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A burn is a type of injury to flesh caused by heat,
electricity, chemicals, light, radiation or friction. Most
burns only affect the skin (epidermal tissue and dermis).
Rarely, deeper tissues, such as muscle, bone, and bloodvessels can also be injured. Burns may be treated with first
aid, in an out-of-hospital setting, or may require more
specialized treatment such as those available at specialised
burn centers.
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1. Stage of neurogenic shock proves to be lethal. It
includes the fright, terror and hysterical reaction ofthe individual especially on the pain produced by the
irritation of nerve endings in the skin
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2. Stage of fluid loss first effect of burns is dilation
of capillaries and increasing permeability. Plasma
seeps out into the surrounding tissue to produce
blisters and edema. Fluid loss reduces the blood
volume, so that the blood becomes thicker, that the
volume of the cellular elements of the bloodincreases in relation to the volume of fluid (plasma)
of the blood. This change makes the circulation less
efficient. The loss of fluid volume is reflected in the
increasing hematocrit. Urinary output is reducedowing to fluid loss and hematocrit level is elevated or
hypovolemia hypotension decrease renal
perfusion and renal shutdown
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3. Stage of Burn slough and infection escharseparates from underlying viable tissue by formation
of slough this leaves a large open wound that is
usually infected. The infecting organism vary in the
upper part of the body often had colon bacilli as theinfecting organism. Infection, however, does not
occur suddenly, it probably begins soon after the
burn occurs and then gradually grows in the
sloughing tissue
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4
. Stage of Repair
repair of the burned area andsystemic repair
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Classification
Burns can be classified by mechanism ofinjury, depth, extent and associated injuries
and comorbidities.
Currently, burns are described according to the depth of
injury to the dermis and are loosely classified into first,
second, third and fourth degrees. This system was devised
by the French barber-surgeon Ambroise Pare and remainsin use today
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Nomenclature LayerInvolved
Appearance Texture Sensation Time ToHealing
Complications
First degreeEpider
mis
Redness
(erythema)Dry Painful
1wk or
lessNone
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NomenclatureLayer
InvolvedAppearance Texture Sensation
Time To
HealingComplications
Second
degree
(superficial
partial
thickness)
Extendsinto
superfic
ial
(papilla
ry)
dermis
Red with
clear
blister.
Blanches
withpressure
Moist Painful 2-3wks
Local
infection/
cellulitis
Distinguishing between the superficial-
thickness burn and the partial-thicknessburn is important, as the former may heal
spontaneously, whereas the latter often
requires surgical excision and skin grafting.
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NomenclatureLayer
Involved
Appearance Texture SensationTime To
Healing
Complications
Second
degree (deep
partial
thickness)
Extends
into deep
(reticular)
dermis
Red-and-
white with
bloody
blisters.
Lessblanching.
Moist Painful
Weeks -
may
progress
to third
degree
Scarring,
contractures
(may require
excision and
skin grafting)
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Nomenclature LayerInvolved
Appearance Texture Sensation Time ToHealing
Complications
Third degree
(full
thickness)
Extends
through
entire
dermis
Stiff and
white/brown
Dry,
leatheryPainless
Requires
excision
Scarring,
contractures,
amputation
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Nomenclature LayerInvolved
Appearance Texture Sensation Time ToHealing
Complications
Fourth
degree
Extends
through
skin,
subcutan
eous
tissue
and intounderlying
muscle
and bone
Charred
with escharDry Painless
Requires
excision
Amputation,significant
functional
impairment
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Burn severity
In order to determine the need for referral to a
specialised burn unit, the American Burn Association
devised a classification system to aid in the decision-
making process. Under this system, burns can be
classified as major, moderate and minor. This is
assessed based on a number of factors, including total
body surface area (TBSA) burnt, the involvement of
specific anatomical zones, age of the person and
associated injuries
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Major burns
Major burns are defined as:
y Age 10-50yrs: Partial thickness burns >25% TBSA
y Age 50: Partial thickness burns >20%TBSA
y Full thickness burns >10%
y Burns involving the hands, face, feet or perineum
y Burns that cross major joints
y Circumferential burns to any extremity
y Any burn associated with inhalational injury
y Electrical burns
y Burns associated with fractures or other trauma
y Burns in infants and the elderly
y Burns in persons at high-risk of developing
complications
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Moderate burns
Moderate burns are defined as:
y Age 10-50yrs: Partial thickness burns involving 15-
25% TBSA
y Age 50: Partial thickness burns involving 10-
20% TBSA
y Full thickness burns involving 2-10% TBSA
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Minor burns
Minor burns are:y Age 10-50yrs: Partial-thickness burns
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Burn surface area
Burns can also be assessed in terms of total body surface area
(TBSA), which is the percentage affected by partial thickness or fullthickness burns. First degree (erythema only, no blisters) burns are
not included in this estimation. The rule of nines is used as a quick
and useful way to estimate the affected TBSA. More accurate
estimation can be made using Lund & Browder charts which takeinto account the different proportions of body parts in adults and
children. The size of a person's hand print (palm and fingers) is
approximately 1% of their TBSA. The actual mean surface area is
0.8% so using 1% will slightly over estimate the size. Burns of 10%
in children or 15% in adults (or greater) are potentially lifethreatening injuries (because of the risk of hypovolaemic shock)
and should have formal fluid resuscitation and monitoring in a
burns unit.
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Causes ofBurns
Chemical - Most chemicals that cause chemical burns
are strong acids or bases. Chemical burns can be
caused by caustic chemical compounds such as
sodium hydroxide or silver nitrate, and acids such assulfuric acid. Hydrofluoric acid can cause damage
down to the bone and its burns are sometimes not
immediately evident
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Electrical burns are caused by either an electric shock
or an uncontrolled short circuit. (A burn from a hot,electrified heating element is not considered an
electrical burn.) Common occurrences of electrical
burns include workplace injuries, or being defibrillated
or cardioverted without a conductive gel. Lightning isalso a rare cause of electrical burns.
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Since normal physiology involves a vast number of applications
of electrical forces, ranging from neuromuscular signaling to
coordination of wound healing, biological systems are veryvulnerable to application of supraphysiologic electric fields.
Some electrocutions produce no external burns at all, as very
little current is required to cause fibrillation of the heart muscle.
Therefore, even when the injury does not involve any visible
tissue damage, electrical shock survivors may experience
significant internal injury. The internal injuries sustained may be
disproportionate to the size of the burns seen (if any), and the
extent of the damage is not always obvious. Such injuries may
lead to cardiac arrhythmias, cardiac arrest, and unexpected fallswith resultant fractures or dislocations
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Radiation burns are caused by protracted exposure to UV
light (as from the sun), tanning booths, radiation therapy
(in people undergoing cancer therapy), sunlamps,radioactive fallout, and X-rays. By far the most common
burn associated with radiation is sun exposure, specifically
two wavelengths of light UVA, and UVB, the latter being
more dangerous. Tanning booths also emit thesewavelengths and may cause similar damage to the skin
such as irritation, redness, swelling, and inflammation.
More severe cases of sun burn result in what is known as
sun poisoningor "heatstroke". Microwave burns arecaused by the thermal effects of microwave radiation.
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Management
First-aid treatment in the home: apply cold water
In the hospital most important goal of initial therapy
1.Solution: Ringers Lactate is the choice because it
most closely resembles the composition of the
extracellular fluid compartment
2.Dosage: 3-4 ml/KBW/%TBSA
3.S
chedule total quantity to be given in 24 hoursOne-half during the first 8 hours
The other during the next 16 hours
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Remove foreign adherent material b gentle washing withiodine-based solution or hexachlorophene and water,
then thoroughly rinsing with NSS
Shave hair from burned area and area immediatelysurrounding it
Excision of fragments of dead devitalized tissues
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Application of topical agents
1.Purpose: 0.5% silver nitrate unknown action; not very
popular because it stains the skin2.Gentamicin cream
3.Mafylon/Sulfamylon orSilver sulfadiazine (Silvadene)
the burn butter
1.purpose: to prevent bacterial invasion2.Method of application: applied evenly 1/8 inch in
thickness
3.Side effects
o Reduces buffering capacities of the blood because it
increases bicarbonate excretion (when bicarbonates are
broken down, they provide a heavy acid load).
Remember:Monitor pH level of blood
o Cause burning sensation for 20 minutes after application
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Dressing may (Closed method) or may not (open method)
be used. If closed method is used, dressings are changed
everydayWound debriment is done with each dressing change to
prevent eschar ( a tough coagulum of necrotic tissue).
Necrotic tissues are not only good media for bacteria,
but also promote growth of granulation tissues
Daily removal of dried cream by soaking in whirlpool
bathtub prior to debridement
Morphine sulfate, gr.1/4 relieve pain of second degreeburns
Use of Bradford frame for ease of turning and
maintenance of good body alignment
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Position flat on bed with legs extended specially during
the first 24-48 hours in order to:
1.Avoid postural shock because of fluid loss,
circulation to the head may become inadequate
2.Support healing of burn wounds
3.Prevent hip contractures
1.Proper splinting
2.Active and passive range of motion exercises
Grafting is done to minimize growth of granulation
tissues which results in contractures and ugly scars
1.Xenograft from animals
2.Homograft from other individuals
3.Autograft - oneself
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