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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins Timby/Smith: Introductory Medical-Surgical Nursing, 10/e Chapter 38: Caring for Clients with Cerebrovascular Disorders
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Page 1: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Timby/Smith: Introductory Medical-Surgical Nursing, 10/e

Timby/Smith: Introductory Medical-Surgical Nursing, 10/e

Chapter 38: Caring for Clients with Cerebrovascular Disorders

Page 2: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

HeadacheHeadache• Cephalgia

– Typically: Transient, benign

– Accompany many disorders

• Meningitis; Increased ICP; Brain tumors; Sinusitis

• Most Common

– Tension; Migraine; Cluster

• Without Sensory Nerves

– Skull; Brain

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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Tension Headache Tension Headache • Pathophysiology, Etiology: Scalp, Facial Muscles

– Vast network of sensory, motor nerves

– Stressful conditions: Can result in prolonged neck, facial muscle contraction

• Assessment Findings: Mild to severe pain

• Diagnostic Findings: CT scan; Brain scan; Radiographs; Angiography

• Medical Management

– Rest; Analgesics

– Stress management; Counseling

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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Migraine Headaches Migraine Headaches • Pathophysiology, Etiology: Dilation of cerebral blood

vessels

• Assessment Findings: Signs and Symptoms

– Aura; Mood change

– Fatigue; Nausea; Vomiting

– Vertigo; Sensitivity to light

• Diagnostic Findings

– CT scan; Angiography

– Brain scan; Radiographs

Page 5: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Migraine HeadachesMigraine Headaches

Figure 38-1 Chemical developments in migraine headaches. Cerebral blood vessels dilate in response to serotonin from platelets. Peptides

released from the trigeminal nerve intensify pain.

Page 6: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Migraine Headaches Migraine Headaches

• Medical Management

– Rest; Drug therapy; Biofeedback techniques

• Nursing Management

– Client instruction

• Self-administration of medications

• Measures to abort the migraine

• Lying in a dark room

• Minimizing noise and other stimuli

Page 7: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cluster Headache Cluster Headache • Pathophysiology, Etiology: Unknown

– Suggested lower-than-normal levels of serotonin

– Possible “triggers”

• Assessment Findings: Severe, unilateral head pain (usually behind one eye); Rhinorrhea

• Diagnostic Findings: HA pattern; Persistent HA: CT scan; Brain scan; Head, neck radiographs; Angiography

• Medical Management: Corticosteroids; Ergotamine derivatives; Vasoconstricting drugs; Anticonvulsants; Oxygen; Surgical intervention (if all else fails)

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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Nursing ProcessNursing Process

• Assessment

– Pain: Location; Characteristics; History; Duration

– Triggers

– Other symptoms

– Clients with chronic headaches

• Complete medical, allergy, and family history

• Frequency and description of pain

• Vital signs

Page 9: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Nursing ProcessNursing Process

• Diagnosis, Planning, and Interventions

– Pain related to

• Muscle tension

• Changes in cerebral blood flow

• Unknown etiology

• Evaluation of Expected Outcome

– Head pain is reduced or eliminated

Page 10: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Transient Ischemic Attacks Transient Ischemic Attacks

• Pathophysiology, Etiology: Temporary interruption in cerebral blood flow

– Common causes

• Assessment Findings: Signs and Symptoms

– Speech, visual disturbances; Confusion; Partial paralysis

• Diagnostic Findings: Neurologic examination; Ultrasound; Carotid arteriogram; CT scan; MRI

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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Transient Ischemic Attacks Transient Ischemic Attacks • Medical, Surgical Management

– Antiplatelet therapy

– Anticoagulant therapy

– Diet therapy

– Surgery

• Carotid endarterectomy

• Balloon angioplasty

Figure 38-2 In endarterectomy, plaque—a potential source of emboli in TIAs and CVAs—is surgically removed from the carotid artery.

Page 12: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Transient Ischemic Attacks Transient Ischemic Attacks

• Nursing Management

– Complete client history

– Vital signs and weight

– Capillary blood sugar check

– Neurologic examination

– Client monitoring after carotid artery surgery

– Client education

Page 13: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cerebrovascular Accident (Stroke) Cerebrovascular Accident (Stroke) • Pathophysiology, Etiology

– Ischemic strokes: Thrombotic; Embolic

– Hemorrhagic strokes

• Assessment Findings: Signs and Symptoms

• Numbness; Weakness

• Mental confusion; Impaired ambulation

• Severe headache

• Hemiplegia; Aphasia; Hemianopia

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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cerebrovascular Accident (Stroke)Cerebrovascular Accident (Stroke)• Diagnostic Findings

– CT Scan; MRI; Transcranial Doppler ultrasonography

– Single photon emission CT; Lumbar puncture; Cerebral angiography

• Medical, Surgical Management

– Prevention measures

– Emergency treatment according to cause

– Supportive treatment

Page 15: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cerebrovascular Accident (Stroke)Cerebrovascular Accident (Stroke)• Nursing Management

– Client education: Medication: Administration, side effects; Eating, swallowing techniques

– Heimlich maneuver

– Follow-up care: Speech pathologist; Dietitian

– Community resources for special care devices

– Regular exercises; Maintain extremities in proper anatomic position

Page 16: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cerebral Aneurysms Cerebral Aneurysms • Pathophysiology, Etiology: Congenital; Secondary to

hypertension and atherosclerosis

Figure 38-7 Intracranial aneurysm within circle of Willis

Page 17: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cerebral Aneurysms Cerebral Aneurysms • Assessment Findings: Signs and Symptoms

– Sudden, severe headache; Dizziness; N/V; Loss of consciousness

• Diagnostic Findings

– Cerebral angiography

– CT Scan; MRI

– Lumbar puncture

– Hunt-Hess classification system

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Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Cerebral Aneurysms Cerebral Aneurysms

• Medical Management

– Complete bed rest; Prevention of rebleeding

– Treatment of complications

– Anticonvulsants; Tranquilizers

– Mechanical ventilation

• Surgical Management

– Craniotomy

– Ligation of carotid artery

Page 19: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

Nursing ProcessNursing Process• Assessment

– Neurologic examination

– Vital signs; History

• Diagnosis, Planning, and Interventions

– Increased intracranial pressure

– Seizures

– Pain

• Evaluation of Expected Outcomes

Page 20: Chapter038

Copyright © 2010 Wolters Kluwer Health | Lippincott Williams & Wilkins

End of Presentation