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Pericardial Effusion Pericardial EffusionPericardial EffusionPericardial EffusionPericardial EffusionPericardial EffusionPericardial EffusionPericardial Effusion

Apr 13, 2018

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    PERICARDIAL EFFUSION

    Present by:

    Sharifah Faseha

    Supervisor:

    dr. Abdul Hakim Alkatiri, SpJP.FIHA

    Cardiology Department

    Medical Faculty

    Hasanuddin University

    Case Report

    2013

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    History Taking

    Chief complaint: Shortness of breath

    Further anamnesis: It was felt since 3 days

    before she was admitted to the hospital.

    Dyspnea (+), DOE(+), PND (+), Orthopnea(+),

    chest pain(+) occur along with SOB.nausea(-),

    vomiting (-), fever(-) Urination and defecation

    were normal.

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    History of past illness

    History of hypertension (-)

    History of Diabetes Mellitus (-)

    History of heart disease (-) Family history of heart disease (-)

    History of breast cancer (+) undergone total

    right mastectomy

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    Clinical Examination

    General condition:

    moderate illness/ normal weight/ conscious

    Vital Sign

    BP: 120/80 mmHg R : 26x/minHR: 88 bpm T : 36,8oC (afebrile)

    Head: anemia (-), jaundice (-)

    Neck: JVP R+3cm H20

    Lung: BS: bronchovesicular

    Rh -/- basal Wh -/-

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    Clinical Examination

    Cardiac Examination

    Inspection : Ictus cordis wasntvisible

    Palpation : Ictus cordis wasntpalpable.

    Percussion : Right heart border in right

    parasternalis line, left heart border one finger

    from left midclavicle line ICS V.

    Auscultation : Heart Sounds = S I/II regular.

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    Clinical Examination

    Abdominal Examination

    Inspection : Flat, following breath movement

    Auscultation : Peristaltic sound (+), normal

    Palpation : Liver and spleen are unpalpable Percussion : Tympani (+)

    Extremities

    Oedema pretibial -/-

    Oedema dorsum pedis -/-

    Cyanosis (-), Clubbing finggers (-)

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    Electrocardiogram on 25th April 2013

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    ECG Interpretation

    Rhythm : Sinus Rhythm

    HR / QRS rate : 100bpm

    Axis : Normoaxis

    Regularity : Regular

    P wave : 0,08 s

    PR interval : 0,12 s

    QRS complex : 3 small squares (0,12 s)

    ST segment : Normal

    Conclusion : Sinus rhythm, HR 100bpm, normoaxis

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    Laboratory Findings

    Test Result Normal Value

    WBC 7,75 [10^3u/L] 4,010,0

    RBC 4,38 [10^6u/L] 4,06,0

    HGB 11,4 [g/dL] 13,017,0

    HCT 35,9 [%] 40,054,0

    PLT 309 [10^3u/L] 150 - 500

    Ureum 14 [mg/dL] 10 - 50

    Creatinine 0,6 [mg/dL] M(

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    Chest X-Ray

    Duplex pulmonarytuberculosis

    Cardiomegaly with

    dilatation of aorta

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    Echocardiogram

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    Echocardiogram description

    Good LV systolic function. EF 81%

    Hyperkinetic

    RA.RV collapse

    Heart valves : Mitral: Good function and movement

    : Aorta : 3cuspis. Calcification (-).Good function and movement

    : Tricuspid : Good function andmovement

    : Pulmonal : Good function andmovement

    Conclusion : Impending cardiac temponade

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    Working Diagnosis

    Pericardial Effusion

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    Therapy

    IVFD Nacl 0,9% 500cc

    Ceftriaxone 2gr/24hrs/iv (antibiotic)

    Pericardiocenthesis

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    Planning

    Cytology analysis of pericardial fluid

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    DISCUSSION

    PERICARDIAL EFFUSION

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    Pericardium

    Fibrous sac surrounding heart-dense network ofcollagen fibres

    Serous membranetwo continuous layersseparated by a small amount of fluid lubricant (30-50mls straw coloured (serous fluid))

    Layers are called visceral and parietal Visceral is inner layer (epicardium)

    Parietal is continuous with diaphragm and outer walls ofgreat arteries

    Surrounds the heart Continuous with the great arteries and the

    diaphragm

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    What is its function?

    Stabilises the position of the heart within the

    chest

    Prevents friction between the moving heart

    and adjacent structures

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    Pericardial effusion

    Pericardial effusion("fluid around the heart")

    is an abnormal accumulation of fluid in

    the pericardial cavity. Because of the limited

    amount of space in the pericardialcavity, fluid accumulation leads to an

    increased intrapericardial pressure which can

    negatively affect heart function.

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    Causes

    Infective (viral or bacterial)

    Following a myocardial infarction or cardiac surgery

    Radiation therapy

    Neoplastic disease (commonly lung or breast)

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    Pathophysiology

    Inflammation of pericardium due to infectionmetastasis process or lymphatic obstruction

    Disturb the equilibrium between the production and

    re-absorption of pericardial fluid

    Pericardium exude fluid, fibrin, blood cells.

    Increases fluid volume in pericardial space

    Pericardial effusion

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    Investigations and clinical signs

    Clinical examinationSOB, orthopnoea, tachycardia(varying degrees)

    Auscultationmay have muffled heart sounds

    ECG may show low amplitude QRS complexes and

    alternating axis CXRglobular appearance to heart and therefore

    increased cardiothoracic ratio

    Echosize of effusion and haemodynamic effect of it

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    CXR

    Being describe aswater bottle heart

    shape

    Widening of cardiac

    sillhoutte

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    Echocardiogram

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    Treatment

    Depends on the underlying cause and the severity of theheart impairment.

    If the pericardial effusion is due to a condition such as lupus,

    treatment with anti-inflammatory medications may help.

    If the effusion is compromising heart function and causingcardiac tamponade, it will need to be drained, most

    commonly by a needle inserted through the chest wall and

    into the pericardial space called pericardiocentesis.

    In some cases, surgical drainage may be required by cuttingthrough the pericardium creating a pericardial window, which

    allows ongoing drainage of fluid externally or internally such

    as into the pleural cavity

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    Additional info

    Pericardial effusion develops in 5% to 15% of patients withcancer and is sometimes the initial manifestation ofmalignancy.

    Most pericardial effusions in cancer patients result fromobstruction of the lymphatic drainage of the heartsecondary to metastases.

    The typical presentation is that of a patient with knowncancer who is found to have a large pericardial effusionwithout signs of inflammation.

    The most common malignant causes of pericardialeffusions are lung and breast cancers, leukemias(specifically acute myelogenous, lymphoblastic, and chronicmyelogenous leukemia [blast crisis]), and lymphomas.