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Page 1: Bone infection dina patho
Page 2: Bone infection dina patho

Proliferative periostitis. Cellular and reactive vital bone with individual trabeculae oriented perpendicular to the surface.

Page 3: Bone infection dina patho

Chronic osteomyelitis, ill-defined area of radiolucency of the right body of the mandible

adjacent to a recent extraction site.

B, after the initial intervention. The patient failed to return for follow-up because of lack of

significant pain. An enlarged, ill-defined radiolucency of the

right body of the mandible was discovered 2 years after the initial surgery.

Page 4: Bone infection dina patho

Acute suppurative osteomyelitis

Page 5: Bone infection dina patho

Acute osteomyelitis with sequestrum. Radiolucencyof the right body of the mandible with central radiopaque massof necrotic bone.

Page 6: Bone infection dina patho

CHRONIC OSTEOMYELITIS IN RADIATED MANDIBLE.

Page 7: Bone infection dina patho

Chronic osteomyelitis of the mandible associated with periodontal disease. Note moth-eaten radiolucentappearance.

Page 8: Bone infection dina patho
Page 9: Bone infection dina patho

CHRONIC OSTEOMYELITIS OF THE MANDIBLE

Page 10: Bone infection dina patho

CHRONIC OSTEOMYELITIS IN THE REGION OF THIRD-MOLAR EXTRACTION.

Page 11: Bone infection dina patho

Acute steomyelitis. Nonvital bone exhibits loss of the osteocytes from the lacunae. Peripheral resorption. Bacterial colonization. And surrounding inflammatory response also can be seen .

Page 12: Bone infection dina patho

CHRONIC OSTEOMYELITIS SHOWING FIBROUS MARROW AND OSTEOCLASTIC RESORPTION OF RESIDENT BONE.

Page 13: Bone infection dina patho
Page 14: Bone infection dina patho

Late-stage chronic osteomyelitis. A sequestrum trapped in acavity within the bone. It is surrounded by fibrous tissue containing aninfiltrate of inflammatory cells. Surgical intervention is needed to removean infected sequestrum such as this.

Page 15: Bone infection dina patho

High power view of a sequestrum showing non-vital bone (theosteocyte lacunae are empty), and eroded outline with superficial lacunaeproduced by osteoclastic resorption, and a dense surface growth ofbacteria.

Page 16: Bone infection dina patho

FOCAL SCLEROSING OSTEITIS.

Page 17: Bone infection dina patho

DIFFUSE SCLEROSING OSTEOMYELITIS.

Page 18: Bone infection dina patho
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Chronic osteomyelitis with proliferative periostitis (garré's osteomyelitis(

Page 21: Bone infection dina patho

Chronic osteomyelitis with proliferative periostitis (garré's osteomyelitis(

Page 22: Bone infection dina patho

Chronic osteomyelitis with proliferative periostitis (garré's osteomyelitis( of the right mandible (A(. B,note periosteal expansion in the radiograph. C, tissue from the central mandible is minimally inflamed and has afibroosseous appearance. D, periosteal tissue shows sclerotic laminations.

Page 23: Bone infection dina patho

Chronic osteomyelitis with proliferative periostitis (garré's osteomyelitis) of the right mandible (A). B,note periosteal expansion in the radiograph. C, tissue from the central mandible is minimally inflamed and has afibroosseous appearance. D, periosteal tissue shows sclerotic laminations.

Page 24: Bone infection dina patho
Page 25: Bone infection dina patho

Osteoradionecrosis. Same patient as depicted in note fistula formation of the left submandibular area resulting from osteoradionecrosis of the mandibular body.

Page 26: Bone infection dina patho

Osteoradionecrosis. Ulceration overlying leftbody of the mandible with exposure and sequestration of superficial alveolar bone.

Page 27: Bone infection dina patho

Osteoradionecrosis. Multiple ill-defined areas ofradiolucency and radiopacity of the mandibular body.

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Osteoradionecrosis of the lingual mandible precipitated by trauma.

Page 29: Bone infection dina patho

OSTEORADIONECROSIS OF THE MANDIBLE.

Page 30: Bone infection dina patho

Dry socket. Typical appearances of chronic alveolar osteitis; thesocket is empty and the bony lamina dura is visible.

Sequestration in a severe dry socket. Almost the whole of the lamina dura and attached trabeculae have become necrotic, forming a sequestrum. Healing is delayed while the sequestrum remains in place. Most dry sockets are not associated with sequestration, or with only small sequestra.