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CARDIAC EMERGENCY AND ARREST MANALI H SOLANKI F.Y.M.SC.NURSING J G COLLEGE OF NURSING
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Cardiac emergency ppt

May 07, 2015

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Page 1: Cardiac emergency ppt

CARDIAC EMERGENCY AND ARREST

MANALI H SOLANKI

F.Y.M.SC.NURSING

J G COLLEGE OF NURSING

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TERMINOLOGIES:

Preload:The amount of myocardial stretch just before systole caused by the pressure created by the volume of blood within a ventricle

Afterload:

The amount of resistance to ejection of blood from a ventricle

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Heart failure (HF):

The inability of the heart to pump sufficient blood to meet the needs of the tissues for oxygen and nutrients; signs and symptoms of pulmonary and systemic congestion may or may not be present

Stroke volume (SV):

Amount of blood pumped out of the ventricle with each contraction

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INTRODUCTION

• Cardiovascular disease is the number one killer of adults. Prompt recognition and initiation of appropriate treatment can save lives during three of the most deadly cardiac emergencies: angina pectoris, myocardial infarction and congestive cardiac failure.

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ANATOMY AND PHYSIOLOGY OF HEART

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ANGINA PECTORIS:

• DEFINITION:

Angina (angina pectoris - Latin for squeezing of the chest) is chest discomfort that occurs when there is a decreased blood oxygen supply to an area of the heart muscle.

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Types of Angina

• The major types of angina are stable, unstable, variant (Prinzmetal's), and microvascular.

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Stable Angina

• Stable angina is the most common type of angina. It occurs when the heart is working harder than usual. Stable angina has a regular pattern.

• Stable angina isn't a heart attack, but it suggests that a heart attack is more likely to happen in the future.

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Unstable Angina

• Unstable angina doesn't follow a pattern. It may occur more often and be more severe than stable angina.

• Unstable angina is very dangerous and requires emergency treatment. This type of angina is a sign that a heart attack may happen soon.

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Variant (Prinzmetal's) Angina

• Variant angina is rare. A spasm in a coronary artery causes this type of angina. Variant angina usually occurs while you're at rest, and the pain can be severe. It usually happens between midnight and early morning. Medicine can relieve this type of angina

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Microvascular Angina

• Microvascular angina can be more severe and last longer than other types of angina. Medicine may not relieve this type of angina.

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CAUSES:

• Coronary artery spasm• Coronary-artery disease with

partial blockage or spasm of arteries that supply blood to the heart.

• Anaemia.• Overactive thyroid gland.• Heart-valve disease.

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RISK FACTORS:Diabetes

Family history of coronary heart disease before age 50

High blood pressure

High LDL cholesterol and low HDL cholesterol

Obesity

Smoking

Cold weather

Emotional stress

Large meals

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PATHO-PHYSIOLOGY

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SIGN AND SYMPTOMS

• Tightness, squeezing, pressure or ache in the chest.

• Sudden breathing difficulty (sometimes).• Chest pain similar to indigestion.• A choking feeling in the throat.• Chest pain that radiates to the jaw, teeth

or earlobes.• Heaviness, numbness, tingling or ache in

the chest, arm, shoulder, elbow or hand usually on the left side.

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• A feeling of indigestion or heartburn• Dizziness or light-headedness• Nausea, vomiting, and sweating• Palpitations• Shortness of breath• Unexplained tiredness after activity

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ASSESSMENT AND DIAGNOSTIC FINDINGS

• electrocardiogram (ECG)

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Exercise stress test

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Thallium stress test

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Coronary angiogram

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MEDICAL MANAGEMENT• Nitrates

Glyceryl Trinitrate

• Isosorbide dinitrate

• Beta blockerse.g. Propranolol, Atenolol, Metoprolol, Pindolol, Oxprenolol

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Surgical Treatment

• Coronary Angioplasty

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Coronary artery bypass grafting

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NURSING MANAGEMENT

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MYOCARDIAL INFARCTION

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DEFINITION:

• Myocardial infarction (MI) or acute myocardial infarction (AMI), commonly known as a heart attack, is the interruption of blood supply to a part of the heart, causing heart cells to die

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CAUSES

• Atherosclerosis• High cholesterol levels• High blood pressure• Diabetes• Obesity and Overweight• Smoking• Heredity

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PATHO-PHYSIOLOGY

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SIGN AND SYMPTOMS

• Chest discomfort, mild pain• Coughing• Crushing chest pain• Dizziness• Dyspnea (shortness of breath) • Face seems gray

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• A feeling of terror that your life is coming to its end

• Feeling really awful (general feeling)

• Nausea• Restlessness• The person is clammy and sweaty• Vomiting

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ASSESSMENT AND DIAGNOSTIC FINDINGS

• Physical examination • Electrocardiogram

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• Blood tests• Chest X-ray

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COMPLICATIONS

• Arrhythmia.• Cardiogenic shock.• Heart failure• Pulmonary edema• Pericarditis.

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Other complications for Myocardial Infarction (MI) include:

• Rupture of the atrial or Ventricular septum,

• Ventricular wall, or valves;

• Ventricular aneurysms

• Cerebral or pulmonary emboli

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MEDICAL MANAGEMENT

• Anti-platelet medicines• Nitroglycerin• ACE (angiotensin converting enzyme)

inhibitors• Beta-blocking agents• Coronary Angioplasty• Coronary Artery Bypass Grafting

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NURSING MANAGEMENT

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CONGESTIVE CARDIAC FAILURE

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DEFINITION

• Heart failure, also called "congestive heart failure," is a disorder where the heart loses its ability to pump blood efficiently.

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CAUSES:

• coronary artery disease and heart attack (which may be "silent")

• cardiomyopathy• high blood pressure (hypertension)• heart valve disease• congenital heart disease• alcoholism and drug abuse

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PATHO-PHYSIOLOGY

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SIGN AND SYMPTOMS

• Fatigue• Swelling (oedema) of the ankles and

legs or abdomen• An increase in body weight• Shortness of breath• Increased urination

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ASESSMENT AND DIAGNOSTIC FINDINGS

• Chest x-ray• Electrocardiogram (ECG)• Blood tests• Echocardiogram• MUGA scan• Stress test

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MEDICAL MANAGEMENT

• Angiotensin II receptor blockers (ARBs)• Vasodilators/Nitrates• Potassium or magnesium • Aldosterone inhibitors (spironolactone)• Calcium channel blockers (for diastolic

dysfunction only) • Inotropic therapy• Angiotensin-converting enzyme (ACE)

inhibitors• Beta blockers

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NURSING MANAGEMENT

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CARDIAC ARREST

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DEFINITION:

• Sudden cardiac arrest (SCA) is a condition in which the heart suddenly and unexpectedly stops beating. If this happens, blood stops flowing to the brain and other vital organs.

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CAUSES

• Coronary artery disease• Heart attack• Enlarged heart (cardiomyopathy)• Valvular heart disease• Congenital heart disease• Electrical problems in the heart

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Risk Factors

• Who have had a heart attack (myocardial infarction)

• Who have experienced heart failure (poor pumping heart)

• Who have survived a previous sudden cardiac arrest

• With a family history of sudden cardiac arrest

• With a low ejection fraction

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PATHO-PHYSIOLOGY

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SIGN AND SYMPTOMS

• Breathlessness  • Fatigue• Breathlessness on lying down• Waking up gasping for breath at night• Swollen abdomen• Enlarged liver• Visible venous pulse in neck • Swollen legs and ankles • Decreased exercise tolerance • Enlarged heart

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DIAGNOSTIC TEST

• Echocardiogram• Electrocardiogram (ECG)• Chest x-ray• Exercise test (stress test)• Cardiac catheterization• Laboratory studies may include CBC, ESR,

electrolytes, BUN/creatinine, glucose, TSH, calcium, magnesium, and phosphorus

• Blood cultures

 

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MEDICAL MANAGEMENT

• Angiotensin II receptor blockers (ARBs)• Vasodilators/Nitrates• Potassium or magnesium • Aldosterone inhibitors (spironolactone)• Calcium channel blockers (for diastolic

dysfunction only) • Inotropic therapy• Diuretics• Angiotensin-converting enzyme (ACE)

inhibitors

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SURGICAL MANAGEMENT

• Coronary artery bypass surgery

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Heart transplant

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NURSING MANAGEMENT

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Nursing Diagnosis

• Decreased Cardiac Output related to impaired contractility and increased preload and afterload

• Impaired Gas Exchange related to alveolar edema due to elevated ventricular pressures

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• Excess Fluid Volume related to sodium and water retention

• Activity Intolerance related to oxygen supply and demand imbalance

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BIBLIOGRAPHY:

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