obstetric emergencies.pptx

OBSTETRIC EMERGENCIES

OBSTETRIC EMERGENCIES

CONDITIONS

SHOCK VASA PREVIA CORD PRESENTATION AND CORD PROLAPSE AMNIOTIC FLUID EMBOLISM INVERSION OF UTERUS

1. SHOCK

Shock is defined as a state of circulatory inadequacy with poor tissue perfusion resulting in generalized cellular hypoxia leading to dysfunction of organs and cells

CLASSIFICATIONA. HYPOVOLEMIC SHOCK:- The result of a

reduction of intravascular volume Hemorrhagic shock: PPH, Post abortal hemorrhage,

ectopic pregnancy, placenta previa, abruptio placenta, obstetric sx , rupture of uterus etc…

Fluid loss shock: excessive diarrhoea, vomiting, rapid removal of amniotic fluid

Supine hypotensive syndrome: compression of inferior venacava.

Shock associated with DIC, intra uterine dead fetus syndrome, amniotic fluid embolism.

B. CARDIOGENIC SHOCK:-Impaired ability of the heart to pump blood

Failure of the left ventricular ejection in cardiac arrest and myocardial infarction

Failure of left ventricular filling associated with cardiac tamponade pulmonary embolism

C. NEUROGENIC SHOCK:- Chemical injury- Associated with aspiration of

gastrointestinal contents during general anaesthesia , especially caesarean section

Drug induced associated with spinal anaesthesia

D. SEPTIC SHOCK

Septic abortion, chorioamnionitis, polynephritis and rarely postpartum endometritis.

A. HYPOVOLEMIC SHOCK The body reacts to the loss of circulating fluid in stages as follows

INITIA STAGE:-

Reduction in fluid or blood

decrease the venous return to the heart

inadequate fill in ventricles

reduction in stroke volume and cardiac output

blood pressure decreases

decreased supply of oxygen to tissue

cell function will affect

COMPENSATORY STAGE:-

Drop in cardiac output

1. response from the sympathetic nervous system through the activation of receptors in the aorta and carotid arteries.

redistribution of blood to vital organs

heart rate increases to improve cardiac output and BP

Signs:- skin becoming pale and cool: peristalsis slows, urinary output is reduced and exchange of gas in the lungs is impaired. Pupils dilates. The sweat glands are stimulated and skin becomes moist and clammy.

2. Adrenaline and aldosterone from adrenal glands and anti diuretic hormone from posterior pituitary gland are secreted causing vasoconstriction

an increased cardiac output and a decrease in urinary output

Venous return to the heart will increase but, unless the fluid loss is replaced, this will not be sustained

PROGRESSIVE STAGE

This stage leads to multisystem failure. Compensatory mechanisms begin to fail, with vital organs lacking adequate perfusion. Volume depletion caused further fall in blood pressure and cardiac output. The coronary arteries suffer lack of supply. Peripheral circulation is poor. With weak or absent pulses.

LATE STAGE

Hypotension continues and cannot be reversed by replacement of fluid because of stagnation of blood at the micro vascular level. Colour of skin becomes ashen gray. Metabolic acidosis starts and for elimination of accumulated carbon dioxide, the respiratory rates becomes rapid. low volume pulse, oliguria and mental confusion occur. Multisystem failure and cell destruction are irreparable. Death ensures.

MANAGEMENT Urgent resuscitation is needed to prevent the mothers condition

from deteriorating and causing irreversible damage. The priorities are to:

1. Maintaining the airway

2. Replace fluid (infusion and transfusion)

3. Avoid warmth

4. Control of hemorrhage.

Clinical observations for the mother in shock

Assess the level of consciousness. Signs of restlessness and confusion are to be noted

Monitor blood pressure every 30 minutes or continuously.

Assess skin colour and temperature hourly. Assess central venous pressure and fluid balance for

adequacy for circulating volume Watch for occurrence of any further bleeding Detailed observation charts are to be maintained and

the mother may be transferred to a critical care unit

SEPTIC SHOCKInfection commonly from gram negative organisms (E.coli,

Proteus, or Pseudomonas) female genital tract Endotoxins present in their cells trigger body’s immune response

The placental site is the main point of entry for an infection associated with pregnancy and childbirth. This may occur following septic abortion, prolonged rupture of membranes, obstetric trauma or in the presence of retained placental tissue.

The body’s primary response to infection is alteration in the peripheral circulation. Cells damaged by the infecting organisms release histamine and enzymes that contribute to vasodilation and increased permeability of the capillaries.

Clinical signs In the initial phase, there is marked flushing of the

face and the skin feels warm. Temperature rise varies between 1010 to 1050F Tachycardia, tachypnoea and rigors occur. Hemorrhage may be present, which either could be

due to the events of childbearing or because of disseminated intravascular coagulation

As vasodilatation continues, hypotension leads to kidney damage with reduced glomerular filtration acute tubular necrosis and oliguria

If the shock conditions does not improve, the patient passes clinically to the stage of irreversible shock. She remains cold and clammy with ashen-gray cyanotic appearance

Anuria, cardiac or respiratory distress and coma will occur

Disseminated intravascular coagulation is also a feature

Multisystem organ failure will result as an effect of the continued hypotension and myocardial depression. Failure of the liver, brain and respiratory system follows, and death result.

Management of septic shockManagement is based on preventing further deterioration by restoring circulatory volume and eradication of the infection. A full infection screening should be carried out including a high vaginal swab, midstream urine and blood cultures. Retained products of conceptions if detected on ultrasound should be removed.

Antibiotics: Broad spectrum antibiotics are given to start after confirming the sensitivity, specifics antibiotics are given intravenously

Intravenous fluids and electrolytes: Septic shock associated hemorrhagic hypotension is treated with liberal infusion and blood transfusion. Oliguria with high specific gravity is an indication for liberal fluid administration

Correction of acidosis: Bicarbonate is administered to correct metabolic acidosis.

Maintenance of blood pressure: Inotropic agents such as adrenaline, noradrenaline, dopamine and dobutamine are administered to increase the cardiac contractility.

Corticosteroids are given to exert an antiendotoxin effect As a prophylactic measure for DIC, heparin may be given. In unresponsive septic shock following septic abortion or

puerperal sepsis, hysterectomy may be done to eliminate the source of infection.

3. VASA PREVIAThis term is used when a foetal blood vessel lies over the os in front of the presenting part. This usually occurs when foetal vessels from a velamentous insertion of the cord cross the area for the internal os to the placenta and also succenturiatelobe.

Vas previa occurs in less than 0.2% of pregnancies.

Diagnosis

Bimanual examination A speculum examination may be done to visualise the

blood vessel. It may be also visualised on ultrasound. Fresh vaginal bleeding, which commences at the time

of rupture of membranes, may be due to ruptured vasa previa.

Management Immediate consultation with physician Monitor fetal heart rate If fetus is alive emergency LSCS. If the mother in 2nd stage delivery should be expected

and a vaginal birth may be achieved. Paediatrician should be present May require blood transfusion for the baby

CORD PRESENTATION AND CORD PROLAPSE

There are three clinical types of abnormal descent of the umbilical cord by the side of the presenting part.

Occult prolapse: The cord is placed by the side of the presenting part and is not felt by the fingers on internal examination

Cord presentation: The cord is slipped down below the presenting part and is felt lying in the intact bag of membranes

Cord prolapse: the cord is lying inside the vagina or outside the vulva following rupture of membranes

Incidence: the incidence of cord prolapse is about 1 in 300

Predisposing factors; These are same for both presentation and prolapse of cord

Malpresentations Prematurity Multiple pregnancy Polyhydramnions High head High parity

Diagnosis Occult prolapse: this is difficult to diagnose. The

possibility should be suspected if there is (1)persistence of variable deceleration of fetal heart rate pattern detected on continuous fetal monitoring in an otherwise normal delivery or (2) persistent fetal soufflé with irregular heart sounds.

Cord presentation: Bimanual examination Cord prolapse The cord is felt below or beside the presenting part on

vaginal examination. A loop of cord may be visible at the vulva:

Risk to mothers Maternal

The maternal risks are incidental due to emergency operative delivery, which involves the risk of anaesthesia, blood loss and infection.

Fetal

The fetus is at risk of anoxia due to acute placental insufficiency from the moment cord is prolapsed. The danger is more in vertex presentation, especially when the prolapse through the anterior segment of the pelvis or when the cervix is partially dilated.

Management of cord presentationThe aim is to preserve the membranes and to expedite delivery The midwife should discontinue vaginal examination in order

to reduce the risk of rupturing the membranes Medical help should be summoned immediately Fetal heart should be auscultated as frequently as possible or

obtained through continuous electronic monitoring. Caesarean section is the most likely method of delivery During the time of preparing the woman for operative delivery,

she is kept in exaggerated Sim’s position to minimise compression

Management of cord prolapse

1. Immediate action Calls for urgent assistance. The mother and her family must be given explanation about the

findings and the emergency measures that will be needed. Stop oxytocin infusion If the baby is alive, the aim of immediate management to

minimize pressure on the cord until such time when the woman is prepared for assisted delivery of risk transferred to an equipped hospital. For this, the gloved fingers are to be introduced into the vagina to lift the presenting part of the cord. The fingers should be placed inside the vagina until definitive treatment is instituted

Postural treatment is given until the delivery of the baby, either vaginally or by caesarean section. The woman is placed in exaggerated elevated Sim’s position with pillow under the hip. The foot end of the bed may be elevated. High trendlenberg position or knee chest position, which has been traditionally mentioned, is very tiring and distressing to the woman

If the cord lies outside the vagina, it should be replaced in to the vaginal to minimize vasospasm due to irritation and to maintain the temperature.

If much of the cord are outside the vulva, it should be covered with sterile wet gauze, to prevent spasm of the umbilical vessels

2. Definitive management Caesarean section is the ideal management If immediate caesarean section is not possible or the baby is

too premature, reposition of the cord wrapped in a large piece of sterile roller gauze, is manually pushed above the presenting part under general anaesthesia. This is followed by stimulation of uterine contraction with oxytocin drip, if necessary. When the cervix is about three fourths dilated, ventouse traction may be applied to deliver the baby. This is possible only with vertex presentation and carries high fetal risks.

If the head is engaged, delivery is completed by forceps. With breech engaged, a breech extraction is done

If fetus is confirmed dead, labour is allowed to proceed, awaiting spontaneous termination.

AMNIOTIC FLUID EMBOLISM This condition occurs when amniotic fluid enters the maternal

circulation through a tear in the membranes or placenta. The body responds in two phases

– this initial phase is one of vasospasm causing hypoxia, hypotension, and cardiovascular collapse.

- This second phase is the development of left ventricular failure, with hemorrhage and coagulation disorder followed y pulmonary edema.

Mortality and morbidity are very high

Predisposing factors Transfer of amniotic fluid from the uterus to the maternal

circulation Amniotic fluid under pressure may enter maternal circulation

in the first phase of hypoxia during hypertonic uterine activity Procedures such as insertion of an intrauterine catheter and

artificial rupture of membranes In cases o placental abruption It can occur during a ceasaren section or termination of

pregnancy. Trauma may occur during intrauterine manipulation such as

internal podalic version

Clinical features Sudden onset of maternal respiratory distress: the

woman becomes severely dyspnic and cyanosed There is maternal hypotension and uterine hypertonia Fetal distress in response to hypoxia caused by

hypertonia Fetal distress in response to hypoxia caused by

hypertonia Cardiopulmonary arrest follows quickly in minutes Many others present with convulsions immediately

preceding the collapse

Emergency management

The mother may be in a state of collapse and resuscitation must be started at once.

Specific management of the condition is life support and high levels of oxygen are required.

Mothers who survive may suffer neurological impairment.

Complications Disseminated intravascular coagulation (DIC) Acute renal failure occurs due to heavy bleeding and

the prolonged hypovolemic hypotension

Effect on the fetus

Perinatal mortality and morbidity are high where amniotic fluid embolism occurs before the birth of the baby.

INVERSION OF UTERUS It is an extremely rare but a life threatening

complication in third stage in which the uterus is turned inside out partially or completely.

The incidence is about 1 in 20,000 deliveries

Varieties First degree – there is dimpling of the fundus which

still remains above the level of internal os. Second degree- the fundus passes through the cervix

but lies inside the vagina Third degree (complete) – the endometrium with or

without the attached placenta is visible outside the vulva. The cervix and part of the vagina may also be involved in the process.

It may occur before or after separation of placenta

Etiology Spontaneous (40%): this is brought about by localised atony on the

placental site over the fundus associated with sharp intra abdominal pressure as coughing, sneezing or bearing down effort. Fundal attachment of the placenta, short cord and placenta accrete are often associated.

Iatrogenic(60%) : This is due to the mismanagement of third stage of labour.

Pulling the cord – when the uterus is atonic specially when combined with fundal pressure

fundal pressure – while the uterus is relaxed – faulty technique or manual removal

common risk factors are uterine over enlargement, prolonged labour, fetal macrosomia, uterine malformations, morbid adherent placenta, short umbilical cord, and manual removal of placenta. It is more common in women with collagen disease.

Dangers Shock Haemorrhage Pulmonary embolism If left uncared for, it may lead to – infection, uterine sloughing and chronic

one

Diagnosis

Symptoms : acute lower abdominal pain with bearing down sensation

Signs : Abdominal examination – cupping or dimpling of the fundal surface,

bimanual examination not only helps to confirm the diagnosis but also the degree

Songraphy can confirm the diagnosis when clinical examination is not clear

Prevention

Do not employ the method to expel the placenta out when the uterus is relaxed.

Avoid pulling the cord

Management

Call for help Before shock develops, urgent manual replacement

even without anaesthesia.

Principal steps To replace that part first which is not inverted . To apply counter support by the other hand placed on the

abdomen After replacement, the hand should remain inside the uterus

until the uterus becomes contracted by parenteral oxytocin The placement is to removed manually only after the uterus

which facilitates replacement –a) to reduce the bulk which facilitates replacement or b) if partially separated to minimise the blood loss

Usual treatment of shock including blood transfusion should be arranged simultaneously

After the shock develops

Principal steps are The treatment of shock should be instituted with an

urgent normal saline drip and blood transfusion To push the uterus inside the vaginal if possible and

pack the vagina with antiseptic roller gauze Foot end of bed is raised Replacement f of the uterus either manually or

hydrostatic method (o Sullivan)under general anaesthesia is to be done along with resuscitative measures. Hydrostatic method is quite effective and less shock producing.

HYDROSTATIC REDUCTION (O’SULLIVAN’S TECHNIQUE) Hydrostatic reduction is a method of reinverting the uterus by infusing

warm saline into the vagina. The women may be placed in the Tredelenburg position to assist gravity

and reduce traction on the infundibulo-pelvic ligaments, round ligaments and the ovaries.

Method one Attach a 2 x 1 litre bags of warmed saline to a Cystoscopy giving set.

Additional fluids may be required. Insert the hand into the vagina with the open end of the tubing near the

posterior fornix. Obtain a seal at the vaginal entrance by enclosing the labia around the

wrist/hand to prevent fluid leakage. Infuse warmed fluid under gravity. Several litres of fluid may be required.

Sub acute stage To improve the general condition by blood transfusion Antibiotics are given to control sepsis Reposition of the uterus either manually or by

hydrostatic method may be tried If fails, reposition may be done by abdominal

operation (Haultain’s operation)