Case presentation Huda Al-Shibli E.M . R1
Dec 14, 2014
Case presentation
Huda Al-ShibliE.M.R1
Outline;
Case presentation Discussiontake home massages
4 yr-old child2 day-h/o abdominal pain, loose motion and vomiting
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History 1 survey
2 survey
DDx Investigations
Rx
Disposition
1 survey
A: patentB: normal, RR 35, sPO2 97% in RA,
C: P 110, BP 100/84, T 37.0 cD: GCS; 15/15, pupils 2 mm reacting b/l ,
E: NADWt : 13 kg
What do you want to do?
History
Abdominal pain X 2 daysassociated with vomiting 5 times /day , small to moderate amount , non
projectile, Loose motion 5 times /day , small amount semisolid , no blood or mucous Child is less active , not feeding well
h/o cough and cold during the last week and now improvedNo h/o fever
No past h/o medical problem
2 survey
O/E: Small boy , looks unwell, tachypnic , but alert and responding
Not jaundiced or paleDry mucous membrane Capillary refilling 3 sec
ears : clearThroat : mild congestion and mild enlarged tonsilsChest ….. Clear
CVS …… S1+S2, no gallop, no murmur Abdomen: soft with ?epigastric tenderness, BS +ve, no hepto-splenomegaly CNS :no neurologic deficit, no meningeal signs.
What is your impression?What is your next step?
Impression : gastroenteritis with moderate dehydration ( 5-7%) Plan:
Blood investigations: cbc, ue1IVF 20 ml /kg NS bolus , then dextrose saline 0.45 % @100 ml/hr + kcl 10 mmol/500 ml
To give ondasterone and try oral intake Reassess later
Reassessment
After 1 hour: The child looks unwellNot active , but still alert and responding
RR 37Capillary refilling 3 sec
The mother said he is drinking good amount of water but he vomited 3 times and had one
small amount of loose stool .
Investigations
Lab: CbcHb : 12WCC: 12UE1
130:Na K: 4.0
13:Hco3Urea and creatinine :WNL
It was the end of the shift The case handed over to the next coming teamAfter reassessment , they sent VBG and showed pH of 7.2 and they check reflow it was 16 mmol/l So it was DKA and they started management
DKA
DKA is the leading cause of morbidity and mortality in children with T1DM ranging from 0.15 % to 0.31 . %
In addition, DKA also can occur in children with T 2 DM; this presentation is most common among
youth of African-American descent .
DEFINITION
Consensus statements from the European Society for Paediatric Endocrinology/Lawson Wilkins Pediatric Endocrine Society in 2004, the American Diabetes Association in 2006, and the International Society for Pediatric and Adolescent Diabetes in 2007 defined the
following biochemical criteria for the diagnosis of DKA: 1-Hyperglycemia, defined as a blood glucose of >200
mg/dL (11 mmol/L) AND 2-Metabolic acidosis, defined as a venous pH <7.3 and/or
3-a plasma bicarbonate <15 meq/L (15 mmol/L)
Take home messages
1-Not every loose motion and vomiting is a GE .2-If a dehydrated child not responded to fluid
therapy , and it is unlikely to be sepsis , ask yourself : is it a DKA?
3-check reflow for pt with ketone positive in the urine .