter interspersed within the white matter of the brain stem
and certain regions of the spinal cord, diencephalon, and
cerebellum. The reticular activation system (RAS), one
component of the reticular formation, is responsible for
maintaining wakefulness and alertness and for filtering out
unimportant sensory information. Other components of the
reticular formation are responsible for maintaining muscle
tone and regulating visceral motor muscles.
The cerebellum consists of a central region, the vermis, and two
winglike lobes, the cerebellar hemispheres. Like that of the
cerebrum, the surface of the cerebellum is convoluted, but the gyri,
called folia, are parallel and give a pleated appearance. The
cerebellum evaluates and coordinates motor movements by
comparing actual skeletal movements to the movement that was
intended.
The limbic system is a network of neurons that extends over a wide
range of areas of the brain. The limbic system imposes an emotional
aspect to behaviors, experiences, and memories. Emotions such as
pleasure, fear, anger, sorrow, and affection are imparted to events and
experiences. The limbic system accomplishes this by a system of fiber
tracts (white matter) and gray matter that pervades the diencephalon and
Increase oxygen demand Decrease oxygen supply in the blood
encircles the inside border of the cerebrum. The following components are
included:
The hippocampus (located in the cerebral hemisphere)
The denate gyrus (located in cerebral hemisphere)
The amygdala (amygdaloid body) (an almond-shaped body
associated with the caudate nucleus of the basal ganglia)
The mammillary bodies (in the hypothalamus)
The anterior thalamic nuclei (in the thalamus)
The fornix (a bundle of fiber tracts that links components of the
limbic system)
VII. PATHOPHYSIOLOGY
Dizziness, stiffening of extremeties, and non projectile
vomiting
Cerebrovascular disease
Motor, sensory, cranial nerves
disrupted
Cell injury and death
Inadequate blood perfusion
Cerebrovascular disease or brain attack happened due to modifiable factors possessed by the patient such as smoking, ingesting fatty foods, and hypertension that leads to vasospasm and an embolus that dislodged from an area of origin to the brain that results to increase oxygen demand and decrease oxygen supply in the blood. Because of inadequate blood perfusion it leads to brain cells injury and death, at this point neurons are no longer able to maintain aerobic respiration that caused to produce neurological dysfunction.
VIII. COURSE ON THE WARD
Date/Shift Approach/Intervention
07/14/09 - Admitted a 66 y/o male with the chief complaint of body weakness and vomiting and fetched in a stretcher
3-11 - routine care done- S/C ERMEOD Dr. Anluete, and MROD Dr. Solero, MIOD with made and carried out
- hooked to O2 inhalation with 2-3 LPM via nasal cannula
- hooked to cardiac monitor BP 260/100 mmHg HR 60 bpm
3:00pm - venicolysis started hooked IVF of PNSSL x KVO
5:00pm - received patient on bed awake via stretcher accompanied
ERMEOD, transferred to bed safely
- on NPO except meds
- with ongoing IVF of PNSSL @ 750 cc level regulated @ 10gtts/minand SD nicardipine 10mg + 90ml of D5W reg. @ 10gtts/min infusing well and hooked to infusion pump @ 5:20pm
5:30pm - hooked to cardiac monitor and pulse oximetry
- with NGT connected to bedside bottle
- with the ff. labs: cranial CT scan-TF and CBG @5:30pm
- seen and examined by Dr. Martinez with orders meds and carried out
- clopidogel 5 tabs stat then OD given
- for 2Decho with Doppler- to request AAC
10:25pm - shift citicoline drops to IV as ordered by Dr. Solero
- adequate UO
- V/S q hour, medicine clerk informed
- no complaints
- needs attended
- endorsed
11-7 - flaccid patient on bed
- with IVF of PNSSL @ 650 level q 6hr
- with nicardipine hold
- on NPO except meds
- assess; BP 170/100
- O2 @ 2LPM via nasal cannula
- on CBR without BPR
- on CTscan-TF
- urinalysis, creatinine
- for sputum AFB
- for sputum GS/CS
- CBG monitoring q 12
- for FBS, hemoglobin,A1C
- V/S taken and recorded
- due meds given
- above IVF hooked and consumed @ same rate
- (-) BM
- needs attended
- endorsed
07/15/09
7-3 - received patient ongoing PNSS with same regulation and rate; afebrile - with O2 @ 2LPM connected to nasal cannula
- with NGT intact
- with CBG monitoring q 12
- for sputum AFB
- for 2Decho with Doppler
- BP: 130/90 mmHg
- endorsed
Addendum - start feeding AP order
- for SGOT
- (-)gag reflex
3-11 - received patient on bed with ongoing IVF of PNSSL
- with NGT to start of 1600 kcal in feedings, DM diet
- with O2 inhalation @ 2LPM via nasal cannula
- with FC to urobag
- with CBG monitoring
- for 2Decho with Doppler
- sputum GS/CS-TF
- still for sputum AFB
4:30pm - S/E by Dr. Martinez, orders were made and carried out
- start dilantin suspension, to load 12ml x 6doses q 4 then
4ml q 6
- for repeat scan (plain) on Thursday to reg. AAC
5:00pm - dilantin 100mg IV given slow push
7:30pm - s. electrolytes and SGPT result in referred to Dr. Simon
- due meds given
- refer prn
- no BM, afebrile
- endorsed
11-7 - received patient on bed - with ongoing IVF PNSS @ level of 100cc regulated @ 21gtts/min
- on 1600kcal feedings DM diet
- sputum GS/CS-TF
- CBG monitoring q 12
- for sputum AFB
- for repeat plain CTscan
1;15am - above IVF consumed and hooked same IVF and rate
- V/s taken and recorded
- due meds given
- I&O monitored and recorded
- no BM, afebrile
- refer prn
- needs attended
- endorsed
07/16/09
7-3 - received patient lying on bed
- with ongoing IVF PNSS with same reg. and rate
- afebrile, BP: 100/70mmHg
- with NGT intact
- with O2 @ 2LPM via nasal cannula
- for sputum AFB x 5 days
- for 2Decho
- needs attended
- endorsed
3-11 - received patient awake on bed- with ongoing IVF PNSS reg. @ same rate
- with FC connected to urobag
- with OF 1600kcal; 6 feedings
- for 2Decho
- for sputum GS/CS
- on CBR without SBR
- repeat CTscan plain-TF
- due meds given
8;00pm - (+) restlessness- MROD endorsed to give
Diphenhydramine 1 amp- given as ordered
9;30pm - Dr, Martinez made rounds with new order made to
Carried out
- if no restless until tomorrow may TROC, if (+) restless
@ 11pm, to give rizomil 2mg tab sat
- dilantin 125mg/5ml was ↓ freq. @ q 8- carried out
- V/S monitored and recorded
- I&O monitored and recorded
- needs attended
- endorsed
07/17/09
7-3 - received on bed with ongoing PNSS IVF @ 250cc level With same reg.- afebrile, BP: 130/70mmHg
- repeat CTscan (plain)
10:35am - due meds given
- possible TPOC
- BP: 140/80mmHg
- endorsed
3-11 - with NGT, OF 1600kcal feedings
- for sputum GS/CS
- for CTscan-TF
- V/S taken and recorded
07:00pm - (+) restlessness; refer to Dr. Solero
- diazepam 5mg given
- for CBG and Creatinine
- seen from time to time
- I&O monitored and recorded
- V/S taken and recorded
- refer prn
- endorsed
11-7 - received patient lying on bed, asleep - with IVF PNSS @ 900cc
- with cardiac monitoring q 12
- with NGT, OF 1600kcal and 6 feedings
- with 02 @ 2LPM via nasal cannula
- on CBR without BPR
- T:36.5C, HR:53bpm, RR:20cpm BP:130/70mmHg
- with FC connected to urobag
- still for sputum AFB
- for 2Decho
- repeat CTscan plain-TF
- due meds given
- morning care done
- (-)BM, afbrile
- needs attended
- endorsed
07/18/09
7-3 - received patient on bed- with IVF PNSS @ 520cc level with same reg.
- afebrile, BP: 130/80mmHg
- with patent NGT
- with FC connect to urobag
- 2Decho
- sputum GS/CS
- due meds given
-endorsed
IX. NURSING CARE PLAN
XI. DISCHARGE PLANNING
M- Instructed immediate relatives to facilitate the patient to continue
taking the drugs given to her on the right time and with the right dose to facilitate continuity of care.
E- Encouraged immediate relatives to facilitate regular exercise such as
brisk walking but not making herself too much tired.
-Encouraged her not to carry heavy loads and do not force herself too much in doing household chores. Encouraged patient to limit number of hours in playing domino.
T- encouraged patient to have enough rest and comply to the physicians
when ever health problems occur
H-Encouraged and explained to her the benefits and advantages of proper
hygiene to promote wellness.
O- instructed patient to come back for follow up check up on the date
ordered.
D- advised patient to eat nutritional foods like fruits and vegetables. Eat a
well balanced diet. Instructed patient to limit eating foods high in fats and with cholesterols. And also avoid salty foods.
S- Encouraged pt to continue her habits in going to church every day and