I. Introduction Pneumonia, an inflammation of the pulmonary parenchyma, is common in childhood, occurring more frequently in infancy and early childhood. Clinically, pneumonia may occur either as a primary disease or as a complication of another illness. A report published by UNICEF in cooperation with the World Health Organization, in 2006 has identified pneumonia as the forgotten killer of children. According to the report, pneumonia kills more children than any other illness – more than AIDS, malaria and measles combined. Over 2 million children die from pneumonia each year, accounting for almost 1 in 5 underfive deaths worldwide. Yet, little attention is paid to this disease. Pneumonia can be classified according to morphology, etiologic agent, or clinical form. According to morphology, there are three types: Lobar pneumonia, Bronchopneumonia or Interstitial pneumonia. In this case, the study will be all about bronchopneumonia, where it begins in the terminal bronchioles which become clogged with mucopurulent exudate to form consolidated patches in nearby lobules. Another way to classify it is based on the etiologic agent. It may be caused by a virus, bacteria, mycoplasm or aspiration of foreign substances. The causative agent is usually introduced into the lungs through inhalation or from the bloodstream. In the
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Transcript
I. Introduction
Pneumonia, an inflammation of the pulmonary parenchyma, is common in
childhood, occurring more frequently in infancy and early childhood. Clinically,
pneumonia may occur either as a primary disease or as a complication of another
illness.
A report published by UNICEF in cooperation with the World Health
Organization, in 2006 has identified pneumonia as the forgotten killer of children.
According to the report, pneumonia kills more children than any other illness – more
than AIDS, malaria and measles combined. Over 2 million children die from pneumonia
each year, accounting for almost 1 in 5 underfive deaths worldwide. Yet, little attention
is paid to this disease.
Pneumonia can be classified according to morphology, etiologic agent, or clinical
form. According to morphology, there are three types: Lobar pneumonia, Bronchopneumonia or Interstitial pneumonia. In this case, the study will be all about
bronchopneumonia, where it begins in the terminal bronchioles which become
clogged with mucopurulent exudate to form consolidated patches in nearby lobules.
Another way to classify it is based on the etiologic agent. It may be caused by a
virus, bacteria, mycoplasm or aspiration of foreign substances. The causative agent is
usually introduced into the lungs through inhalation or from the bloodstream. In the
whole case, it will deal about bacterial pneumonia, the causative agent of the
bronchopneumonia of the patient.
Bacterial pneumonia is often a serious infection. The pathogenetic mechanisms
involved are often aspiration or hematogenous dissemination. The cause varies
depending on the child’s age, underlying illness, and degree of immunosuppression or
immunocompetence.
In the 3-month to 5-year age group, Streptococcus pneumoniae, Moraxella
catarrhalis, and Group-A streptococci are common causes. Haemophilus influenzae
type b is causing fewer infections because of the Hib vaccine. Staphylococcus aureus
pneumonia is also now rarely seen in infants and toddlers. Mycoplasma pneumoniae
and S. pneumoniae are the dominant organisms in children over 5 years of age.
The clinical manifestations of pneumonia vary depending on the etiologic agent,
the child’s age, the child’s systemic reaction to the infection, the extent of the lesions,
and the degree of bronchial and bronchiolar obstruction. For bacterial pneumonia,
clinical manifestations are fever and toxic appearance. Infants and young children
develop more severe symptoms than older children. Respiratory distress may or may
not be present. In some cases, the only finding is an increased respiratory rate.
II. Nursing Process
A. ASSESSMENT
1. Personal History
Mother Bear mentioned that she gave birth via normal spontaneous delivery without any
complications at full term assisted by a midwife in the hospital. She was not picky on the
foods she eats during her pregnancy, she will eat whatever food is available in their
home and according to the mother, she usually have her prenatal check up at the
barangay health center.
FeedingBaby bear was breastfed right after he was born, until now.
Immunization Status
Baby bear had a complete immunization for his age. He had received 1 dose of BCG, 1
dose of DPT, 1 OPV, HepaB.
Growth and Development
Erik Erikson(Theory of Trust and
Mistrust)
Infancy- 0-1 year old
This is the period of
infancy through the
first one or two
years of life. The
child, well - handled,
nurtured, and loved,
develops trust and
security and a basic
optimism. Badly
handled, he
becomes insecure
and mistrustful.
Normal Response
The infant would be
able to gain a sense
of trust with his
parents, particularly
with her mother
because they are
able to meet their
responsibility to
provide warmth,
comfort, security,
sensory stimulation,
food to the infant.
Untoward Response
The infant failed to
develop a sense of
trust with his
parents, particularly
the mother because
they are not able to
provide basic
support failure to
meet infants’ needs.
Client’s response
Currently, baby bear
is within this Trust
vs. Mistrust stage.
As observed, he is
capable of
developing his trust
with his parents,
especially with his
mother. This is
evident when Baby
stops crying after
being cuddled by
his mother.
Sigmund Freud(Psychosexual Theory-Oral Stage)
Birth to 1 year old
During this stage, the
child's main focus is
around the rooting and
suckling reflex.
Pleasure and self-
gratification are
acquired by the mouth.
Because a sense of
satisfaction is being
acquired during this
stage, it also leads to a
sense of trust for the
infant.
Normal Response
The baby exhibits
concern for the
gratification that
can be felt from
oral stimuli as
evidenced by
pleasure from is
eating. The child
also engages in
activities like
sucking, biting,
swallowing and
manipulating
various parts of
the mouth.
Untoward Response
The child is
unable to elicit
gratification from
oral stimuli and is
passive in
activities like
sucking, biting,
swallowing and
manipulating
various parts of
the mouth.
Client’s Response
Baby bear is able
to demonstrate
activities such as
sucking and
swallowing.
2. Family Health Illness History
3. History of Past and Present Illness
Past Illness:
This is baby’s first hospitalization diagnosed with bronchopneumonia. He never
had any mild or severe past illness. He hasn’t developed any signs and symptoms prior
to asthma, although his family had a history of it.
Present Illness:
The patient is diagnosed with bronchopneumonia. He was admitted on
November 10, 2009. The patient experienced fever and cough last November 6, 2009.
However, mama thought that it is just a common colds and fever so she gave
paracetamol (tempra) for medications. But then on November 10, 2009 the patient
experienced difficulty of breathing and cyanosis. He was then rushed and admitted to
Mabalacat District Hospital. Due to the observed signs and symptoms manifested by the
patient, and after laboratory diagnosis was done, the doctor suspected that he has
wastes from the circulation”. Respiratory System also helps in maintaining the acid-base
balance of the body through the excretion of carbon dioxide from the blood.
The lungs are the major part of the Respiratory System and are considered to be
the largest organ and resemble large pink sponges because of their appearance. The
left lung is slightly smaller in size compare with the right lung because it shares space
with the heart and so as to accommodate the two. The two lungs are divided into lobes;
two in the left lung and three in the right. The pleura, which is a slippery membrane
covers and lines the inside of the chest wall. This helps the lungs move and glide
smoothly during each breath cycle.
Lower Respiratory System
Larynx
The larynx or “voice box” is a short passageway connecting laryngopharynx with
the trachea. It is situated at the midline of the neck anterior to the fourth through sixth
cervical vertebrae. Its wall consists of nine pieces of cartilage. Three occur singly
(thyroid cartilage, epiglottis, and cricoid cartilage), and three occur in pairs (arytenoid,
cuneiform, and corniculate cartilages). The lining of the larynx has cilia and goblet cells.
The mucus produced by the said structure helps trap dust not expelled in the upper
passages. The cilia in the upper respiratory tract move mucus and trapped particles
down toward the pharynx, the cilia in the lower respiratory tract move them up toward
the pharynx.
Trachea
The trachea or “windpipe” is a tubular passageway for air that is about 5 inches
long and 1 inch in diameter. It is located anterior to the esophagus and extends from
the larynx towards the superior border of the fifth thoracic vertebra, thereon it divides
into right and left primary bronchi. The epithelium on the lining of the trachea provides
the same protection as the membrane lining the nasal cavity and larynx against foreign
material such as dust.
There are 16-20 incomplete, horizontal rings of hyaline cartilage resembling the
letter C and is stacked one on top of the other. The open part of each cartilage ring
faces the esophagus. The cartilage rings provide a semi-rigid support so that the
tracheal wall does not collapse inward and obstruct the air passageway and during
inhalation and expiration as well.
Bronchi
After the trachea, it divides into a right primary bronchus, which goes into the
right lung, and a left primary bronchus, which goes into the left lung. The right primary
bronchus is more vertical, shorter, and wider than the left. The bifurcation or the point
of intersection where the trachea divides into right and left primary bronchi is called the
carina. Like the trachea, the primary bronchi contain incomplete rings of cartilage, and
the carina is formed by an inferior projection of the last tracheal cartilage. The mucous
membrane of the carina is one of the most sensitive areas for triggering a cough reflex.
Going deeper into the lungs, the main or primary bronchi divide to form the
secondary (lobar) bronchi, one for each lobe of the lung (three on the right and two on
the left). The secondary bronchi continue to branch, forming still smaller bronchi, called
tertiary (segmental) bronchi, that divide into bronchioles, which branch into even smaller
terminal bronchioles. This branching from the trachea going down resembles an
inverted tree and is commonly referred to as the “bronchial tree”. Some of the
bronchioles are no larger than 0.5 mm (0.02 inches) in diameter. The bronchioles divide
many more times in the lungs into an upside-down tree-like structure with progressively
smaller branches.
Alveoli
Tiny air sacs called alveoli are at the end of every bronchioles. The alveoli
comprise most of the lung tissue, with about 150 million alveoli per lung, and resemble
bunches of grapes. The alveoli send oxygen to the circulatory system while removing
carbon dioxide. Alveoli have thin elastic walls, thus allowing air to flow into them when
they expand; they collapse when the air is exhaled. Alveoli are arranged in clusters, and
a dense network of capillaries surrounds each cluster. The walls of the capillaries are
very thin; thus the air in the wall of the alveoli is very near to the blood in the capillaries
(only about 0.1 to 0.2 microns). Carbon dioxide is one of the waste products that are
excreted into the outside environment from the cells. The oxygen diffuses from the
alveoli to the capillaries since the concentration of oxygen is much higher in the alveoli
than in the capillaries. From the capillaries, the oxygen flows into larger vessels and is
then carried to the heart where it is pumped to the rest of the body. The forces of
exhalation cause the carbon dioxide to go back up through the respiratory passages
and out of the body. Numerous macrophages are interspersed among the alveoli.
Macrophages are large white blood cells that remove foreign substances from the
alveoli that have not been previously filtered out. The presence of the macrophages
ensures that the alveoli are protected from infection; they are the last line of defense of
the respiratory system.
7. The Patient and His Illness
Definition of the disease
Pneumonia is an illness of the lungs and respiratory system in which the alveoli
(microscopic air-filled sacs of the lung responsible for absorbing oxygen from the
atmosphere) become inflamed and flooded with fluid. Pneumonia can result from a
variety of causes, including infection with bacteria, viruses, fungi, or parasites, and
chemical or physical injury to the lungs.
Pneumonia is an acute infection of one or both lungs that can be caused by a
bacterium, usually Streptococcus or by a virus, fungus, or other organism. The causal
organisms reach the lungs through the respiratory passages. Usually an upper
respiratory infection precedes the disease. The lungs' air sacs fill with pus, mucus, and
other liquid and can not function properly. Oxygen cannot reach the blood. If there is not
enough oxygen in the blood, body cells cannot work right and might die. Alcoholism,
extreme youth or age, debility, immunosuppressive disorders and therapy, and
compromised consciousness are predisposing factors. When one or more entire lobes
of the lung are involved, the infection is considered a lobar pneumonia. When the
disease is confined to the air spaces adjacent to the bronchial area, it is considered a
bronchial pneumonia.
Predisposing/Precipitating Factors
Non-modifiable Factors:
Age- At extremes of ages, different body systems and processes are either
immature or degenerating. For infants, their body defenses and immunologic
responses are just starting to develop. Such condition increases their
susceptibility to different pathologic conditions.
Lack of normal anatomical structure- There are certain inherited defects of
cilia which result in less effective protection. Cigarette smoke, inhaled directly by
a smoker or second-hand by an innocent bystander, interferes significantly with
ciliary’s function, as well as inhibiting macrophage function.
Modifiable Factors:
Chronic conditions- Predispose a person to infection with pneumonia. These
include asthma, cystic fibrosis, diabetes, sickle cell anemia, lymphoma, leukemia,
emphysema and neuromuscular diseases; interfere with the seal of the epiglottis.
This increases the risk of aspiration into the lungs of those stomach contents with
their resident bacteria.
Environment- The mode of transmission of pneumonia is through airborne or
person contact because of the droplets that can be inhaled from an infected
person.
Health Status/ Body’s resistance- Health Status clearly points out on how an
individual will fight or favor a pathologic condition. Certainly, poor or unstable
health status will hasten the occurrence of any type of disease since poor health
suggests poor resistance and defense against disease.
Parasitic infection- It also includes some previously rare parasitic, such as
worms which would be able to cause illness in an individual possessing a normal
immune system.
Viruses - It interfere with ciliary’s function, allowing other microorganism
invaders (such as bacteria) access to the lower respiratory tract. In recent years
virus has resulted in a huge increase in the incidence of pneumonia. It may
cause by certain viruses and associated with symptoms of fever, cough, and
shortness of breath.
Smoke - Millions of microscopic hairs (cilia) cover the surface of the cells lining
the bronchial tubes. The hairs beat in a wave-like fashion to clear airways of
normal secretions, but irritants such as tobacco smoke paralyze the cilia,
causing secretions to accumulate. If these secretions contain bacteria, they can
develop into pneumonia.
Alcohol - interferes with normal gag reflex as well as with the action of the
white blood cells that fight infection.
Are exposed to certain chemicals or pollutants. The risk of developing some
types of pneumonia may be increased if an individual works in agriculture,
construction or around certain industrial chemicals or even with animals.
Exposure to air pollution or toxic fumes can also contribute to lung
inflammation, which makes it harder for the lungs to clear themselves.
Contact to a Person with Pneumonia – Pneumonia is a communicable
disease, thus having a close contact with person or an article, which is
contaminated, can contribute to having Pneumonia.
General signs of pneumonia:
Fever (usually quite high)
Cough: unproductive to productive with whitish sputum
Tachypnea
Breath sounds: rhonchi or fine crackles
Dullness with percussion
Chest pain
Retractions
Nasal flaring
Pallor to cyanosis (depends on severity)
Diffuse or patchy infiltration with peribronchial distribution on CXR
Irritable, restless or lethargic
Anorexia, vomiting, diarrhea, abdominal pain
Initially, the cough is usually hacking and nonproductive, and breath sounds are
diminished or heard as scattered crackles. When consolidation is present, breath
sounds may be tubular in quality with no adventitious noises. As the infection resolves,
coarse crackles and wheezing are heard, and the cough becomes productive with
purulent sputum.
Lack of specific signs indicating infection makes diagnosis in infancy particularly
difficult. An early sign of infection is often irritability or lethargy and poor feeding. Abrupt
fever may be accompanied by seizures. Respiratory distress is evident with air hunger,
tachypnea and circumoral cyanosis. Because pneumonia in newborns carries a high
morbidity and mortality, bacterial infection should be suspected in all neonates with
respiratory symptoms.
Staphylococcal pneumonia is rare but particularly progressive and must be
treated aggressively. The onset is rapid, with rapid deterioration. Conjunctivitis and
furuncles are signs of a probable staphylococcal infection.
Synthesis of the Disease (Book-based)
Invasion of microorganism which lodges in the upper respiratory tract
Reaches the lower respiratory tract causing damage to the lung tissues
Stimulates inflammatory response
Release of chemical mediators
(cytokine, bradykinin, histamine)
Attraction of neutrophils and accumulation of fibrinous exudates & bacteria
Increase in WBC
Lung parenchyma & alveoli consolidation
Bradykinin Histamine
Stimulation of hypothalamus
Increase body temperature
Hyperthermia
Stimulates goblet cells
Increase in mucosal secretions
Causes narrowing of the airways
Air passes through narrowed lumen
Cytokine
Accumulation of secretions
(+) rales
(+) cough
Decrease blood oxygenation and ineffective tissue
perfusion
DOB & Dyspnea
Synthesis of the disease (client-centered)
Invasion of microorganism which lodges in the upper respiratory tract
Reaches the lower respiratory tract causing damage to the lung tissues
Stimulates inflammatory response
Release of chemical mediators
(cytokine, bradykinin, histamine)
Attraction of neutrophils and accumulation of fibrinous exudates & bacteria
Lung parenchyma &
alveoli consolidation
Bradykinin Histamine
Stimulation of hypothalamus
Increase body temperature
Hyperthermia
Stimulates goblet cells
Increase in mucosal secretions
Causes narrowing of the airways
Air passes through narrowed lumen
Cytokine
Accumulation of secretions
(+) rales
(+) cough
Decrease blood oxygenation and ineffective tissue
perfusion
DOB & Dyspnea
(Decreased Hct and Hgb)
Non-modifiable factors: Age
Modifiable factors: Body’s resistance, contact to a person with pneumonia, environment, health status
B. PLANNING (NCP)
PROBLEM #1 Ineffective Airway Clearance r/t presence of productive cough 2º to Bronchopneumonia
ASSESSMENT NURSING DIAGNOSIS
SCIENTIFIC EXPLANATION
OBJECTIVES NURSING INTERVENTION
RATIONALE EXPECTED OUTCOMES
S = Ø
O = Patient manifested:
-Appears weak & restless
-Appears tachypneic
-With changes in rate, rhythm and depth of breathing
-With DOB and (+) wheezes on the right lung
-Appears cyanotic
-With (+) non-
Ineffective Airway Clearance r/t presence of
productive cough 2º to
Bronchopneumonia
The inflammation and increased secretions make it difficult to maintain a patent airway, which is cause by decrease ability to expel the excessive mucus produced that will lead to extensive obstruction of the airway.
SHORT-TERM:
After 4 hours of NI, the SO
will demonstrate behaviors to
improve airway
patency.
LONG-TERM:
After 4 days of NI, the
patient will be able to
maintain airway
patency.
INDEPENDENT NURSING
FUNCTION:
-Establish rapport.
-Monitor V/S especially respiratory rate
-Auscultate breath sounds, note areas of decreased/adventitious breath sounds as well as fremitus
-Elevate HOB or change position every 2 hours as necessary
- To gain patient’s trust.
-To evaluate degree of compromise
-To ascertain status and note progress or complications
-To enhance ventilation to various lung segments
SHORT-TERM:
After 4 hours of NI, the SO
shall have demonstrated behaviors to
improve airway
patency.
LONG-TERM:
After 4 days of NI, patient
shall have been able to
maintain airway
productive cough
Patient may manifest:
-Appears tachycardiac
-Wide-eyed
-Keep environment allergen-free
-Encourage client to increase OFI to at least 2000 ml/day within level of cardiac tolerance.
-Encourage adequate rest and limit activities to within client tolerance.
DEPENDENT NURSING
FUNCTION:
-Administer medications such as bronchodilators/ expectorants as indicated.
-For adequate patent airway
-To help liquefy secretions
-To promote wellness
-To treat underlying conditions and mobilize secretions
patency.
PROBLEM # 2 Hyperthermia
ASSESSMENT NURSING DIAGNOSIS
SCIENTIFIC EXPLANATION
OBJECTIVES NURSING INTERVENTION
RATIONALE EXPECTED OUTCOMES
S = Ø
O = Patient
manifested:
-Appears weak
and restless
-Diaphoretic
-warm skin
when touched
-increased
body
temperature
(T= 37.9°C)
Hyperthermia Because of the
inflammatory
response, there
will be release
of chemical
mediators.
Cytokine, a
chemical
mediator will act
on the
hypothalamus
which will result
in increase in
epinephrine
and
norepinephrine,
vasoconstriction
SHORT-
TERM:
After 4 hours
of NI, the
patient will
have
decrease in
body
temperature
from 37.9 to
37.2 ºC.
LONG-TERM:
After 3 days
INDEPENDENT
NURSING FUNCTION:
-Establish rapport.
-Monitor VS
-Kept dry back
-Encourage SO to dress
pt in comfortable and
loose clothing.
- To gain patient’s
trust.
-To obtain baseline
data.
-To prevent further
respiratory
complication.
-To promote heat
loss.
SHORT-
TERM:
After 4 hours
of NI, the
patient shall
have gained
a decrease in
body
temperature
from 37.9 to
37.2 ºC.
LONG-
TERM:
-convulsions of cutaneous
vessels. The
heat will be
produced as
peripheral
vasodilation
results in skin
flushing and
skin is warm to
touch.
of NI, the
patient will
manifest
normal VS
specifically
temperature.
-Perform TSB
-Encourage client to
increase OFI
-Encourage adequate
rest and limit activities to
within client tolerance
DEPENDENT
NURSING FUNCTION:
-Administer anti-pyretic
medication
-To promote
evaporation of heat.
-For mobilization of
secretions
-To regain lost
energy
- To decrease the
elevated body temp.
After 3 days
of NI, the
patient will
have
manifested
normal VS
specifically
temperature.
PROBLEM # 3 Ineffective Breathing Pattern r/t dyspnea 2º Bronchopneumonia